Role of TLR signaling in Francisella tularensis-LPS-induced, antibody-mediated protection against Francisella tularensis challenge. - Wikidata - wikimedia - TriplyDB

Role of TLR signaling in Francisella tularensis-LPS-induced, antibody-mediated protection against Francisella tularensis challenge.

Role of TLR signaling in Francisella tularensis-LPS-induced, antibody-mediated protection against Francisella tularensis challenge.

http://www.wikidata.org/entity/Q30424747

about

Immunotherapy for tularemia Nasal Acai polysaccharides potentiate innate immunity to protect against pulmonary Francisella tularensis and Burkholderia pseudomallei Infections Monophosphoryl Lipid A Enhances Efficacy of a Francisella tularensis LVS-Catanionic Nanoparticle Subunit Vaccine against F. tularensis Schu S4 Challenge by Augmenting both Humoral and Cellular Immunity.Novel catanionic surfactant vesicle vaccines protect against Francisella tularensis LVS and confer significant partial protection against F. tularensis Schu S4 strain B1b cells recognize protective antigens after natural infection and vaccination.A mucosal subunit vaccine protects against lethal respiratory infection with Francisella tularensis LVS.Production of anti-LPS IgM by B1a B cells depends on IL-1β and is protective against lung infection with Francisella tularensis LVS Antigen-specific memory in B-1a and its relationship to natural immunity.Kdo hydrolase is required for Francisella tularensis virulence and evasion of TLR2-mediated innate immunity.Temporal transcriptional response during infection of type II alveolar epithelial cells with Francisella tularensis live vaccine strain (LVS) supports a general host suppression and bacterial uptake by macropinocytosis.Interleukin-17 protects against the Francisella tularensis live vaccine strain but not against a virulent F. tularensis type A strain.Mitogen-activated protein kinases (MAPKs) are modulated during Francisella tularensis infection, but inhibition of extracellular-signal-regulated kinases (ERKs) is of limited therapeutic benefit TLR Agonists as Modulators of the Innate Immune Response and Their Potential as Agents Against Infectious Disease.Tularemia vaccines.Microinjection of Francisella tularensis and Listeria monocytogenes reveals the importance of bacterial and host factors for successful replication Modeling early events in Francisella tularensis pathogenesis.Hematopoietic MyD88 and IL-18 are essential for IFN-γ-dependent restriction of type A Francisella tularensis infection.Adaptive Immunity to Francisella tularensis and Considerations for Vaccine Development.

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Role of TLR signaling in Francisella tularensis-LPS-induced, antibody-mediated protection against Francisella tularensis challenge.

http://www.wikidata.org/entity/Q30424747

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2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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Role of TLR signaling in Franc ...... ncisella tularensis challenge.

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Role of TLR signaling in Franc ...... ncisella tularensis challenge.

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Role of TLR signaling in Franc ...... ncisella tularensis challenge.

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Role of TLR signaling in Franc ...... ncisella tularensis challenge.

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Role of TLR signaling in Franc ...... ncisella tularensis challenge.

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Journal of Leukocyte Biology

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Role of TLR signaling in Franc ...... ncisella tularensis challenge.

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P2093

Barbara J Mann

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Kari Ann Shirey

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Katharina Richard

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Kirsty L Chesko

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Leah E Cole

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Patricia J Gearhart

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Rose M Viscardi

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Stefanie N Vogel

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Yang Yang

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P2860

Neutralization of gamma interferon and tumor necrosis factor alpha blocks in vivo synthesis of nitrogen oxides from L-arginine and protection against Francisella tularensis infection in Mycobacterium bovis BCG-treated mice

Pyogenic bacterial infections in humans with IRAK-4 deficiency

The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5

Biological warfare. A historical perspective

Essential role of IRAK-4 protein and its kinase activity in Toll-like receptor-mediated immune responses but not in TCR signaling

Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme

Alternative activation of macrophages: mechanism and functions

The AIM2 inflammasome is essential for host defense against cytosolic bacteria and DNA viruses

The family of five: TIR-domain-containing adaptors in Toll-like receptor signalling

Enhancement of antigen-specific immunity via the TLR4 ligands MPL adjuvant and Ribi.529.

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787-797

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scholarly article

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10.1189/JLB.0111014

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4

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90

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2011-07-12T00:00:00Z

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51486612

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21750122

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Toll-like receptor

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3177696

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