Herpes simplex virus 2 infection impacts stress granule accumulation - Wikidata - wikimedia - TriplyDB

Herpes simplex virus 2 infection impacts stress granule accumulation

Herpes simplex virus 2 infection impacts stress granule accumulation

http://www.wikidata.org/entity/Q30010094

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Who Regulates Whom? An Overview of RNA Granules and Viral Infections Shutoff of Host Gene Expression in Influenza A Virus and Herpesviruses: Similar Mechanisms and Common Themes Alphaherpesvirus Subversion of Stress-Induced Translational Arrest Interplay between Inflammation and Cellular Stress Triggered by Flaviviridae Viruses Viral and cellular proteins containing FGDF motifs bind G3BP to block stress granule formation Analysis of the interaction between host factor Sam68 and viral elements during foot-and-mouth disease virus infections Diversion of stress granules and P-bodies during viral infection.Role of Sam68 in post-transcriptional gene regulation.Herpes Simplex Virus 2 Virion Host Shutoff Endoribonuclease Activity Is Required To Disrupt Stress Granule Formation.DDX3 Interacts with Influenza A Virus NS1 and NP Proteins and Exerts Antiviral Function through Regulation of Stress Granule Formation.The herpes simplex virus 2 virion-associated ribonuclease vhs interferes with stress granule formation The Herpes Simplex Virus Virion Host Shutoff Protein Enhances Translation of Viral True Late mRNAs Independently of Suppressing Protein Kinase R and Stress Granule Formation.1st International Symposium on Stress-associated RNA Granules in Human Disease and Viral Infection.KSHV inhibits stress granule formation by viral ORF57 blocking PKR activation.

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P2860

Herpes simplex virus 2 infection impacts stress granule accumulation

http://www.wikidata.org/entity/Q30010094

description

2012 nî lūn-bûn

@nan

2012 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

Herpes simplex virus 2 infection impacts stress granule accumulation

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Herpes simplex virus 2 infection impacts stress granule accumulation

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Herpes simplex virus 2 infection impacts stress granule accumulation

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Herpes simplex virus 2 infection impacts stress granule accumulation

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Herpes simplex virus 2 infection impacts stress granule accumulation

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Herpes simplex virus 2 infection impacts stress granule accumulation

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Journal of Virology

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Herpes simplex virus 2 infection impacts stress granule accumulation

@en

P2093

Bruce W Banfield

http://www.w3.org/2001/XMLSchema#string

Kyle R Pangka

http://www.w3.org/2001/XMLSchema#string

Renée L Finnen

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P2860

The apoptosis-promoting factor TIA-1 is a regulator of alternative pre-mRNA splicing

The herpes simplex virus type 1 U(S)11 protein interacts with protein kinase R in infected cells and requires a 30-amino-acid sequence adjacent to a kinase substrate domain

The HTLV-1 Tax protein inhibits formation of stress granules by interacting with histone deacetylase 6

Stimulation of mammalian translation initiation factor eIF4A activity by a small molecule inhibitor of eukaryotic translation.

Cell proteins TIA-1 and TIAR interact with the 3' stem-loop of the West Nile virus complementary minus-strand RNA and facilitate virus replication.

Stress granule assembly is mediated by prion-like aggregation of TIA-1

Herpes simplex virus 1 induces cytoplasmic accumulation of TIA-1/TIAR and both synthesis and cytoplasmic accumulation of tristetraprolin, two cellular proteins that bind and destabilize AU-rich RNAs

Regulation of herpesvirus macromolecular synthesis. I. Cascade regulation of the synthesis of three groups of viral proteins

The RasGAP-associated endoribonuclease G3BP assembles stress granules

Inhibition of ribosome recruitment induces stress granule formation independently of eukaryotic initiation factor 2alpha phosphorylation

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8119-8130

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scholarly article

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2267687

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10.1128/JVI.00313-12

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15

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86

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2012-05-23T00:00:00Z

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22623775

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3421649

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