Inactivation of Smad5 in endothelial cells and smooth muscle cells demonstrates that Smad5 is required for cardiac homeostasis
about
SM22alpha-targeted deletion of bone morphogenetic protein receptor 1A in mice impairs cardiac and vascular development, and influences organogenesisEndocardial Brg1 represses ADAMTS1 to maintain the microenvironment for myocardial morphogenesisNumb family proteins are essential for cardiac morphogenesis and progenitor differentiationEpicardial calcineurin-NFAT signals through Smad2 to direct coronary smooth muscle cell and arterial wall development.Altered prostate epithelial development and IGF-1 signal in mice lacking the androgen receptor in stromal smooth muscle cells.Connective tissue growth factor acts within both endothelial cells and beta cells to promote proliferation of developing beta cells.SMAD1 and SMAD5 Expression Is Coordinately Regulated by FLI1 and GATA2 during Endothelial Development.Cardiomyocyte overexpression of miR-27b induces cardiac hypertrophy and dysfunction in miceSmad1/5/8 are myogenic regulators of murine and human mesoangioblastsSmooth muscle cell-specific runx2 deficiency inhibits vascular calcification.Loss of neurotrophin-3 from smooth muscle disrupts vagal gastrointestinal afferent signaling and satiation.CHD7 interacts with BMP R-SMADs to epigenetically regulate cardiogenesis in miceThe role of Smad signaling in vascular and hematopoietic development revealed by studies using genetic mouse models.BMP signaling in vascular development and disease.VEGF, Notch and TGFβ/BMPs in regulation of sprouting angiogenesis and vascular patterning.Pericytes as targets in hereditary hemorrhagic telangiectasia.Targeting of Mesenchymal Stromal Cells by Cre-Recombinase Transgenes Commonly Used to Target Osteoblast Lineage CellsGenetic polymorphism of SMAD5 is associated with Kawasaki disease.Spatial and temporal regulation of coronary vessel formation by calcineurin-NFAT signaling.
P2860
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P2860
Inactivation of Smad5 in endothelial cells and smooth muscle cells demonstrates that Smad5 is required for cardiac homeostasis
description
2007 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2007
@ast
im Mai 2007 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2007/05/01)
@sk
vědecký článek publikovaný v roce 2007
@cs
wetenschappelijk artikel (gepubliceerd op 2007/05/01)
@nl
наукова стаття, опублікована в травні 2007
@uk
مقالة علمية (نشرت في مايو 2007)
@ar
name
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@ast
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@en
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@nl
type
label
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@ast
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@en
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@nl
prefLabel
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@ast
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@en
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@nl
P2093
P2860
P50
P1476
Inactivation of Smad5 in endot ...... quired for cardiac homeostasis
@en
P2093
Karin Sipido
Kjell Laperre
Liesbeth Vermeire
Lieve Moons
Lieve Umans
Virginie Bito
P2860
P304
P356
10.2353/AJPATH.2007.060839
P407
P577
2007-05-01T00:00:00Z