Central serotonergic neurons are differentially required for opioid analgesia but not for morphine tolerance or morphine reward.
about
Impaired emotional-like behavior and serotonergic function during protracted abstinence from chronic morphineUnidirectional cross-activation of GRPR by MOR1D uncouples itch and analgesia induced by opioids15 years of genetic approaches in vivo for addiction research: Opioid receptor and peptide gene knockout in mouse models of drug abuseThe atypical antidepressant mianserin exhibits agonist activity at κ-opioid receptorsActivation of the kappa opioid receptor in the dorsal raphe nucleus mediates the aversive effects of stress and reinstates drug seeking.Adenoviral vectors for highly selective gene expression in central serotonergic neurons reveal quantal characteristics of serotonin release in the rat brain.Inducible gene manipulations in brain serotonergic neurons of transgenic rats.Tetracycline inducible gene manipulation in serotonergic neurons.Sweet preferences and analgesia during childhood: effects of family history of alcoholism and depressionDistinct mu, delta, and kappa opioid receptor mechanisms underlie low sociability and depressive-like behaviors during heroin abstinence.Construction of a global pain systems network highlights phospholipid signaling as a regulator of heat nociception.Descending control of itch transmission by the serotonergic system via 5-HT1A-facilitated GRP-GRPR signalingSelective p38α MAPK deletion in serotonergic neurons produces stress resilience in models of depression and addiction.Molecular and cellular mechanisms that initiate pain and itch.Opioid receptors: distinct roles in mood disorders.Nociceptin/Orphanin FQ Receptor Structure, Signaling, Ligands, Functions, and Interactions with Opioid Systems.Genetically expressed transneuronal tracer reveals direct and indirect serotonergic descending control circuits.5-HT2A Serotonin Receptor Density in Adult Male Rats' Hippocampus after Morphine-based Conditioned Place PreferencePreclinical and early clinical investigations related to monoaminergic pain modulation.Migraine pain: reflections against vasodilatation.B-type natriuretic peptide is neither itch-specific nor functions upstream of the GRP-GRPR signaling pathwayUtility of genetically modified mice for understanding the neurobiology of substance use disorders.Cell fate determination, neuronal maintenance and disease state: The emerging role of transcription factors Lmx1a and Lmx1b.Availability of dopamine and serotonin transporters in opioid-dependent users--a two-isotope SPECT study.Distinct roles of NMB and GRP in itch transmission.Placebo analgesia affects the behavioral despair tests and hormonal secretions in mice.A central neural circuit for itch sensation.Serotonin neurones have anti-convulsant effects and reduce seizure-induced mortality.Additive analgesic effect of dexmedetomidine and dezocine administered intrathecally in a mouse pain model.
P2860
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P2860
Central serotonergic neurons are differentially required for opioid analgesia but not for morphine tolerance or morphine reward.
description
2007 nî lūn-bûn
@nan
2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Central serotonergic neurons a ...... tolerance or morphine reward.
@ast
Central serotonergic neurons a ...... tolerance or morphine reward.
@en
type
label
Central serotonergic neurons a ...... tolerance or morphine reward.
@ast
Central serotonergic neurons a ...... tolerance or morphine reward.
@en
prefLabel
Central serotonergic neurons a ...... tolerance or morphine reward.
@ast
Central serotonergic neurons a ...... tolerance or morphine reward.
@en
P2093
P2860
P356
P1476
Central serotonergic neurons a ...... tolerance or morphine reward.
@en
P2093
Cheng-Shui Zhao
Robert W Gereau
Yan-Gang Sun
Zhong-Qiu Zhao
Zhou-Feng Chen
P2860
P304
14519-14524
P356
10.1073/PNAS.0705740104
P407
P577
2007-08-27T00:00:00Z