Acetylation-dependent interaction of SATB1 and CtBP1 mediates transcriptional repression by SATB1.
about
The structural basis for the oligomerization of the N-terminal domain of SATB1Regulation of DNA replication timing on human chromosome by a cell-type specific DNA binding protein SATB1Global regulator SATB1 recruits beta-catenin and regulates T(H)2 differentiation in Wnt-dependent manner.Effect of dietary restriction and subsequent re-alimentation on the transcriptional profile of bovine ruminal epithelium.Genome organizing function of SATB1 in tumor progression.Phosphorylation-dependent interaction of SATB1 and PIAS1 directs SUMO-regulated caspase cleavage of SATB1.Crystal structure of the ubiquitin-like domain-CUT repeat-like tandem of special AT-rich sequence binding protein 1 (SATB1) reveals a coordinating DNA-binding mechanism.C-Terminal Binding Protein: A Molecular Link between Metabolic Imbalance and Epigenetic Regulation in Breast Cancer.Plakoglobin represses SATB1 expression and decreases in vitro proliferation, migration and invasion.Expression of SATB1 and HER2 in breast cancer and the correlations with clinicopathologic characteristics.SIRT1 deacetylates SATB1 to facilitate MAR HS2-MAR ε interaction and promote ε-globin expression.ConteXt of change--X inactivation and disease.Upregulation of SATB1 is associated with the development and progression of glioma.SATB1 and SATB2 play opposing roles in c-Myc expression and progression of colorectal cancer.SATB1 is overexpressed in metastatic prostate cancer and promotes prostate cancer cell growth and invasion.X inactivation and progenitor cancer cellsOver-expression of the special AT rich sequence binding protein 1 (SATB1) promotes the progression of nasopharyngeal carcinoma: association with EBV LMP-1 expression.Satb1 and Satb2 regulate embryonic stem cell differentiation and Nanog expression.Chromatin organizer SATB1 is an important determinant of T-cell differentiation.The role of SATB1 in breast cancer pathogenesis.Expression and biological roles of SATB1 in human bladder cancer.Tetramerization of SATB1 is essential for regulating of gene expression.SATB1 collaborates with loss of p16 in cellular transformation.Correlation of SATB1 overexpression with the progression of human rectal cancer.PLEIAD/SIMC1/C5orf25, a novel autolysis regulator for a skeletal-muscle-specific calpain, CAPN3, scaffolds a CAPN3 substrate, CTBP1.Special AT-rich sequence-binding protein 1: a novel biomarker predicting cervical squamous cell carcinoma prognosis and lymph node metastasis.Regulation of the oncoprotein KLF8 by a switch between acetylation and sumoylationN-terminal PDZ-like domain of chromatin organizer SATB1 contributes towards its function as transcription regulator.SATB1 promotes tumor metastasis and invasiveness in oral squamous cell carcinoma.The interaction of Jagged-1 cytoplasmic tail with afadin PDZ domain is local, folding-independent, and tuned by phosphorylation.Poor prognosis and SATB1 overexpression in solid tumors: a meta-analysis.
P2860
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P2860
Acetylation-dependent interaction of SATB1 and CtBP1 mediates transcriptional repression by SATB1.
description
2008 nî lūn-bûn
@nan
2008 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Acetylation-dependent interact ...... riptional repression by SATB1.
@ast
Acetylation-dependent interact ...... riptional repression by SATB1.
@en
type
label
Acetylation-dependent interact ...... riptional repression by SATB1.
@ast
Acetylation-dependent interact ...... riptional repression by SATB1.
@en
prefLabel
Acetylation-dependent interact ...... riptional repression by SATB1.
@ast
Acetylation-dependent interact ...... riptional repression by SATB1.
@en
P2093
P2860
P356
P1476
Acetylation-dependent interact ...... criptional repression by SATB1
@en
P2093
Amita S Limaye
Dimple Notani
P Pavan Kumar
Sanjeev Galande
Sunita Singh
P2860
P304
P356
10.1128/MCB.00822-08
P407
P577
2008-12-22T00:00:00Z