Altered intracellular signaling and reduced viability of Alzheimer's disease neuronal cybrids is reproduced by beta-amyloid peptide acting through receptor for advanced glycation end products (RAGE).
about
RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunctionMay "mitochondrial eve" and mitochondrial haplogroups play a role in neurodegeneration and Alzheimer's disease?Cytoplasmic hybrid (cybrid) cell lines as a practical model for mitochondriopathiesDose-specific effect of simvastatin on hypoxia-induced HIF-1α and BACE expression in Alzheimer's disease cybrid cells.Receptor for advanced glycation end products is upregulated in optic neuropathy of Alzheimer's disease.Pinocembrin protects against β-amyloid-induced toxicity in neurons through inhibiting receptor for advanced glycation end products (RAGE)-independent signaling pathways and regulating mitochondrion-mediated apoptosis.Cell and animal models of mtDNA biology: progress and prospects.Mitochondria and cell bioenergetics: increasingly recognized components and a possible etiologic cause of Alzheimer's disease.RAGE is a key cellular target for Abeta-induced perturbation in Alzheimer's disease.Receptor for advanced glycation end products: its role in Alzheimer's disease and other neurological diseases.Mild exposure of RIN-5F β-cells to human islet amyloid polypeptide aggregates upregulates antioxidant enzymes via NADPH oxidase-RAGE: an hormetic stimulus.RAGE: the beneficial and deleterious effects by diverse mechanisms of actions.Crosstalk between calcium, amyloid beta and the receptor for advanced glycation endproducts in Alzheimer's disease.Cell signaling and receptors in toxicity of advanced glycation end products (AGEs): α-dicarbonyls, radicals, oxidative stress and antioxidants.Microglia receptors and their implications in the response to amyloid β for Alzheimer's disease pathogenesis.Investigation of phosphoproteome in RAGE signaling.A Novel, Multi-Target Natural Drug Candidate, Matrine, Improves Cognitive Deficits in Alzheimer's Disease Transgenic Mice by Inhibiting Aβ Aggregation and Blocking the RAGE/Aβ Axis.Mitochondria, Cybrids, Aging, and Alzheimer's Disease.Bioenergetic flux, mitochondrial mass and mitochondrial morphology dynamics in AD and MCI cybrid cell lines.Progress in the development of new drugs in Alzheimer's disease.Mitochondria and Mitochondrial Cascades in Alzheimer's Disease.Amyloid-Beta and Phosphorylated Tau Accumulations Cause Abnormalities at Synapses of Alzheimer's disease Neurons.AGE-RAGE in multiple sclerosis brain.
P2860
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P2860
Altered intracellular signaling and reduced viability of Alzheimer's disease neuronal cybrids is reproduced by beta-amyloid peptide acting through receptor for advanced glycation end products (RAGE).
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Altered intracellular signalin ...... glycation end products (RAGE).
@ast
Altered intracellular signalin ...... glycation end products (RAGE).
@en
type
label
Altered intracellular signalin ...... glycation end products (RAGE).
@ast
Altered intracellular signalin ...... glycation end products (RAGE).
@en
prefLabel
Altered intracellular signalin ...... glycation end products (RAGE).
@ast
Altered intracellular signalin ...... glycation end products (RAGE).
@en
P2093
P1476
Altered intracellular signalin ...... glycation end products (RAGE).
@en
P2093
Isaac G Onyango
James P Bennett
Jeremy B Tuttle
P304
P356
10.1016/J.MCN.2005.02.012
P577
2005-06-01T00:00:00Z