Gene expression analysis exposes mitochondrial abnormalities in a mouse model of Rett syndrome
about
Anaplerotic triheptanoin diet enhances mitochondrial substrate use to remodel the metabolome and improve lifespan, motor function, and sociability in MeCP2-null miceMitochondrial dysfunction in autism spectrum disorders: a systematic review and meta-analysisNeuropathological Mechanisms of Seizures in Autism Spectrum DisorderRole of mecp2 in experience-dependent epigenetic programmingGenetic modifiers of MeCP2 function in Drosophila.A review of Rett syndrome (RTT) with induced pluripotent stem cellsA Comprehensive Genomic Analysis Reveals the Genetic Landscape of Mitochondrial Respiratory Chain Complex DeficienciesMitochondrial localization and function of a subset of 22q11 deletion syndrome candidate genes.Enhanced cell death in MeCP2 null cerebellar granule neurons exposed to excitotoxicity and hypoxiaEnhanced anxiety and stress-induced corticosterone release are associated with increased Crh expression in a mouse model of Rett syndromeCerebellar gene expression profiles of mouse models for Rett syndrome reveal novel MeCP2 targets.Mecp2-null mice provide new neuronal targets for Rett syndrome.The methyl-CpG binding proteins Mecp2, Mbd2 and Kaiso are dispensable for mouse embryogenesis, but play a redundant function in neural differentiation.Mitochondria, bioenergetics, and the epigenome in eukaryotic and human evolutionDownstream targets of methyl CpG binding protein 2 and their abnormal expression in the frontal cortex of the human Rett syndrome brain.Global transcriptional and translational repression in human-embryonic-stem-cell-derived Rett syndrome neurons.Overexpression of Ubiquinol-Cytochrome c Reductase Core Protein 1 May Protect H9c2 Cardiac Cells by Binding with Zinc.Treatments for biomedical abnormalities associated with autism spectrum disorder.Methyl-CpG binding proteins: specialized transcriptional repressors or structural components of chromatin?Mitochondrial dysfunction in CA1 hippocampal neurons of the UBE3A deficient mouse model for Angelman syndromeThe MeCP2/YY1 interaction regulates ANT1 expression at 4q35: novel hints for Rett syndrome pathogenesis.Progressive increase in mtDNA 3243A>G heteroplasmy causes abrupt transcriptional reprogramming.Downregulation of the expression of mitochondrial electron transport complex genes in autism brains.Habituation without NMDA Receptor-Dependent Desensitization of Hering-Breuer Apnea Reflex in a Mecp2 Mutant Mouse Model of Rett SyndromeElevated 5-hydroxymethylcytosine in the Engrailed-2 (EN-2) promoter is associated with increased gene expression and decreased MeCP2 binding in autism cerebellum.Mitochondrial dysfunction can connect the diverse medical symptoms associated with autism spectrum disorders.Energetics, epigenetics, mitochondrial genetics.Brain region-specific expression of Fxyd1, an Mecp2 target gene, is regulated by epigenetic mechanisms.Proteomic analysis and functional characterization of mouse brain mitochondria during aging reveal alterations in energy metabolismNormal mitral cell dendritic development in the setting of Mecp2 mutationF2-dihomo-isoprostanes as potential early biomarkers of lipid oxidative damage in Rett syndrome.Linking epigenetics to human disease and Rett syndrome: the emerging novel and challenging concepts in MeCP2 research.Cytokine Dysregulation in MECP2- and CDKL5-Related Rett Syndrome: Relationships with Aberrant Redox Homeostasis, Inflammation, and ω-3 PUFAsMECP2 Duplication Syndrome: Evidence of Enhanced Oxidative Stress. A Comparison with Rett Syndrome.Rett syndrome: genes, synapses, circuits, and therapeuticsMouse models of neurodevelopmental disease of the basal ganglia and associated circuits.Long-term consequences of in utero irradiated mice indicate proteomic changes in synaptic plasticity related signalling.Correcting deregulated Fxyd1 expression ameliorates a behavioral impairment in a mouse model of Rett syndrome.MeCP2 modulates gene expression pathways in astrocytesX-linked mental retardation and epigenetics.
P2860
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P2860
Gene expression analysis exposes mitochondrial abnormalities in a mouse model of Rett syndrome
description
2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
@ast
im Juli 2006 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2006/07/01)
@sk
vědecký článek publikovaný v roce 2006
@cs
wetenschappelijk artikel (gepubliceerd op 2006/07/01)
@nl
наукова стаття, опублікована в липні 2006
@uk
مقالة علمية (نشرت في يوليو 2006)
@ar
name
Gene expression analysis expos ...... a mouse model of Rett syndrome
@ast
Gene expression analysis expos ...... a mouse model of Rett syndrome
@en
Gene expression analysis expos ...... a mouse model of Rett syndrome
@nl
type
label
Gene expression analysis expos ...... a mouse model of Rett syndrome
@ast
Gene expression analysis expos ...... a mouse model of Rett syndrome
@en
Gene expression analysis expos ...... a mouse model of Rett syndrome
@nl
prefLabel
Gene expression analysis expos ...... a mouse model of Rett syndrome
@ast
Gene expression analysis expos ...... a mouse model of Rett syndrome
@en
Gene expression analysis expos ...... a mouse model of Rett syndrome
@nl
P2093
P2860
P50
P3181
P356
P1476
Gene expression analysis expos ...... a mouse model of Rett syndrome
@en
P2093
John Curtis
Nikki Macleod
P2860
P304
P3181
P356
10.1128/MCB.01665-05
P407
P577
2006-07-01T00:00:00Z