The GAP-related domain of the neurofibromatosis type 1 gene product interacts with ras p21
about
A novel human RasGAP-like gene that maps within the prostate cancer susceptibility locus at chromosome 1q25The small GTPase RalA targets filamin to induce filopodiaRegulation of Tiam1 nucleotide exchange activity by pleckstrin domain binding ligandsGermline mutations in TMEM127 confer susceptibility to pheochromocytomaThe N-terminal region of GAP regulates cytoskeletal structure and cell adhesionHepatitis C virus core protein interacts with the cytoplasmic tail of lymphotoxin-beta receptorExpression of three mammalian cDNAs that interfere with RAS function in Saccharomyces cerevisiaeIQGAP1, a calmodulin-binding protein with a rasGAP-related domain, is a potential effector for cdc42HsIsolation of rsp-1, a novel cDNA capable of suppressing v-Ras transformationMolecular genetics of neurofibromatosis type 1 (NF1)A novel phosphorylation-dependent RNase activity of GAP-SH3 binding protein: a potential link between signal transduction and RNA stability.Capillary malformation-arteriovenous malformation, a new clinical and genetic disorder caused by RASA1 mutationsCytoplasmic interactions of syndecan-4 orchestrate adhesion receptor and growth factor receptor signallingInactivation of the NF1 gene in human melanoma and neuroblastoma cell lines without impaired regulation of GTP.Rasp21ras activation via hemopoietin receptors and c-kit requires tyrosine kinase activity but not tyrosine phosphorylation of p21ras GTPase-activating proteinsar1, a gene from Schizosaccharomyces pombe encoding a protein that regulates ras1Localization of the rap1GAP catalytic domain and sites of phosphorylation by mutational analysisNf1-dependent tumors require a microenvironment containing Nf1+/-- and c-kit-dependent bone marrowIdentification of neurofibromin mutants that exhibit allele specificity or increased Ras affinity resulting in suppression of activated ras allelesSmall bowel gastrointestinal stromal tumours and ampullary cancer in Type 1 neurofibromatosisRASopathies: unraveling mechanisms with animal modelsAlternative splicing of the neurofibromatosis type I pre-mRNAA RASopathy gene commonly mutated in cancer: the neurofibromatosis type 1 tumour suppressorSmall G proteins in the cardiovascular system: physiological and pathological aspectsFrom neurodevelopment to neurodegeneration: the interaction of neurofibromin and valosin-containing protein/p97 in regulation of dendritic spine formationApplicability of superfolder YFP bimolecular fluorescence complementation in vitroAllosteric Modulation of Ras-GTP Is Linked to Signal Transduction through RAF KinasePkh1 and Pkh2 differentially phosphorylate and activate Ypk1 and Ykr2 and define protein kinase modules required for maintenance of cell wall integrity.Sst2, a negative regulator of pheromone signaling in the yeast Saccharomyces cerevisiae: expression, localization, and genetic interaction and physical association with Gpa1 (the G-protein alpha subunit).An essential function of a phosphoinositide-specific phospholipase C is relieved by inhibition of a cyclin-dependent protein kinase in the yeast Saccharomyces cerevisiaeThe RasGAP proteins Ira2 and neurofibromin are negatively regulated by Gpb1 in yeast and ETEA in humans.Overexpression of RPI1, a novel inhibitor of the yeast Ras-cyclic AMP pathway, down-regulates normal but not mutationally activated ras function.A new horizon of moyamoya disease and associated health risks explored through RNF213A novel approach for expression cloning of small GTPases: identification, tissue distribution and chromosome mapping of the human homolog of rhebThe motor protein kinesin-1 links neurofibromin and merlin in a common cellular pathway of neurofibromatosisAntioncogenes and human cancer.Neurofibromatosis type 1 - a model for nervous system tumour formation?Targeting of SCG10 to the area of the Golgi complex is mediated by its NH2-terminal regionAblation of NF1 function in neurons induces abnormal development of cerebral cortex and reactive gliosis in the brainNeurofibromatosis 1 (NF1) heterozygosity results in a cell-autonomous growth advantage for astrocytes
P2860
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P2860
The GAP-related domain of the neurofibromatosis type 1 gene product interacts with ras p21
description
1990 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1990
@ast
im November 1990 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1990/11/16)
@sk
vědecký článek publikovaný v roce 1990
@cs
wetenschappelijk artikel (gepubliceerd op 1990/11/16)
@nl
наукова стаття, опублікована в листопаді 1990
@uk
مقالة علمية (نشرت في 16-11-1990)
@ar
name
The GAP-related domain of the ...... product interacts with ras p21
@ast
The GAP-related domain of the ...... product interacts with ras p21
@en
The GAP-related domain of the ...... product interacts with ras p21
@nl
type
label
The GAP-related domain of the ...... product interacts with ras p21
@ast
The GAP-related domain of the ...... product interacts with ras p21
@en
The GAP-related domain of the ...... product interacts with ras p21
@nl
prefLabel
The GAP-related domain of the ...... product interacts with ras p21
@ast
The GAP-related domain of the ...... product interacts with ras p21
@en
The GAP-related domain of the ...... product interacts with ras p21
@nl
P2093
P3181
P1433
P1476
The GAP-related domain of the ...... product interacts with ras p21
@en
P2093
D. Viskochil
G. A. Martin
H. Haubruck
P. C. McCabe
P. O'Connell
R. M. Cawthon
W. J. Crosier
P304
P3181
P356
10.1016/0092-8674(90)90150-D
P407
P577
1990-11-16T00:00:00Z