B cell maturation antigen deficiency exacerbates lymphoproliferation and autoimmunity in murine lupus - Wikidata - wikimedia - TriplyDB

B cell maturation antigen deficiency exacerbates lymphoproliferation and autoimmunity in murine lupus

B cell maturation antigen deficiency exacerbates lymphoproliferation and autoimmunity in murine lupus

http://www.wikidata.org/entity/Q30426692

about

BAFF regulates follicular helper t cells and affects their accumulation and interferon-γ production in autoimmunity.Neutrophils contribute to excess serum BAFF levels and promote CD4+ T cell and B cell responses in lupus-prone mice.Mcl-1 is essential for the survival of plasma cells.Regulatory roles of the tumor necrosis factor receptor BCMA.A distinct plasmablast and naïve B-cell phenotype in primary immune thrombocytopenia.Prevention of immune nephritis by the small molecular weight immunomodulator iguratimod in MRL/lpr mice.TNF superfamily in inflammatory disease: translating basic insights.Inhibition of B cell activating factor (BAFF) in the management of systemic lupus erythematosus (SLE).Development of systemic lupus erythematosus in NZM 2328 mice in the absence of any single BAFF receptor.Clinical targeting of the TNF and TNFR superfamilies Therapeutic targeting of the BAFF/APRIL axis in systemic lupus erythematosus.The prospects and promise of chimeric antigen receptor immunotherapy in multiple myeloma.Zoom Zoom: racing CARs for multiple myeloma.Editorial: the BAFFling immunology of systemic lupus erythematosus: beyond B cells.Differential Development of Systemic Lupus Erythematosus in NZM 2328 Mice Deficient in Discrete Pairs of BAFF Receptors.Delayed onset of autoreactive antibody production and M2-skewed macrophages contribute to improved survival of TACI deficient MRL-Fas/Lpr mouse.Association of BAFF, APRIL serum levels, BAFF-R, TACI and BCMA expression on peripheral B-cell subsets with clinical manifestations in systemic lupus erythematosus.TACI-dependent APRIL signaling maintains autoreactive B cells in a mouse model of systemic lupus erythematosus.

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B cell maturation antigen deficiency exacerbates lymphoproliferation and autoimmunity in murine lupus

http://www.wikidata.org/entity/Q30426692

description

2011 nî lūn-bûn

@nan

2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

B cell maturation antigen defi ...... d autoimmunity in murine lupus

@ast

B cell maturation antigen defi ...... d autoimmunity in murine lupus

@en

type

label

B cell maturation antigen defi ...... d autoimmunity in murine lupus

@ast

B cell maturation antigen defi ...... d autoimmunity in murine lupus

@en

prefLabel

B cell maturation antigen defi ...... d autoimmunity in murine lupus

@ast

B cell maturation antigen defi ...... d autoimmunity in murine lupus

@en

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JIMMUNOL.1001931

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Journal of Immunology

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B cell maturation antigen defi ...... d autoimmunity in murine lupus

@en

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Chao Jiang

http://www.w3.org/2001/XMLSchema#string

Erin J Greenley

http://www.w3.org/2001/XMLSchema#string

Kenneth S Tung

http://www.w3.org/2001/XMLSchema#string

Loren D Erickson

http://www.w3.org/2001/XMLSchema#string

William M Loo

http://www.w3.org/2001/XMLSchema#string

P2860

TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease

Identification of a receptor for BLyS demonstrates a crucial role in humoral immunity

BAFF-R, a newly identified TNF receptor that specifically interacts with BAFF

BCMA is essential for the survival of long-lived bone marrow plasma cells

Development of murine lupus involves the combined genetic contribution of the SLAM and FcgammaR intervals within the Nba2 autoimmune susceptibility locus.

Deregulation of c-Myc Confers distinct survival requirements for memory B cells, plasma cells, and their progenitors

B-cell maturation protein, which binds the tumor necrosis factor family members BAFF and APRIL, is dispensable for humoral immune responses.

Myeloid cells, BAFF, and IFN-gamma establish an inflammatory loop that exacerbates autoimmunity in Lyn-deficient mice.

Loss of TACI causes fatal lymphoproliferation and autoimmunity, establishing TACI as an inhibitory BLyS receptor.

Similarities and differences between selective and nonselective BAFF blockade in murine SLE

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6136-6147

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scholarly article

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10.4049/JIMMUNOL.1001931

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11

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186

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2011-05-02T00:00:00Z

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51092836

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21536804

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3181142

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