Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells. - Wikidata - wikimedia - TriplyDB

Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

http://www.wikidata.org/entity/Q30477570

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Cyclin B1-Cdk1 activation continues after centrosome separation to control mitotic progression Cyclin-dependent kinase 16/PCTAIRE kinase 1 is activated by cyclin Y and is essential for spermatogenesis A quantitative model for cyclin-dependent kinase control of the cell cycle: revisited Cohesin is dispensable for centromere cohesion in human cells The 3' untranslated region of the cyclin B mRNA is not sufficient to enhance the synthesis of cyclin B during a mitotic block in human cells Cellular contractility requires ubiquitin mediated proteolysis Cell cycle effects resulting from inhibition of hepatocyte growth factor and its receptor c-Met in regenerating rat livers by RNA interference The APC/C recruits cyclin B1-Cdk1-Cks in prometaphase before D box recognition to control mitotic exit Studies of haspin-depleted cells reveal that spindle-pole integrity in mitosis requires chromosome cohesion Spindly/CCDC99 is required for efficient chromosome congression and mitotic checkpoint regulation Cohesion fatigue induces chromatid separation in cells delayed at metaphase.Human chromokinesins promote chromosome congression and spindle microtubule dynamics during mitosis Alpha-santalol, a chemopreventive agent against skin cancer, causes G2/M cell cycle arrest in both p53-mutated human epidermoid carcinoma A431 cells and p53 wild-type human melanoma UACC-62 cells.Antineoplastic effects of α-santalol on estrogen receptor-positive and estrogen receptor-negative breast cancer cells through cell cycle arrest at G2/M phase and induction of apoptosis.Glypican-1 regulates anaphase promoting complex/cyclosome substrates and cell cycle progression in endothelial cells.Overexpression of the dynein light chain km23-1 in human ovarian carcinoma cells inhibits tumor formation in vivo and causes mitotic delay at prometaphase/metaphase.Kinetochore motors drive congression of peripheral polar chromosomes by overcoming random arm-ejection forces.Adenovirus replaces mitotic checkpoint controls.Inhibition of cell growth and induction of apoptosis in ovarian carcinoma cell lines CaOV3 and SKOV3 by natural withanolide Withaferin A DNA-PKcs Negatively Regulates Cyclin B1 Protein Stability through Facilitating Its Ubiquitination Mediated by Cdh1-APC/C Pathway.Reversal effects of Raloxifene on paclitaxel resistance in 2 MDR breast cancer cells.Greatwall kinase and cyclin B-Cdk1 are both critical constituents of M-phase-promoting factor.An integrated mechanobiochemical feedback mechanism describes chromosome motility from prometaphase to anaphase in mitosis.The multiple layers of ubiquitin-dependent cell cycle control.Mitotic p21Cip1/CDKN1A is regulated by cyclin-dependent kinase 1 phosphorylation.Cdk1 inactivation terminates mitotic checkpoint surveillance and stabilizes kinetochore attachments in anaphase.Less understood issues: p21(Cip1) in mitosis and its therapeutic potential.Research advances in HMGN5 and cancer.PP1 initiates the dephosphorylation of MASTL, triggering mitotic exit and bistability in human cells.Mechanisms regulating phosphatase specificity and the removal of individual phosphorylation sites during mitotic exit.Inefficient degradation of cyclin B1 re-activates the spindle checkpoint right after sister chromatid disjunction.A chromosome separation checkpoint: A midzone Aurora B gradient mediates a chromosome separation checkpoint that regulates the anaphase-telophase transition.p21Waf1/Cip1 deficiency causes multiple mitotic defects in tumor cells.Clusterin knockdown sensitizes prostate cancer cells to taxane by modulating mitosis.Connexin 43 enhances paclitaxel cytotoxicity in colorectal cancer cell lines.The BEG (PP2A-B55/ENSA/Greatwall) pathway ensures cytokinesis follows chromosome separation Evidence that mitotic exit is a better cancer therapeutic target than spindle assembly.Meiotic progression in Arabidopsis is governed by complex regulatory interactions between SMG7, TDM1, and the meiosis I-specific cyclin TAM.BubR1 is frequently repressed in acute myeloid leukemia and its re-expression sensitizes cells to antimitotic therapy Forkhead transcription factor FoxM1 regulates mitotic entry and prevents spindle defects in cerebellar granule neuron precursors.

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Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

http://www.wikidata.org/entity/Q30477570

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2006 nî lūn-bûn

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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2006 թվականի մայիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

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Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

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type

label

Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

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Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

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prefLabel

Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

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Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

@en

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SJ.EMBOJ.7601163

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The EMBO Journal

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Dose-dependent effects of stable cyclin B1 on progression through mitosis in human cells.

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Cornelia Wandke

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Frank Wolf

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Nina Isenberg

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Stephan Geley

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P2860

Shugoshin prevents dissociation of cohesin from centromeres during mitosis in vertebrate cells

Regulation of human separase by securin binding and autocleavage

Securin degradation is mediated by fzy and fzr, and is required for complete chromatid separation but not for cytokinesis

The human chromokinesin Kid is a plus end-directed microtubule-based motor

The chromokinesin Kid is necessary for chromosome arm orientation and oscillation, but not congression, on mitotic spindles

The Xenopus chromokinesin Xkid is essential for metaphase chromosome alignment and must be degraded to allow anaphase chromosome movement

Xkid, a chromokinesin required for chromosome alignment on the metaphase plate

Securin is required for chromosomal stability in human cells

The anaphase-promoting complex: proteolysis in mitosis and beyond

Anaphase-promoting complex/cyclosome-dependent proteolysis of human cyclin A starts at the beginning of mitosis and is not subject to the spindle assembly checkpoint

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