Induction of neostriatal neurogenesis slows disease progression in a transgenic murine model of Huntington disease - Wikidata - wikimedia - TriplyDB

Induction of neostriatal neurogenesis slows disease progression in a transgenic murine model of Huntington disease

Induction of neostriatal neurogenesis slows disease progression in a transgenic murine model of Huntington disease

http://www.wikidata.org/entity/Q30480065

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BDNF control of adult SVZ neurogenesis Epigenetic principles and mechanisms underlying nervous system functions in health and disease Mouse models of polyglutamine diseases in therapeutic approaches: review and data table. Part II.Amitriptyline improves motor function via enhanced neurotrophin signaling and mitochondrial functions in the murine N171-82Q Huntington disease model.Neurodegenerative disease and adult neurogenesis Mutant huntingtin alters cell fate in response to microtubule depolymerization via the GEF-H1-RhoA-ERK pathway Genome-wide loss of 5-hmC is a novel epigenetic feature of Huntington's disease Testosterone modulation of angiogenesis and neurogenesis in the adult songbird brain Hdac6 knock-out increases tubulin acetylation but does not modify disease progression in the R6/2 mouse model of Huntington's disease Neuropeptide Y (NPY) in cerebrospinal fluid from patients with Huntington's Disease: increased NPY levels and differential degradation of the NPY1-30 fragment.Human glia can both induce and rescue aspects of disease phenotype in Huntington disease.In Situ Pluripotency Factor Expression Promotes Functional Recovery From Cerebral Ischemia Impaired adult olfactory bulb neurogenesis in the R6/2 mouse model of Huntington's disease.Exercise is not beneficial and may accelerate symptom onset in a mouse model of Huntington's disease Use of Genetically Altered Stem Cells for the Treatment of Huntington's Disease.BDNF signaling and survival of striatal neurons.BDNF overexpression in the forebrain rescues Huntington's disease phenotypes in YAC128 mice In vivo reprogramming of astrocytes to neuroblasts in the adult brain.Electroconvulsive shock ameliorates disease processes and extends survival in huntingtin mutant mice.Advances in gene therapy for movement disorders.Neonatal hypoxic/ischemic brain injury induces production of calretinin-expressing interneurons in the striatum Gene therapy in mouse models of huntington disease.Therapeutic application of neural stem cells and adult neurogenesis for neurodegenerative disorders: regeneration and beyond Stem cells for cell replacement therapy: a therapeutic strategy for HD?Conditional BDNF release under pathological conditions improves Huntington's disease pathology by delaying neuronal dysfunction.HD iPSC-derived neural progenitors accumulate in culture and are susceptible to BDNF withdrawal due to glutamate toxicity.Cellular therapy and induced neuronal replacement for Huntington's disease.Effects of neuroinflammation on the regenerative capacity of brain stem cells Rapid transgene expression in multiple precursor cell types of adult rat subventricular zone mediated by adeno-associated type 1 vectors.Potential drugs and methods for preventing or delaying the progression of Huntington's disease.Extensive changes in DNA methylation are associated with expression of mutant huntingtin.Increased Olfactory Bulb BDNF Expression Does Not Rescue Deficits in Olfactory Neurogenesis in the Huntington's Disease R6/2 Mouse.Perivascular instruction of cell genesis and fate in the adult brain.Striatal oligodendrogliogenesis and neuroblast recruitment are increased in the R6/2 mouse model of Huntington's disease Neural stem cells confer unique pinwheel architecture to the ventricular surface in neurogenic regions of the adult brain.Cell migration in the normal and pathological postnatal mammalian brain.Stem and Progenitor Cell-Based Therapy of the Central Nervous System: Hopes, Hype, and Wishful Thinking.Molecular mechanisms and potential therapeutical targets in Huntington's disease.Integration and maturation of newborn neurons in the adult olfactory bulb--from synapses to function.Strategies for regenerating striatal neurons in the adult brain by using endogenous neural stem cells

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Induction of neostriatal neurogenesis slows disease progression in a transgenic murine model of Huntington disease

http://www.wikidata.org/entity/Q30480065

description

2007 nî lūn-bûn

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2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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Induction of neostriatal neuro ...... ne model of Huntington disease

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Induction of neostriatal neuro ...... ne model of Huntington disease

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Journal of Clinical Investigation

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Induction of neostriatal neuro ...... ne model of Huntington disease

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Abdellatif Benraiss

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Amer Samdani

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Eva Chmielnicki

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Steven A Goldman

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Sung-Rae Cho

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P2860

The Spemann organizer signal noggin binds and inactivates bone morphogenetic protein 4

Neuronal production, migration, and differentiation in a vocal control nucleus of the adult female canary brain

Doublecortin is a microtubule-associated protein and is expressed widely by migrating neurons

Huntingtin interacts with REST/NRSF to modulate the transcription of NRSE-controlled neuronal genes

Loss of huntingtin-mediated BDNF gene transcription in Huntington's disease

Heparan sulfate proteoglycans retain Noggin at the cell surface: a potential mechanism for shaping bone morphogenetic protein gradients

Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

Changes in cortical and striatal neurons predict behavioral and electrophysiological abnormalities in a transgenic murine model of Huntington's disease

Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain

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10.1172/JCI31778

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10

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117

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Aris Economides

Steven A. Goldman

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2007-10-01T00:00:00Z

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17885687

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Huntington disease

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1978427

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