Toll-like receptor 3 gene polymorphisms and severity of pandemic A/H1N1/2009 influenza in otherwise healthy children. - Wikidata - wikimedia - TriplyDB

Toll-like receptor 3 gene polymorphisms and severity of pandemic A/H1N1/2009 influenza in otherwise healthy children.

Toll-like receptor 3 gene polymorphisms and severity of pandemic A/H1N1/2009 influenza in otherwise healthy children.

http://www.wikidata.org/entity/Q30423584

about

Genetic variants in IL1A and IL1B contribute to the susceptibility to 2009 pandemic H1N1 influenza A virus.Host genetic determinants of influenza pathogenicity.Highly pathogenic avian influenza A H5N1 and pandemic H1N1 virus infections have different phenotypes in Toll-like receptor 3 knockout mice.Heterogeneous pathological outcomes after experimental pH1N1 influenza infection in ferrets correlate with viral replication and host immune responses in the lung.IFITM3, TLR3, and CD55 Genes SNPs and Cumulative Ge-netic Risks for Severe Outcomes in Chinese Patients with H7N9 / H1N1pdm09 Influenza.Innate immunity to influenza virus infection.Loss of TLR3 aggravates CHIKV replication and pathology due to an altered virus-specific neutralizing antibody response Poly(I:C) induces antiviral immune responses in Japanese flounder (Paralichthys olivaceus) that require TLR3 and MDA5 and is negatively regulated by Myd88.Proteomic analysis reveals down-regulation of surfactant protein B in murine type II pneumocytes infected with influenza A virus.Involvement of the different lung compartments in the pathogenesis of pH1N1 influenza virus infection in ferrets.--LUBAC deficiency perturbs TLR3 signaling to cause immunodeficiency and autoinflammation.The complex link between influenza and severe sepsis Toll-like receptor 3 in viral pathogenesis: friend or foe?The amazing innate immune response to influenza A virus infection.Baseline and Dynamic Expression of Activating NK Cell Receptors in the Control of Chronic Viral Infections: The Paradigm of HIV-1 and HCV.Cancer-associated toll-like receptor modulation and insinuation in infection susceptibility: association or coincidence?TRIF-dependent TLR signaling, its functions in host defense and inflammation, and its potential as a therapeutic target.Severe influenza in children: incidence and risk factors.Genetic polymorphisms and the development of invasive bacterial infections in children.Association between TLR4 A299G polymorphism and pneumonia risk: a meta-analysis.Single-walled carbon nanotubes modulate pulmonary immune responses and increase pandemic influenza a virus titers in mice.Association of gene polymorphisms of pattern-recognition receptor signaling pathway with the risk and severity of hand, foot, and mouth disease caused by enterovirus 71 in Chinese Han population.Correlation between TLR2, TLR3, TLR4, and TLR9 polymorphisms and susceptibility to and prognosis of severe hepatitis among the newborns.Phosphatidyl Inositol 3 Kinase-Gamma Balances Antiviral and Inflammatory Responses During Influenza A H1N1 Infection: From Murine Model to Genetic Association in Patients.Epidemiology and Immune Pathogenesis of Viral Sepsis

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P2860

Toll-like receptor 3 gene polymorphisms and severity of pandemic A/H1N1/2009 influenza in otherwise healthy children.

http://www.wikidata.org/entity/Q30423584

description

2012 nî lūn-bûn

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2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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Toll-like receptor 3 gene poly ...... in otherwise healthy children.

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Toll-like receptor 3 gene poly ...... in otherwise healthy children.

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Toll-like receptor 3 gene poly ...... in otherwise healthy children.

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Toll-like receptor 3 gene poly ...... in otherwise healthy children.

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Toll-like receptor 3 gene poly ...... in otherwise healthy children.

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Toll-like receptor 3 gene poly ...... in otherwise healthy children

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Alessandro Plebani

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Alessia Scala

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Cinzia Mazza

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Claudio Giuseppe Molteni

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Laura Tagliaferri

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Nicola Principi

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Silvia Giliani

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P2860

Toll-like receptor 3 associates with c-Src tyrosine kinase on endosomes to initiate antiviral signaling

Innate immune recognition of viral infection

Pathogen recognition and innate immunity

Autophagy-dependent viral recognition by plasmacytoid dendritic cells

Superior immunogenicity of inactivated whole virus H5N1 influenza vaccine is primarily controlled by Toll-like receptor signalling.

Augmented lung inflammation protects against influenza A pneumonia.

Oseltamivir-induced resistant pandemic A/H1N1 influenza virus in a child with cystic fibrosis and Pseudomonas aeruginosa infection.

Cross-protective immunity against influenza virus infections induced by intranasal vaccination together with a TLR3-mucosal adjuvant.

Toll-like receptor pre-stimulation protects mice against lethal infection with highly pathogenic influenza viruses.

Detrimental contribution of the Toll-like receptor (TLR)3 to influenza A virus-induced acute pneumonia

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1743-422x-9-270

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270

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10.1186/1743-422X-9-270

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9

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Susanna Esposito

Emilio F Fossali

Claudio Pelucchi

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2012-11-15T00:00:00Z

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1013715490

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Toll-like receptor

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