Constitutive activation of the Ras/mitogen-activated protein kinase signaling pathway promotes androgen hypersensitivity in LNCaP prostate cancer cells.
about
Roles of the Raf/MEK/ERK pathway in cell growth, malignant transformation and drug resistanceMolecular pathology of prostate cancerActivation of the RalGEF/Ral pathway promotes prostate cancer metastasis to boneAcquisition of androgen independence by human prostate epithelial cells during arsenic-induced malignant transformation.Non-Coding RNAs in Castration-Resistant Prostate Cancer: Regulation of Androgen Receptor Signaling and Cancer MetabolismInsights into Chemoresistance of Prostate CancerThe mutational landscape of prostate cancerInorganic arsenic and human prostate cancerCompensatory pathways induced by MEK inhibition are effective drug targets for combination therapy against castration-resistant prostate cancerThe neuroendocrine-derived peptide parathyroid hormone-related protein promotes prostate cancer cell growth by stabilizing the androgen receptor.Simian virus 40 small t antigen mediates conformation-dependent transfer of protein phosphatase 2A onto the androgen receptor.Restoration of PTEN expression alters the sensitivity of prostate cancer cells to EGFR inhibitors.Linkage of progestin and epidermal growth factor signaling: phosphorylation of progesterone receptors mediates transcriptional hypersensitivity and increased ligand-independent breast cancer cell growth.Upregulation of RASGRP3 expression in prostate cancer correlates with aggressive capabilities and predicts biochemical recurrence after radical prostatectomyERBB2 increases metastatic potentials specifically in androgen-insensitive prostate cancer cells.Nemo-like kinase induces apoptosis and inhibits androgen receptor signaling in prostate cancer cellsPolyisoprenylated methylated protein methyl esterase as a putative drug target for androgen-insensitive prostate cancer.RasGRP3 contributes to formation and maintenance of the prostate cancer phenotype.Androgen receptor phosphorylation and stabilization in prostate cancer by cyclin-dependent kinase 1.Gene expression profiling in human high-grade astrocytomas.Androgen receptor remains critical for cell-cycle progression in androgen-independent CWR22 prostate cancer cellsGene signatures ESC, MYC and ERG-fusion are early markers of a potentially dangerous subtype of prostate cancer.Mechanisms mediating androgen receptor reactivation after castration.Plasma membrane-associated sialidase (NEU3) regulates progression of prostate cancer to androgen-independent growth through modulation of androgen receptor signalingMechanisms of acquired androgen independence during arsenic-induced malignant transformation of human prostate epithelial cells.Antagonists of growth hormone-releasing hormone inhibit growth of androgen-independent prostate cancer through inactivation of ERK and Akt kinasesRegulation of chemoresistance via alternative messenger RNA splicingTarget gene-specific regulation of androgen receptor activity by p42/p44 mitogen-activated protein kinase.AKT upregulates B-Raf Ser445 phosphorylation and ERK1/2 activation in prostate cancer cells in response to androgen depletion.Ras signaling influences permissiveness of malignant peripheral nerve sheath tumor cells to oncolytic herpesNew insights into the functional mechanisms and clinical applications of the kallikrein-related peptidase family.Differential requirement for MEK Partner 1 in DU145 prostate cancer cell migration.Polycomb group protein enhancer of zeste 2 is an oncogene that promotes the neoplastic transformation of a benign prostatic epithelial cell lineHypoxia, notch signalling, and prostate cancer.Androgen action and metabolism in prostate cancer.Prostate cancer stem cells: deciphering the origins and pathways involved in prostate tumorigenesis and aggression.A glycolytic phenotype is associated with prostate cancer progression and aggressiveness: a role for monocarboxylate transporters as metabolic targets for therapy.RhoGDIα downregulates androgen receptor signaling in prostate cancer cells.Regulation of androgen receptor-dependent transcription by coactivator MED1 is mediated through a newly discovered noncanonical binding motif.Differential regulation of insulin-like growth factor-I receptor gene expression by wild type and mutant androgen receptor in prostate cancer cells.
