A transforming growth factor-beta control element required for SM alpha-actin expression in vivo also partially mediates GKLF-dependent transcriptional repression.
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Krüppel-like factors 4 and 5: the yin and yang regulators of cellular proliferationMicroRNA-145, a novel smooth muscle cell phenotypic marker and modulator, controls vascular neointimal lesion formationThe Role of Krüppel-like Factor 4 in Renal FibrosisMicroRNA-1 regulates smooth muscle cell differentiation by repressing Kruppel-like factor 4Uniaxial mechanical strain modulates the differentiation of neural crest stem cells into smooth muscle lineage on micropatterned surfacesKrüppel-like factor 4 inhibits proliferation by platelet-derived growth factor receptor beta-mediated, not by retinoic acid receptor alpha-mediated, phosphatidylinositol 3-kinase and ERK signaling in vascular smooth muscle cellsPIAS1 activates the expression of smooth muscle cell differentiation marker genes by interacting with serum response factor and class I basic helix-loop-helix proteins.Kruppel-like factor 4, Elk-1, and histone deacetylases cooperatively suppress smooth muscle cell differentiation markers in response to oxidized phospholipids.Gut-enriched Krüppel-like factor interaction with Smad3 inhibits myofibroblast differentiationWhole animal knockout of smooth muscle alpha-actin does not alter excisional wound healing or the fibroblast-to-myofibroblast transitionCooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.Interleukin-1β modulates smooth muscle cell phenotype to a distinct inflammatory state relative to PDGF-DD via NF-κB-dependent mechanisms.PIAS1 mediates TGFbeta-induced SM alpha-actin gene expression through inhibition of KLF4 function-expression by protein sumoylationSmooth and cardiac muscle-selective knock-out of Kruppel-like factor 4 causes postnatal death and growth retardation.5' CArG degeneracy in smooth muscle alpha-actin is required for injury-induced gene suppression in vivo.The RhoA activator GEF-H1/Lfc is a transforming growth factor-beta target gene and effector that regulates alpha-smooth muscle actin expression and cell migrationSp1-dependent activation of KLF4 is required for PDGF-BB-induced phenotypic modulation of smooth muscle.PDGF-DD, a novel mediator of smooth muscle cell phenotypic modulation, is upregulated in endothelial cells exposed to atherosclerosis-prone flow patternsControl of SRF binding to CArG box chromatin regulates smooth muscle gene expression in vivo.Conditional deletion of Krüppel-like factor 4 delays downregulation of smooth muscle cell differentiation markers but accelerates neointimal formation following vascular injuryMultiple repressor pathways contribute to phenotypic switching of vascular smooth muscle cells.Regulation of alpha-smooth muscle actin expression in granulation tissue myofibroblasts is dependent on the intronic CArG element and the transforming growth factor-beta1 control element.Protein inhibitor of activated STAT1 interacts with and up-regulates activities of the pro-proliferative transcription factor Krüppel-like factor 5.Gene expression profiles in rat mesenteric lymph nodes upon supplementation with conjugated linoleic acid during gestation and sucklingKLF3 regulates muscle-specific gene expression and synergizes with serum response factor on KLF binding sitesKLF4 promotes hydrogen-peroxide-induced apoptosis of chronic myeloid leukemia cells involving the bcl-2/bax pathwayMolecular regulation of contractile smooth muscle cell phenotype: implications for vascular tissue engineering.Generation of mice deficient in both KLF3/BKLF and KLF8 reveals a genetic interaction and a role for these factors in embryonic globin gene silencingKLF11-mediated repression antagonizes Sp1/sterol-responsive element-binding protein-induced transcriptional activation of caveolin-1 in response to cholesterol signaling.Induction of KLF4 in response to heat stress.Cell contact-dependent regulation of epithelial-myofibroblast transition via the rho-rho kinase-phospho-myosin pathway.Derivation of smooth muscle cells with neural crest origin from human induced pluripotent stem cells.Tough beginnings: alterations in the transcriptome of cloned embryos during the first two cell cyclesTransforming growth factor-β and smooth muscle differentiationErythroid Krüppel-like factor directly activates the basic Krüppel-like factor gene in erythroid cells.miR-145 regulates myofibroblast differentiation and lung fibrosis.Smooth muscle alpha-actin expression and myofibroblast differentiation by TGFbeta are dependent upon MK2.Transcriptional regulation of SM22alpha by Wnt3a: convergence with TGFbeta(1)/Smad signaling at a novel regulatory element.Roles of Krüpel-like factor 4 in normal homeostasis, cancer and stem cells.Upregulation of the constitutively expressed HSC70 by KLF4.
P2860
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P2860
A transforming growth factor-beta control element required for SM alpha-actin expression in vivo also partially mediates GKLF-dependent transcriptional repression.
description
2003 nî lūn-bûn
@nan
2003 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
A transforming growth factor-b ...... nt transcriptional repression.
@ast
A transforming growth factor-b ...... nt transcriptional repression.
@en
type
label
A transforming growth factor-b ...... nt transcriptional repression.
@ast
A transforming growth factor-b ...... nt transcriptional repression.
@en
prefLabel
A transforming growth factor-b ...... nt transcriptional repression.
@ast
A transforming growth factor-b ...... nt transcriptional repression.
@en
P356
P1476
A transforming growth factor-b ...... nt transcriptional repression.
@en
P2093
P304
48004-48011
P356
10.1074/JBC.M301902200
P407
P50
P577
2003-09-10T00:00:00Z