Antibody blockade of ICAM-1 and VCAM-1 ameliorates inflammation in the SAMP-1/Yit adoptive transfer model of Crohn's disease in mice.
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CCL25 and CCL28 promote alpha4 beta7-integrin-dependent adhesion of lymphocytes to MAdCAM-1 under shear flowAttenuation of TNF-driven murine ileitis by intestinal expression of the viral immunomodulator CrmDShining a light on intestinal trafficOsteopontin expression by CD103- dendritic cells drives intestinal inflammationTNF-alpha neutralization ameliorates the severity of murine Crohn's-like ileitis by abrogation of intestinal epithelial cell apoptosis.CD44 deficiency attenuates chronic murine ileitis.The fimbriae of enteroaggregative Escherichia coli induce epithelial inflammation in vitro and in a human intestinal xenograft model.Partial normalization of pubertal timing in female mice with DSS colitis treated with anti-TNF-α antibody.SAMP1/YitFc mouse strain: a spontaneous model of Crohn's disease-like ileitis.Spontaneous, immune-mediated gastric inflammation in SAMP1/YitFc mice, a model of Crohn's-like gastritis.Beta7 integrin deficiency suppresses B cell homing and attenuates chronic ileitis in SAMP1/YitFc mice.Colitis causes delay in puberty in female mice out of proportion to changes in leptin and corticosteroneA CD8+/CD103high T cell subset regulates TNF-mediated chronic murine ileitis.Expanded B cell population blocks regulatory T cells and exacerbates ileitis in a murine model of Crohn diseaseProbiotics promote gut health through stimulation of epithelial innate immunity.Leukocyte adhesion molecules in animal models of inflammatory bowel diseaseThe primary defect in experimental ileitis originates from a nonhematopoietic source.Critical role of endothelial P-selectin glycoprotein ligand 1 in chronic murine ileitis.Direct crosstalk between mast cell-TNF and TNFR1-expressing endothelia mediates local tissue inflammation.Blockade of PSGL-1 attenuates CD14+ monocytic cell recruitment in intestinal mucosa and ameliorates ileitis in SAMP1/Yit mice.In vivo demonstration of T lymphocyte migration and amelioration of ileitis in intestinal mucosa of SAMP1/Yit mice by the inhibition of MAdCAM-1.Increased lymphocyte trafficking to colonic microvessels is dependent on MAdCAM-1 and C-C chemokine mLARC/CCL20 in DSS-induced mice colitis.Quantitative lymphatic vessel trait analysis suggests Vcam1 as candidate modifier gene of inflammatory bowel disease.Clinical Response to Vedolizumab in Ulcerative Colitis Patients Is Associated with Changes in Integrin Expression ProfilesConcurrent infection with an intestinal helminth parasite impairs host resistance to enteric Citrobacter rodentium and enhances Citrobacter-induced colitis in mice.Helminth-primed dendritic cells alter the host response to enteric bacterial infectionICAM-1 as a molecular target for triple negative breast cancerRetinoic acid determines the precise tissue tropism of inflammatory Th17 cells in the intestine.The Role of the Mesentery in Crohn's Disease: The Contributions of Nerves, Vessels, Lymphatics, and Fat to the Pathogenesis and Disease Course.Role of the endothelium in inflammatory bowel diseases.Antiadhesion molecule therapy in inflammatory bowel disease.Multiparametric and semiquantitative scoring systems for the evaluation of mouse model histopathology--a systematic review.The chemokine receptor CCR9 is required for the T-cell-mediated regulation of chronic ileitis in mice.Mutant KRAS-induced expression of ICAM-1 in pancreatic acinar cells causes attraction of macrophages to expedite the formation of precancerous lesions.Effect of probiotics Lactobacillus acidophilus on Citrobacter rodentium colitis: the role of dendritic cells.Transcriptional modulation of intestinal innate defense/inflammation genes by preterm infant microbiota in a humanized gnotobiotic mouse modelEmerging biotherapies for inflammatory bowel disease.Novel biological strategies in inflammatory bowel diseases.Management of ulcerative colitis and Crohn's disease.Physiological basis for novel drug therapies used to treat the inflammatory bowel diseases. I. Immunology and therapeutic potential of antiadhesion molecule therapy in inflammatory bowel disease.
P2860
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P2860
Antibody blockade of ICAM-1 and VCAM-1 ameliorates inflammation in the SAMP-1/Yit adoptive transfer model of Crohn's disease in mice.
description
2001 nî lūn-bûn
@nan
2001 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Antibody blockade of ICAM-1 an ...... el of Crohn's disease in mice.
@ast
Antibody blockade of ICAM-1 an ...... el of Crohn's disease in mice.
@en
type
label
Antibody blockade of ICAM-1 an ...... el of Crohn's disease in mice.
@ast
Antibody blockade of ICAM-1 an ...... el of Crohn's disease in mice.
@en
prefLabel
Antibody blockade of ICAM-1 an ...... el of Crohn's disease in mice.
@ast
Antibody blockade of ICAM-1 an ...... el of Crohn's disease in mice.
@en
P2093
P921
P356
P1433
P1476
Antibody blockade of ICAM-1 an ...... el of Crohn's disease in mice.
@en
P2093
Cominelli F
Matsumoto S
Moskaluk CA
Rivera-Nieves J
P304
P356
10.1053/GAST.2001.29568
P407
P577
2001-12-01T00:00:00Z