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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

http://www.wikidata.org/entity/Q30482779

about

Altered longevity-assurance activity of p53:p44 in the mouse causes memory loss, neurodegeneration and premature death Emerging Link between Alzheimer's Disease and Homeostatic Synaptic Plasticity From FMRP function to potential therapies for fragile X syndrome.DBA/2J genetic background exacerbates spontaneous lethal seizures but lessens amyloid deposition in a mouse model of Alzheimer's disease.Soy-based diet exacerbates seizures in mouse models of neurological disease Alzheimer's disease and Down syndrome rodent models exhibit audiogenic seizures.Altered theta oscillations and aberrant cortical excitatory activity in the 5XFAD model of Alzheimer's disease.Probing the biology of Alzheimer's disease in mice.Abnormal intracellular accumulation and extracellular Aβ deposition in idiopathic and Dup15q11.2-q13 autism spectrum disorders Fragile X mental retardation protein expression in Alzheimer's disease Fragile X Syndrome and Alzheimer's Disease: Another story about APP and beta-amyloid Autism, Alzheimer disease, and fragile X: APP, FMRP, and mGluR5 are molecular links.Early onset of hypersynchronous network activity and expression of a marker of chronic seizures in the Tg2576 mouse model of Alzheimer's disease.Neurologic and motor dysfunctions in APP transgenic mice.Soluble Aβ oligomers impair hippocampal LTP by disrupting glutamatergic/GABAergic balance.Seizure-Induced Regulations of Amyloid-β, STEP61, and STEP61 Substrates Involved in Hippocampal Synaptic Plasticity.Finding novel distinctions between the sAPPα-mediated anabolic biochemical pathways in Autism Spectrum Disorder and Fragile X Syndrome plasma and brain tissue Overexpression of Hsp27 ameliorates symptoms of Alzheimer's disease in APP/PS1 mice.A novel nicotinic mechanism underlies β-amyloid-induced neuronal hyperexcitation.MPEP reduces seizure severity in Fmr-1 KO mice over expressing human Abeta Positive and negative early life experiences differentially modulate long term survival and amyloid protein levels in a mouse model of Alzheimer's disease.APP Causes Hyperexcitability in Fragile X Mice.Amyloid precursor protein modulates Nav1.6 sodium channel currents through a Go-coupled JNK pathway.Taking STEPs forward to understand fragile X syndrome.Genetic suppression of transgenic APP rescues Hypersynchronous network activity in a mouse model of Alzeimer's disease.Copper-uptake is critical for the down regulation of synapsin and dynamin induced by neocuproine: modulation of synaptic activity in hippocampal neurons.Cognition and dementia in older patients with epilepsy.

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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

http://www.wikidata.org/entity/Q30482779

description

2008 nî lūn-bûn

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2008 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հունվարին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

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International Journal of Clinical and Experimental Pathology

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Seizure susceptibility and mortality in mice that over-express amyloid precursor protein

@en

P2093

Ashley M Beard

http://www.w3.org/2001/XMLSchema#string

Cara J Westmark

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James S Malter

http://www.w3.org/2001/XMLSchema#string

Pamela R Westmark

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Sharon M Hildebrandt

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P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Homo- and heterodimerization of APP family members promotes intercellular adhesion.

Selective localization of amyloid precursor-like protein 1 in the cerebral cortex postsynaptic density

GABAA receptor beta 1, beta 2, and beta 3 subunits: comparisons in DBA/2J and C57BL/6J mice

Amyloid precursor protein is localized in growing neurites of neonatal rat brain.

FMRP mediates mGluR5-dependent translation of amyloid precursor protein.

Neurobehavioral characterization of APP23 transgenic mice with the SHIRPA primary screen.

Seizure disorders in the elderly.

Expression in brain of amyloid precursor protein mutated in the alpha-secretase site causes disturbed behavior, neuronal degeneration and premature death in transgenic mice.

A focus on the synapse for neuroprotection in Alzheimer disease and other dementias.

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157-168

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2008-01-01T00:00:00Z

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18784809

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2480559

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