The loss of TGF-beta signaling promotes prostate cancer metastasis. - Wikidata - wikimedia - TriplyDB

The loss of TGF-beta signaling promotes prostate cancer metastasis.

The loss of TGF-beta signaling promotes prostate cancer metastasis.

http://www.wikidata.org/entity/Q34771729

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Endoglin phosphorylation by ALK2 contributes to the regulation of prostate cancer cell migration TGFbeta in Cancer A review of the past, present, and future directions of neoplasia Coming of Age in the Life of Molecular signatures of the primitive prostate stem cell niche reveal novel mesenchymal-epithelial signaling pathways Prostate cancer induced by loss of Apc is restrained by TGFβ signaling Differential expression of TGFbeta-stimulated clone 22 in normal prostate and prostate cancer.BMP7, a putative regulator of epithelial homeostasis in the human prostate, is a potent inhibitor of prostate cancer bone metastasis in vivo Epithelial-to-mesenchymal transition in prostate cancer: paradigm or puzzle?Gene targeting to the stroma of the prostate and bone.Distinct mechanisms of TGF-beta1-mediated epithelial-to-mesenchymal transition and metastasis during skin carcinogenesis.The role of GATA2 in lethal prostate cancer aggressiveness.Cathepsin B is the driving force of esophageal cell invasion in a fibroblast-dependent manner.Mouse models of prostate cancer: picking the best model for the question.TGFbeta signaling supports survival and metastasis of endometrial cancer cells.Local microenvironment provides important cues for cell differentiation in lingual epithelia.Network-based prediction for sources of transcriptional dysregulation using latent pathway identification analysis.A microRNA gene expression signature predicts response to erlotinib in epithelial cancer cell lines and targets EMT.Survey of genetically engineered mouse models for prostate cancer: analyzing the molecular basis of prostate cancer development, progression, and metastasis.MicroRNA and Transcription Factor Gene Regulatory Network Analysis Reveals Key Regulatory Elements Associated with Prostate Cancer Progression.Pro-metastasis function of TGFbeta mediated by the Smad pathway.Transforming growth factor-beta in cancer and metastasis.Androgenic control of transforming growth factor-beta signaling in prostate epithelial cells through transcriptional suppression of transforming growth factor-beta receptor II.Dysfunctional transforming growth factor-beta receptor II accelerates prostate tumorigenesis in the TRAMP mouse model Stroma-epithelium crosstalk in prostate cancer.Prostate cancer: the need for biomarkers and new therapeutic targets.SFMBT2 (Scm-like with four mbt domains 2) negatively regulates cell migration and invasion in prostate cancer cells.Expression of a Y-located human proto-oncogene TSPY in a transgenic mouse model of prostate cancer.TGF-beta signalling and immunity in prostate tumourigenesis.Growth factor signaling in metastasis: current understanding and future opportunities.Modeling prostate cancer in mice: something old, something new, something premalignant, something metastatic.4C-seq revealed long-range interactions of a functional enhancer at the 8q24 prostate cancer risk locus PIAS1-modulated Smad2/4 complex activation is involved in zinc-induced cancer cell apoptosis.Bone morphogenetic protein 7 induces mesenchymal-to-epithelial transition in melanoma cells, leading to inhibition of metastasis.PLP2, a potent deubiquitinase from murine hepatitis virus, strongly inhibits cellular type I interferon production.Promoter methylation correlates with reduced Smad4 expression in advanced prostate cancer.TGF-beta signaling and androgen receptor status determine apoptotic cross-talk in human prostate cancer cells.Prostate cancer cells regulate growth and differentiation of bone marrow endothelial cells through TGFbeta and its receptor, TGFbetaRII.Survey of differentially methylated promoters in prostate cancer cell lines.A Network-Based Integrative Workflow to Unravel Mechanisms Underlying Disease Progression.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

http://www.wikidata.org/entity/Q34771729

description

2003 nî lūn-bûn

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2003 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2003 թվականի մայիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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type

label

The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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The loss of TGF-beta signaling promotes prostate cancer metastasis.

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Agnieszka E Gorska

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Naoya Masumori

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Neil A Bhowmick

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Richard L Roberts

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Robert J Matusik

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Scott B Shappell

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Susan Kasper

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Tania Z Thomas

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Tom Case

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William H Tu

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P2860

Specificity in transforming growth factor beta-induced transcription of the plasminogen activator inhibitor-1 gene: interactions of promoter DNA, transcription factor muE3, and Smad proteins

Increased smad expression and activation are associated with apoptosis in normal and malignant prostate after castration

TGF-beta signaling in tumor suppression and cancer progression

A probasin-large T antigen transgenic mouse line develops prostate adenocarcinoma and neuroendocrine carcinoma with metastatic potential.

Promoter deletion and loss of retinoblastoma gene expression in human prostate carcinoma.

TGF-beta signaling blockade inhibits PTHrP secretion by breast cancer cells and bone metastases development.

Modulation of type beta transforming growth factor activity in bone cultures by osteotropic hormones.

Expression of a dominant-negative type II transforming growth factor beta (TGF-beta) receptor in the epidermis of transgenic mice blocks TGF-beta-mediated growth inhibition.

Expression of a truncated, kinase-defective TGF-beta type II receptor in mouse skeletal tissue promotes terminal chondrocyte differentiation and osteoarthritis.

Large fragment of the probasin promoter targets high levels of transgene expression to the prostate of transgenic mice.

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10.1016/S1476-5586(03)80058-1

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Harold L. Moses

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2003-05-01T00:00:00Z

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12869309

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