Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome - Wikidata - wikimedia - TriplyDB

Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

http://www.wikidata.org/entity/Q28714223

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Decrease of 5-hydroxymethylcytosine in rat liver with subchronic exposure to genotoxic carcinogens riddelliine and aristolochic acid Dioxin induces Ahr-dependent robust DNA demethylation of the Cyp1a1 promoter via Tdg in the mouse liver Hydroxymethylation as a Novel Environmental Biosensor Iron and cancer: more ore to be mined.Rapid reprogramming of epigenetic and transcriptional profiles in mammalian culture systems.Reciprocal changes in DNA methylation and hydroxymethylation and a broad repressive epigenetic switch characterize FMR1 transcriptional silencing in fragile X syndrome Evidence that the capacity of nongenotoxic carcinogens to induce oxidative stress is subject to marked variability.Simultaneous single-molecule epigenetic imaging of DNA methylation and hydroxymethylation.Dynamic changes in 5-hydroxymethylation signatures underpin early and late events in drug exposed liver.TETonic shift: biological roles of TET proteins in DNA demethylation and transcription.Defending the genome from the enemy within: mechanisms of retrotransposon suppression in the mouse germline TET proteins: on the frenetic hunt for new cytosine modifications.5-hydroxymethylcytosine profiling as an indicator of cellular state.Recent advances in the bioanalysis of modified nucleotides in epigenetic studies.The role of 5-hydroxymethylcytosine in human cancer.Comparative analysis of affinity-based 5-hydroxymethylation enrichment techniques.The application of genome-wide 5-hydroxymethylcytosine studies in cancer research.Dose-response analysis of epigenetic, metabolic, and apical endpoints after short-term exposure to experimental hepatotoxicants.Ontogeny, distribution and potential roles of 5-hydroxymethylcytosine in human liver function.DNA immunoprecipitation semiconductor sequencing (DIP-SC-seq) as a rapid method to generate genome wide epigenetic signatures Loss of Tet1-Associated 5-Hydroxymethylcytosine Is Concomitant with Aberrant Promoter Hypermethylation in Liver Cancer.Balance of DNA methylation and demethylation in cancer development.Epigenetic modification of the PD-1 (Pdcd1) promoter in effector CD4(+) T cells tolerized by peptide immunotherapy.5-Hydroxymethylcytosine signatures in cell-free DNA provide information about tumor types and stages.What can epigenomics do for you?The allure of the epigenome Phenobarbital induces cell cycle transcriptional responses in mouse liver humanized for constitutive androstane and pregnane x receptors.Dynamics of ten-eleven translocation hydroxylase family proteins and 5-hydroxymethylcytosine in oligodendrocyte differentiation.Defining baseline epigenetic landscapes in the rat liver.Antibody-Based Detection of Global Nuclear DNA Methylation in Cells, Tissue Sections, and Mammalian Embryos.Affinity-Based Enrichment Techniques for the Genome-Wide Analysis of 5-Hydroxymethylcytosine.Modelling non-alcoholic fatty liver disease in human hepatocyte-like cells.

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

http://www.wikidata.org/entity/Q28714223

description

2012 nî lūn-bûn

@nan

2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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type

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

@ast

Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Non-genotoxic carcinogen exposure induces defined changes in the 5-hydroxymethylome

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Arne Müller

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Colm E Nestor

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Diana Reinhardt

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Edward J Oakeley

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Jonathan G Moggs

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Rémi Terranova

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P2860

Ultrafast and memory-efficient alignment of short DNA sequences to the human genome

DNA methylation patterns and epigenetic memory

The nuclear DNA base 5-hydroxymethylcytosine is present in Purkinje neurons and the brain

Hydroxylation of 5-methylcytosine by TET1 promotes active DNA demethylation in the adult brain

Impaired hydroxylation of 5-methylcytosine in myeloid cancers with mutant TET2

Thymine DNA glycosylase is essential for active DNA demethylation by linked deamination-base excision repair

Conversion of 5-methylcytosine to 5-hydroxymethylcytosine in mammalian DNA by MLL partner TET1

Genome-scale DNA methylation maps of pluripotent and differentiated cells

Tet-mediated formation of 5-carboxylcytosine and its excision by TDG in mammalian DNA

Genome-wide maps of chromatin state in pluripotent and lineage-committed cells

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