Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection. - Wikidata - wikimedia - TriplyDB

Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

http://www.wikidata.org/entity/Q30432023

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TIM-3 Regulates Distinct Functions in Macrophages Role of Tim-3 in hepatitis B virus infection: An overview Beyond CTLA-4 and PD-1, the Generation Z of Negative Checkpoint Regulators Emerging and Novel Functions of Complement Protein C1q Dendritic cell-based immunity and vaccination against hepatitis C virus infection Interleukin 12: still a promising candidate for tumor immunotherapy?Tim-3 directly enhances CD8 T cell responses to acute Listeria monocytogenes infection Production and pathogenicity of hepatitis C virus core gene products.Synergistic effect of IL-12 and IL-18 induces TIM3 regulation of γδ T cell function and decreases the risk of clinical malaria in children living in Papua New Guinea Tim-3 and its role in regulating anti-tumor immunity.A randomized, double-blind, placebo-controlled assessment of BMS-936558, a fully human monoclonal antibody to programmed death-1 (PD-1), in patients with chronic hepatitis C virus infection.Cis association of galectin-9 with Tim-3 differentially regulates IL-12/IL-23 expressions in monocytes via TLR signaling Tim-3 protects decidual stromal cells from toll-like receptor-mediated apoptosis and inflammatory reactions and promotes Th2 bias at the maternal-fetal interface.Alcohol-induced miR-27a regulates differentiation and M2 macrophage polarization of normal human monocytes.Up-regulation of A20/ABIN1 contributes to inefficient M1 macrophage polarization during Hepatitis C virus infection.The Tim3-galectin 9 pathway induces antibacterial activity in human macrophages infected with Mycobacterium tuberculosis Differential regulation of interleukin-12 (IL-12)/IL-23 by Tim-3 drives T(H)17 cell development during hepatitis C virus infection.KLRG1 negatively regulates natural killer cell functions through the Akt pathway in individuals with chronic hepatitis C virus infection.HCV core protein binds to gC1qR to induce A20 expression and inhibit cytokine production through MAPKs and NF-κB signaling pathways.Tim-3: an activation marker and activation limiter of innate immune cells T cell immunoglobulin and mucin domain-containing molecule 3 on CD14+ monocytes serves as a novel biological marker for diabetes duration in type 2 diabetes mellitus.Enhanced virus-specific CD8+ T cell responses by Listeria monocytogenes-infected dendritic cells in the context of Tim-3 blockade TIM-3 as a Target for Cancer Immunotherapy and Mechanisms of Action.Hepatitis C virus-mediated modulation of cellular immunity.Immune exhaustion and immune senescence: two distinct pathways for HBV vaccine failure during HCV and/or HIV infection.Viral (hepatitis C virus, hepatitis B virus, HIV) persistence and immune homeostasis.Too much of a good thing? Tim-3 and TCR signaling in T cell exhaustion.Cancer stem cells and tumor-associated macrophages: a roadmap for multitargeting strategies.Human galectin-9 on the porcine cells affects the cytotoxic activity of M1-differentiated THP-1 cells through inducing a shift in M2-differentiated THP-1 cells.T-bet-mediated Tim-3 expression dampens monocyte function during chronic hepatitis C virus infection.Hepatitis A virus cellular receptor 2 (HAVCR2) is decreased with viral infection and regulates pro-labour mediators OA.MicroRNA-155 regulates interferon-γ production in natural killer cells via Tim-3 signalling in chronic hepatitis C virus infection.Tim-3 suppression combined with TLR3 activation enhances antiviral immune response in patients with chronic HCV infection.HCV-induced miR146a controls SOCS1/STAT3 and cytokine expression in monocytes to promote regulatory T-cell development.Plasma soluble Tim-3 emerges as an inhibitor in sepsis: sepsis contrary to membrane Tim-3 on monocytes.Tim-3 alters the balance of IL-12/IL-23 and drives TH17 cells: role in hepatitis B vaccine failure during hepatitis C infection.HCV-infected hepatocytes drive CD4+ CD25+ Foxp3+ regulatory T-cell development through the Tim-3/Gal-9 pathway.Role of A20 in interferon-α-mediated functional restoration of myeloid dendritic cells in patients with chronic hepatitis C.Lipoarabinomannan Decreases Galectin-9 Expression and Tumor Necrosis Factor Pathway in Macrophages Favoring Mycobacterium tuberculosis Intracellular Growth.Immune checkpoint inhibition: prospects for prevention and therapy of hepatocellular carcinoma.

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Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

http://www.wikidata.org/entity/Q30432023

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2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

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Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

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type

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Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

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Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

@en

prefLabel

Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

@ast

Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

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Tim-3 negatively regulates IL-12 expression by monocytes in HCV infection.

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Cheng J Ma

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Jia M Wang

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Jonathan P Moorman

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Xiao J Ji

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Xiao Y Wu

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Ying Zhang

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Zhan S Jia

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Zhi Q Yao

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P2860

Interaction between complement receptor gC1qR and hepatitis C virus core protein inhibits T-lymphocyte proliferation.

A crucial role for Kupffer cell-derived galectin-9 in regulation of T cell immunity in hepatitis C infection

Galectin-9 induces maturation of human monocyte-derived dendritic cells

Programmed death-1-induced interleukin-10 production by monocytes impairs CD4+ T cell activation during HIV infection

Dysregulated T cell expression of TIM3 in multiple sclerosis

Galectin-9/TIM-3 interaction regulates virus-specific primary and memory CD8 T cell response

Production of Infectious Hepatitis C Virus of Various Genotypes in Cell Cultures

Upregulation of PD-1 Expression on Circulating and Intrahepatic Hepatitis C Virus-Specific CD8+ T Cells Associated with Reversible Immune Dysfunction

High Level of PD-1 Expression on Hepatitis C Virus (HCV)-Specific CD8+ and CD4+ T Cells during Acute HCV Infection, Irrespective of Clinical Outcome

Differential regulation of SOCS-1 signalling in B and T lymphocytes by hepatitis C virus core protein

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