P2860
Q24645668-569CB33C-227D-4F68-B762-E549216242DFQ24672410-BC129ECB-BA84-420C-B662-A9603BEF74F0Q24681479-FFAF4D43-AE3A-4A5E-9755-B2C2CBD73413Q24815477-103D1EFD-8EC2-468B-9094-D041859A28BCQ26774456-A7F8B642-A476-4797-9703-F198AE5B9459Q26795589-5641FB5F-3D4D-472A-94F1-4FE267D19C45Q26851631-C96E67D3-3B8E-475C-80BE-C2D6A09C2CFFQ28383469-0F9A7153-2D22-44B1-8785-52CDA36B9904Q30425261-867D1F30-65B7-4C0E-A8F1-70B93706D90EQ30434799-F12BF2D7-FFF8-451C-83CD-640DC6560141Q30435809-D6696A76-7FB9-4B7B-94C7-735DAD04C557Q30438487-334E5BC7-3B9C-4B6A-A695-5F0E565B25F9Q33266857-3E821E95-34AD-4785-910D-7E33C250C217Q33607833-806D1E7E-A00E-400B-94E5-3EEE0FB2AF99Q33768865-F5B26E7F-5921-48EC-A4C2-E16B8EABD641Q34012237-FBDA7105-CB06-4D11-BEFF-45ABEAE1F17FQ34135178-EF88D9A0-F30D-4BA3-838C-C412482C86C9Q34203989-A280D5B4-77CA-45F9-AA91-656463A35D30Q35127580-7BC1FB65-BDCD-4080-934F-7742A7996C9CQ35151550-4AAE17DC-2CDF-4402-938B-08CE5B588624Q35221988-CA0BEC93-EB4C-4328-B6AC-0D7327BB9CEEQ35223291-FA5139AB-4212-4438-A50C-6348DF443EC8Q35633454-7E42D21A-4069-44BB-8A14-B63529EBE714Q35657424-FF96DAE1-8F8A-464B-8E5D-DA2781100DCFQ35672221-E55BF0BB-800F-4D9D-AAA9-5ABDDD504EF5Q35749794-B185B901-5D8E-4E8C-B43B-F9817CF481C8Q35822883-53CD9786-3C65-4301-9354-D0C7B6A63FC1Q36971853-0F441930-165B-44C8-800E-E1A80E8EB3E7Q36977833-F43F7FD1-100B-4C0D-B6F3-952C313E0681Q37061120-AD676B7E-DDC4-4B2C-A471-6D56D103750EQ37453804-77D2E340-8356-4A28-B4A7-AB22AE319AA7Q37458788-125E9C76-A173-41FD-83B6-7915953B40E9Q37478998-FECA6613-28FC-4793-A742-4F2CCA7E9B84Q37589392-C7890F33-8DDC-48D8-A112-28E950AA94BAQ37997571-5FBE850E-F433-4500-8B8B-967804E2A6A2Q38321512-2F81A350-3515-4144-9AF9-362AD0DF0F69Q38886986-DEC77561-7966-4475-80DB-F215AECD4C67Q39114221-3AB4CE68-29E1-47EF-86EF-51865BF83BFAQ39439753-C3BD0905-272A-4AA8-91E4-1CC85BC6CE53Q39711107-803EBDA2-E189-4635-B598-F278234D1040
P2860
Constitutive activation of the Ras/mitogen-activated protein kinase signaling pathway promotes androgen hypersensitivity in LNCaP prostate cancer cells.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Constitutive activation of the ...... n LNCaP prostate cancer cells.
@ast
Constitutive activation of the ...... n LNCaP prostate cancer cells.
@en
type
label
Constitutive activation of the ...... n LNCaP prostate cancer cells.
@ast
Constitutive activation of the ...... n LNCaP prostate cancer cells.
@en
prefLabel
Constitutive activation of the ...... n LNCaP prostate cancer cells.
@ast
Constitutive activation of the ...... n LNCaP prostate cancer cells.
@en
P2093
P1433
P1476
Constitutive activation of the ...... n LNCaP prostate cancer cells.
@en
P2093
Daniel Gioeli
Eric A Bissonette
Michael J Weber
Robert A Sikes
Robert E Bakin
P304
P407
P577
2003-04-01T00:00:00Z