AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
about
Serum and Glucocorticoid Regulated Kinase 1 in Sodium HomeostasisSmall G proteins in the cardiovascular system: physiological and pathological aspectsRab GAPs AS160 and Tbc1d1 play nonredundant roles in the regulation of glucose and energy homeostasis in miceProtein kinase WNK1 promotes cell surface expression of glucose transporter GLUT1 by regulating a Tre-2/USP6-BUB2-Cdc16 domain family member 4 (TBC1D4)-Rab8A complex.Effects of aldosterone on insulin sensitivity and secretion.Neural precursor cell-expressed developmentally down-regulated protein 4-2 (Nedd4-2) regulation by 14-3-3 protein binding at canonical serum and glucocorticoid kinase 1 (SGK1) phosphorylation sites.PKB-Mediated Thr649 Phosphorylation of AS160/TBC1D4 Regulates the R-Wave Amplitude in the HeartAkt Links Insulin Signaling to Albumin Endocytosis in Proximal Tubule Epithelial Cells.Phosphorylation-dependent 14-3-3 protein interactions regulate CFTR biogenesisInsulin and AMPK regulate FA translocase/CD36 plasma membrane recruitment in cardiomyocytes via Rab GAP AS160 and Rab8a Rab GTPaseAkt Substrate of 160 kD Regulates Na+,K+-ATPase Trafficking in Response to Energy Depletion and Renal IschemiaThe insulin response integrates increased TGF-β signaling through Akt-induced enhancement of cell surface delivery of TGF-β receptors.Regulation of ENaC trafficking in rat kidney.AS160 controls eukaryotic cell cycle and proliferation by regulating the CDK inhibitor p21Dissecting the genetic components of a quantitative trait locus for blood pressure and renal pathology on rat chromosome 3.Membrane proteins as 14-3-3 clients in functional regulation and intracellular transport.Rab GTPases regulate the trafficking of channels and transporters - a focus on cystic fibrosis.Cleavage of endogenous γENaC and elevated abundance of αENaC are associated with increased Na⁺ transport in response to apical fluid volume expansion in human H441 airway epithelial cells.Rab-GAP TBC1D4 (AS160) is dispensable for the renal control of sodium and water homeostasis but regulates GLUT4 in mouse kidney.Elucidation of the distal convoluted tubule transcriptome identifies new candidate genes involved in renal Mg(2+) handling.
P2860
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P2860
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
description
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2010
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im Juni 2010 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2010/06/15)
@sk
vědecký článek publikovaný v roce 2010
@cs
wetenschappelijk artikel (gepubliceerd op 2010/06/15)
@nl
наукова стаття, опублікована в червні 2010
@uk
مقالة علمية (نشرت في 15-6-2010)
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name
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
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AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@en
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
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type
label
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@ast
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@en
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@nl
prefLabel
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@ast
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@en
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@nl
P2093
P2860
P3181
P356
P1476
AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking
@en
P2093
Kathryn W. Peters
Michael B. Butterworth
Raymond A. Frizzell
Xiubin Liang
P2860
P304
P3181
P356
10.1091/MBC.E10-01-0042
P577
2010-06-15T00:00:00Z