Elevation of intracellular calcium in smooth muscle causes endothelial cell generation of NO in arterioles. - Wikidata - wikimedia - TriplyDB

Elevation of intracellular calcium in smooth muscle causes endothelial cell generation of NO in arterioles.

Elevation of intracellular calcium in smooth muscle causes endothelial cell generation of NO in arterioles.

http://www.wikidata.org/entity/Q30454372

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Intravenous and gastric cerium dioxide nanoparticle exposure disrupts microvascular smooth muscle signaling Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?A xanthine-based KMUP-1 with cyclic GMP enhancing and K(+) channels opening activities in rat aortic smooth muscle Characterization of the thoracodorsal artery: morphology and reactivity.Two functionally distinct pools of eNOS in endothelium are facilitated by myoendothelial junction lipid composition Hemoglobin α/eNOS coupling at myoendothelial junctions is required for nitric oxide scavenging during vasoconstriction.Endothelial cell-specific knockout of connexin 43 causes hypotension and bradycardia in mice.Endothelial cell signaling during conducted vasomotor responses.Integration and Modulation of Intercellular Signaling Underlying Blood Flow Control The myoendothelial junction: connections that deliver the message.Regulation of cellular communication by signaling microdomains in the blood vessel wall.Interaction between nitric oxide signaling and gap junctions: effects on vascular function Endothelial cell expression of haemoglobin α regulates nitric oxide signalling Compartmentalized connexin 43 s-nitrosylation/denitrosylation regulates heterocellular communication in the vessel wall.Connexins: gaps in our knowledge of vascular function.Biological and biophysical properties of vascular connexin channels.Connexins in vascular physiology and pathology.Localized expression of an Ins(1,4,5)P3 receptor at the myoendothelial junction selectively regulates heterocellular Ca2+ communication.Connexin40 and connexin43 in mouse aortic endothelium: evidence for coordinated regulation.The myoendothelial junction: breaking through the matrix?Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries.Enhanced spontaneous Ca2+ events in endothelial cells reflect signalling through myoendothelial gap junctions in pressurized mesenteric arteries Endothelial Ca2+ wavelets and the induction of myoendothelial feedback.Coordination of intercellular Ca(2+) signaling in endothelial cell tubes of mouse resistance arteries.Real-time vascular mechanosensation through ex vivo artery perfusion.Transdifferentiation of human endothelial progenitors into smooth muscle cells TP receptor-mediated vasoconstriction in microperfused afferent arterioles: roles of O(2)(-) and NO.Electrical activation of endothelium evokes vasodilation and hyperpolarization along hamster feed arteries.Nitric oxide-mediated arteriolar dilation after endothelial deformation.Simultaneous arterial calcium dynamics and diameter measurements: application to myoendothelial communication.Endothelial function is impaired in conduit arteries of pannexin1 knockout mice.Bifidobacterium pseudocatenulatum CECT 7765 supplementation restores altered vascular function in an experimental model of obese mice 5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation.VEGF-mediated fusion in the generation of uniluminal vascular spheroids Ca2+ dynamics in a population of smooth muscle cells: modeling the recruitment and synchronization Role of the endothelium on arterial vasomotion.The nanostructure of myoendothelial junctions contributes to signal rectification between endothelial and vascular smooth muscle cells Cell-cell communication in the vessel wall.Invited review: arteriolar smooth muscle mechanotransduction: Ca(2+) signaling pathways underlying myogenic reactivity.Membrane hyperpolarization is not required for sustained muscarinic agonist-induced increases in intracellular Ca2+ in arteriolar endothelial cells

P2860

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P2860

Elevation of intracellular calcium in smooth muscle causes endothelial cell generation of NO in arterioles.

http://www.wikidata.org/entity/Q30454372

description

1997 nî lūn-bûn

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1997 թուականի Յունիսին հրատարակուած գիտական յօդուած

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1997 թվականի հունիսին հրատարակված գիտական հոդված

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1997年の論文

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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Elevation of intracellular cal ...... eneration of NO in arterioles.

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Elevation of intracellular cal ...... eneration of NO in arterioles.

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Elevation of intracellular cal ...... eneration of NO in arterioles.

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Elevation of intracellular cal ...... eneration of NO in arterioles.

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Elevation of intracellular cal ...... eneration of NO in arterioles.

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Elevation of intracellular cal ...... eneration of NO in arterioles.

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Elevation of intracellular cal ...... eneration of NO in arterioles.

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B R Duling

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K A Dora

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M P Doyle

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A new generation of Ca2+ indicators with greatly improved fluorescence properties

Identification of epoxyeicosatrienoic acids as endothelium-derived hyperpolarizing factors

Access of blood-borne vasoconstrictors to the arteriolar smooth muscle.

Arteriolar smooth muscle responses are modulated by an intramural diffusion barrier.

Dye tracers define differential endothelial and smooth muscle coupling patterns within the arteriolar wall.

Connexin 43 and connexin 40 gap junctional proteins are present in arteriolar smooth muscle and endothelium in vivo.

Comparison of conduit vessel and resistance vessel reactivity: influence of intimal permeability.

Electromechanical coupling and the conducted vasomotor response.

Cellular pathways of the conducted electrical response in arterioles of hamster cheek pouch in vitro.

Acetylcholine induces conducted vasodilation by nitric oxide-dependent and -independent mechanisms.

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