Regulation of smooth muscle alpha-actin expression in vivo is dependent on CArG elements within the 5' and first intron promoter regions.
about
Myocardin is a critical serum response factor cofactor in the transcriptional program regulating smooth muscle cell differentiation.Megakaryoblastic leukemia factor-1 transduces cytoskeletal signals and induces smooth muscle cell differentiation from undifferentiated embryonic stem cellsHRC is a direct transcriptional target of MEF2 during cardiac, skeletal, and arterial smooth muscle development in vivoMyocardin is a master regulator of smooth muscle gene expressionBinding of serum response factor to cystic fibrosis transmembrane conductance regulator CArG-like elements, as a new potential CFTR transcriptional regulation pathway.Mechanosensitive mechanisms in transcriptional regulationA Murine Myh6MerCreMer Knock-In Allele Specifically Mediates Temporal Genetic Deletion in Cardiomyocytes after Tamoxifen InductionCoronary Artery Disease Associated Transcription Factor TCF21 Regulates Smooth Muscle Precursor Cells That Contribute to the Fibrous CapAn α-smooth muscle actin (acta2/αsma) zebrafish transgenic line marking vascular mural cells and visceral smooth muscle cellsMyofibroblasts: trust your heart and let fate decide.The short and long of noncoding sequences in the control of vascular cell phenotypesHuman SM22 alpha BAC encompasses regulatory sequences for expression in vascular and visceral smooth muscles at fetal and adult stagesDifferential binding of an SRF/NK-2/MEF2 transcription factor complex in normal versus neoplastic smooth muscle tissuesRegulation of myosin light chain kinase and telokin expression in smooth muscle tissuesVariability in vascular smooth muscle cell stretch-induced responses in 2D cultureThe smooth muscle-selective RhoGAP GRAF3 is a critical regulator of vascular tone and hypertensionSTARS, a striated muscle activator of Rho signaling and serum response factor-dependent transcriptionHepatocyte nuclear factor-3 homologue 1 (HFH-1) represses transcription of smooth muscle-specific genesPIAS1 activates the expression of smooth muscle cell differentiation marker genes by interacting with serum response factor and class I basic helix-loop-helix proteins.Platelet-derived growth factor-BB-mediated activation of Akt suppresses smooth muscle-specific gene expression through inhibition of mitogen-activated protein kinase and redistribution of serum response factorFRS2 via fibroblast growth factor receptor 1 is required for platelet-derived growth factor receptor beta-mediated regulation of vascular smooth muscle marker gene expressionTranscriptional regulation of a contractile gene by mechanical forces applied through integrins in osteoblastsIncreased Smad1 expression and transcriptional activity enhances trans-differentiation of hepatic stellate cellsTGFβ1-induced Baf60c regulates both smooth muscle cell commitment and quiescenceThymine DNA glycosylase represses myocardin-induced smooth muscle cell differentiation by competing with serum response factor for myocardin bindingIdentification of Barx2b, a serum response factor-associated homeodomain proteinSerum response factor is required for cell contact maintenance but dispensable for proliferation in visceral yolk sac endotheliumThe serum response factor coactivator myocardin is required for vascular smooth muscle developmentAnalysis of SM22alpha-deficient mice reveals unanticipated insights into smooth muscle cell differentiation and functionSignaling pathways that control rho kinase activity maintain the embryonic epicardial progenitor state.Positive- and negative-acting Kruppel-like transcription factors bind a transforming growth factor beta control element required for expression of the smooth muscle cell differentiation marker SM22alpha in vivo.Smooth muscle differentiation marker gene expression is regulated by RhoA-mediated actin polymerization.Molecular mechanisms of decreased smooth muscle differentiation marker expression after vascular injury.CArG elements control smooth muscle subtype-specific expression of smooth muscle myosin in vivoThe smooth muscle myosin heavy chain gene exhibits smooth muscle subtype-selective modular regulation in vivo.Whole animal knockout of smooth muscle alpha-actin does not alter excisional wound healing or the fibroblast-to-myofibroblast transitionSmooth muscle cell phenotypic switching in atherosclerosisNuclear factor of activated T cells 5 regulates vascular smooth muscle cell phenotypic modulation.PIAS1 mediates TGFbeta-induced SM alpha-actin gene expression through inhibition of KLF4 function-expression by protein sumoylationPlatelet-derived growth factor-BB and Ets-1 transcription factor negatively regulate transcription of multiple smooth muscle cell differentiation marker genes.
P2860
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P2860
Regulation of smooth muscle alpha-actin expression in vivo is dependent on CArG elements within the 5' and first intron promoter regions.
description
1999 nî lūn-bûn
@nan
1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Regulation of smooth muscle al ...... first intron promoter regions.
@ast
Regulation of smooth muscle al ...... first intron promoter regions.
@en
type
label
Regulation of smooth muscle al ...... first intron promoter regions.
@ast
Regulation of smooth muscle al ...... first intron promoter regions.
@en
prefLabel
Regulation of smooth muscle al ...... first intron promoter regions.
@ast
Regulation of smooth muscle al ...... first intron promoter regions.
@en
P356
P1433
P1476
Regulation of smooth muscle al ...... first intron promoter regions.
@en
P2093
P304
P356
10.1161/01.RES.84.7.852
P577
1999-04-01T00:00:00Z