Hypothesis of K+-Recycling Defect Is Not a Primary Deafness Mechanism for Cx26 (GJB2) Deficiency
about
A deafness mechanism of digenic Cx26 (GJB2) and Cx30 (GJB6) mutations: Reduction of endocochlear potential by impairment of heterogeneous gap junctional function in the cochlear lateral wall.Progressive age-dependence and frequency difference in the effect of gap junctions on active cochlear amplification and hearing.DFNB1 Non-syndromic Hearing Impairment: Diversity of Mutations and Associated Phenotypes.
P2860
Hypothesis of K+-Recycling Defect Is Not a Primary Deafness Mechanism for Cx26 (GJB2) Deficiency
description
2017 nî lūn-bûn
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2017 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2017 թվականի մայիսին հրատարակված գիտական հոդված
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2017年の論文
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2017年論文
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2017年論文
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2017年論文
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2017年論文
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2017年論文
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2017年论文
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name
Hypothesis of K+-Recycling Def ...... ism for Cx26 (GJB2) Deficiency
@ast
Hypothesis of K+-Recycling Def ...... ism for Cx26 (GJB2) Deficiency
@en
type
label
Hypothesis of K+-Recycling Def ...... ism for Cx26 (GJB2) Deficiency
@ast
Hypothesis of K+-Recycling Def ...... ism for Cx26 (GJB2) Deficiency
@en
prefLabel
Hypothesis of K+-Recycling Def ...... ism for Cx26 (GJB2) Deficiency
@ast
Hypothesis of K+-Recycling Def ...... ism for Cx26 (GJB2) Deficiency
@en
P2860
P356
P1476
Hypothesis of K+-Recycling Def ...... ism for Cx26 (GJB2) Deficiency
@en
P2093
Hong-Bo Zhao
P2860
P356
10.3389/FNMOL.2017.00162
P577
2017-05-26T00:00:00Z