Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries. - Wikidata - wikimedia - TriplyDB

Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries.

Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries.

http://www.wikidata.org/entity/Q30485571

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Specialized Functional Diversity and Interactions of the Na,K-ATPase Diet-induced obesity impairs endothelium-derived hyperpolarization via altered potassium channel signaling mechanisms Na+/Ca2+ exchange and Na+/K+-ATPase in the heart Characterization of the thoracodorsal artery: morphology and reactivity.Endoplasmic reticulum-mediated signalling in cellular microdomains.Hemoglobin α/eNOS coupling at myoendothelial junctions is required for nitric oxide scavenging during vasoconstriction.Integration and Modulation of Intercellular Signaling Underlying Blood Flow Control The myoendothelial junction: connections that deliver the message.Regulation of cellular communication by signaling microdomains in the blood vessel wall.Site-specific connexin phosphorylation is associated with reduced heterocellular communication between smooth muscle and endothelium.Localized expression of an Ins(1,4,5)P3 receptor at the myoendothelial junction selectively regulates heterocellular Ca2+ communication.The myoendothelial junction: breaking through the matrix?Low intravascular pressure activates endothelial cell TRPV4 channels, local Ca2+ events, and IKCa channels, reducing arteriolar tone The PPARbeta/delta agonist GW0742 relaxes pulmonary vessels and limits right heart hypertrophy in rats with hypoxia-induced pulmonary hypertension.Down-regulation of KCa2.3 channels causes erectile dysfunction in mice Intermediate-conductance calcium-activated potassium channels participate in neurovascular coupling.Recycling of the Ca2+-activated K+ channel, KCa2.3, is dependent upon RME-1, Rab35/EPI64C, and an N-terminal domain Protection of coronary endothelial function during cardiac surgery: potential of targeting endothelial ion channels in cardioprotection The nanostructure of myoendothelial junctions contributes to signal rectification between endothelial and vascular smooth muscle cells Positive feedback regulation of agonist-stimulated endothelial Ca2+ dynamics by KCa3.1 channels in mouse mesenteric arteries.Upregulation of SK3 and IK1 channels contributes to the enhanced endothelial calcium signaling and the preserved coronary relaxation in obese Zucker rats.Impairment of both nitric oxide-mediated and EDHF-type relaxation in small mesenteric arteries from rats with streptozotocin-induced diabetes.Gap junctions suppress electrical but not [Ca(2+)] heterogeneity in resistance arteries Type 2 Diabetes: Endothelial dysfunction and Exercise.Robust internal elastic lamina fenestration in skeletal muscle arteries.EDHF: spreading the influence of the endothelium.The endothelium: influencing vascular smooth muscle in many ways.Fundamental increase in pressure-dependent constriction of brain parenchymal arterioles from subarachnoid hemorrhage model rats due to membrane depolarization.Activation of endothelial transient receptor potential C3 channel is required for small conductance calcium-activated potassium channel activation and sustained endothelial hyperpolarization and vasodilation of cerebral artery.Control of muscle blood flow during exercise: local factors and integrative mechanisms Stochastic model of endothelial TRPV4 calcium sparklets: effect of bursting and cooperativity on EDH.Intercellular communication in the vascular wall: a modeling perspective The calcium-sensing receptor and calcimimetics in blood pressure modulation Vascular Protection Following Cerebral Ischemia and Reperfusion.Microvascular responsiveness in obesity: implications for therapeutic intervention.Therapeutic potential of KCa3.1 blockers: recent advances and promising trends.Role of reactive oxygen and nitrogen species in the vascular responses to inflammation.Mechanisms involved in the aging-induced vascular dysfunction.Inactivation of Endothelial Small/Intermediate Conductance of Calcium-Activated Potassium Channels Contributes to Coronary Arteriolar Dysfunction in Diabetic Patients.Electrical conduction along endothelial cell tubes from mouse feed arteries: confounding actions of glycyrrhetinic acid derivatives.

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P2860

Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries.

http://www.wikidata.org/entity/Q30485571

description

2008 nî lūn-bûn

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2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի ապրիլին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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Modulation of endothelial cell ...... esenteric resistance arteries.

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Modulation of endothelial cell ...... esenteric resistance arteries.

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Modulation of endothelial cell ...... esenteric resistance arteries.

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Modulation of endothelial cell ...... esenteric resistance arteries.

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CIRCRESAHA.108.172379

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Circulation Research

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Modulation of endothelial cell ...... mesenteric resistance arteries

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Kim A Dora

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Nicola T Gallagher

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P2860

Blockade of the intermediate-conductance calcium-activated potassium channel as a new therapeutic strategy for restenosis

Suppression of K(+)-induced hyperpolarization by phenylephrine in rat mesenteric artery: relevance to studies of endothelium-derived hyperpolarizing factor

Rapid endothelial cell-selective loading of connexin 40 antibody blocks endothelium-derived hyperpolarizing factor dilation in rat small mesenteric arteries

Definitive role for natriuretic peptide receptor-C in mediating the vasorelaxant activity of C-type natriuretic peptide and endothelium-derived hyperpolarising factor

Ca2+ and inositol 1,4,5-trisphosphate-mediated signaling across the myoendothelial junction.

Elevation of intracellular calcium in smooth muscle causes endothelial cell generation of NO in arterioles.

Enhanced spontaneous Ca2+ events in endothelial cells reflect signalling through myoendothelial gap junctions in pressurized mesenteric arteries

Angiotensin II receptor blockade corrects altered expression of gap junctions in vascular endothelial cells from hypertensive rats.

EDHF: bringing the concepts together.

Extracellular calcium sensing and signalling.

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1247-1255

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10.1161/CIRCRESAHA.108.172379

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10

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102

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Alister J. McNeish

Christopher J Garland

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2008-04-10T00:00:00Z

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5448430

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