about
Emerging targets in neuroinflammation-driven chronic painCentral serotonergic neurons are differentially required for opioid analgesia but not for morphine tolerance or morphine reward.Selective inhibition of JNK with a peptide inhibitor attenuates pain hypersensitivity and tumor growth in a mouse skin cancer pain modelActivation of extracellular signal-regulated kinase in the anterior cingulate cortex contributes to the induction and expression of affective pain.Toll-like receptor 4 contributes to chronic itch, alloknesis, and spinal astrocyte activation in male miceInduction of TNF-alpha by LPS in Schwann cell is regulated by MAPK activation signals.The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain conditionβ-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain.Chemokine CXCL1 enhances inflammatory pain and increases NMDA receptor activity and COX-2 expression in spinal cord neurons via activation of CXCR2.TRAF6 upregulation in spinal astrocytes maintains neuropathic pain by integrating TNF-α and IL-1β signalingShifts in cell-type expression accompany a diminishing role of spinal p38-mapkinase activation over time during prolonged postoperative pain.Intrathecal curcumin attenuates pain hypersensitivity and decreases spinal neuroinflammation in rat model of monoarthritis.Short small-interfering RNAs produce interferon-α-mediated analgesiaIdentification of lncRNA expression profile in the spinal cord of mice following spinal nerve ligation-induced neuropathic pain.Emerging role of Toll-like receptors in the control of pain and itch.TLR3 deficiency impairs spinal cord synaptic transmission, central sensitization, and pruritus in mice.Chemokine CCL2 and its receptor CCR2 in the medullary dorsal horn are involved in trigeminal neuropathic pain.CXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5.Involvement of P311 in the affective, but not in the sensory component of pain.Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of miceA long noncoding RNA contributes to neuropathic pain by silencing Kcna2 in primary afferent neurons.Activation of JNK pathway in persistent painJNK-induced MCP-1 production in spinal cord astrocytes contributes to central sensitization and neuropathic pain.Large A-fiber activity is required for microglial proliferation and p38 MAPK activation in the spinal cord: different effects of resiniferatoxin and bupivacaine on spinal microglial changes after spared nerve injury.Chemokine contribution to neuropathic pain: respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons.NFκB-mediated CXCL1 production in spinal cord astrocytes contributes to the maintenance of bone cancer pain in mice.Chemokines, neuronal-glial interactions, and central processing of neuropathic pain.Ligustilide inhibits microglia-mediated proinflammatory cytokines production and inflammatory pain.Chemokines in neuron-glial cell interaction and pathogenesis of neuropathic pain.Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic painCurcumin inhibits LPS-induced CCL2 expression via JNK pathway in C6 rat astrocytoma cells.The increase of intrinsic excitability of layer V pyramidal cells in the prelimbic medial prefrontal cortex of adult mice after peripheral inflammation.Ligustilide Ameliorates Inflammatory Pain and Inhibits TLR4 Upregulation in Spinal Astrocytes Following Complete Freund's Adjuvant Peripheral Injection.Increased autophagic activity in dorsal root ganglion attenuates neuropathic pain following peripheral nerve injury.Exogenous induction of HO-1 alleviates vincristine-induced neuropathic pain by reducing spinal glial activation in mice.Spinal injection of TNF-α-activated astrocytes produces persistent pain symptom mechanical allodynia by releasing monocyte chemoattractant protein-1Light touch induces ERK activation in superficial dorsal horn neurons after inflammation: involvement of spinal astrocytes and JNK signaling in touch-evoked central sensitization and mechanical allodynia.Involvement of spinal neurokinin-1 receptors in the maintenance but not induction of carrageenan-induced thermal hyperalgesia in the rat.Contributions of the anterior cingulate cortex and amygdala to pain- and fear-conditioned place avoidance in rats.NMDA receptors in the anterior cingulate cortex mediate pain-related aversion.
P50
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P50
description
onderzoeker
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researcher
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հետազոտող
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name
Yong-Jing Gao
@ast
Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
@nl
Yong-Jing Gao
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type
label
Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
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Yong-Jing Gao
@sl
P1053
E-6939-2015
P106
P31
P3829
P496
0000-0002-7432-7458