A role for the TGFbeta-Par6 polarity pathway in breast cancer progression - Wikidata - wikimedia - TriplyDB

A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

http://www.wikidata.org/entity/Q30489618

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Epithelial cell polarity: a major gatekeeper against cancer?Cancer stem cell division: when the rules of asymmetry are broken Emerging role of cell polarity proteins in breast cancer progression and metastasis Extracellular matrix as a contextual determinant of transforming growth factor-β signaling in epithelial-mesenchymal transition and in cancer Targeting the Transforming Growth Factor-β Signaling in Cancer Therapy RhoGTPase signalling at epithelial tight junctions: Bridging the GAP between polarity and cancer TGFβ signalling in context Asymmetric cell division of stem and progenitor cells during homeostasis and cancer Loss of aPKCλ in differentiated neurons disrupts the polarity complex but does not induce obvious neuronal loss or disorientation in mouse brains The transcription factors Snail and Slug activate the transforming growth factor-beta signaling pathway in breast cancer Loss of the Par3 polarity protein promotes breast tumorigenesis and metastasis.A complex containing LPP and α-actinin mediates TGFβ-induced migration and invasion of ErbB2-expressing breast cancer cells.MUC4 overexpression augments cell migration and metastasis through EGFR family proteins in triple negative breast cancer cells.TGF-β signaling in breast cancer cell invasion and bone metastasis.Atypical protein kinase C (aPKCzeta and aPKClambda) is dispensable for mammalian hematopoietic stem cell activity and blood formation Expression of polarity genes in human cancer.TGFβ and BMP-2 regulate epicardial cell invasion via TGFβR3 activation of the Par6/Smurf1/RhoA pathway.PAR6B is required for tight junction formation and activated PKCζ localization in breast cancer The regulation of cell-cell adhesion during epithelial-mesenchymal transition, motility and tumor progression.FOXC2 expression links epithelial-mesenchymal transition and stem cell properties in breast cancer.Polarity proteins as regulators of cell junction complexes: implications for breast cancer.The pathophysiology of epithelial-mesenchymal transition induced by transforming growth factor-beta in normal and malignant mammary epithelial cells.Noncanonical TGF-β signaling during mammary tumorigenesis.Deconstructing the mechanisms and consequences of TGF-β-induced EMT during cancer progression.Immunohistochemical characterization and functional identification of mammary gland telocytes in the self-assembly of reconstituted breast cancer tissue in vitro Polarity protein alterations in carcinoma: a focus on emerging roles for polarity regulators.A novel highly potent trivalent TGF-β receptor trap inhibits early-stage tumorigenesis and tumor cell invasion in murine Pten-deficient prostate glands.Tumor microenvironment: driving forces and potential therapeutic targets for breast cancer metastasis Cell polarity as a regulator of cancer cell behavior plasticity.The Scribble-Dlg-Lgl polarity module in development and cancer: from flies to man.Crosstalk between breast cancer stem cells and metastatic niche: emerging molecular metastasis pathway?The TGFbeta superfamily signaling pathway.The microRNA networks of TGFβ signaling in cancer.Apico-basal polarity complex and cancer.Rewiring cell polarity signaling in cancer.Suppression of CIP4/Par6 attenuates TGF-β1-induced epithelial-mesenchymal transition in NRK-52E cells.Loss of polarity protein AF6 promotes pancreatic cancer metastasis by inducing Snail expression.Cell Polarity Proteins in Breast Cancer Progression.TGFβ-induced phosphorylation of Par6 promotes migration and invasion in prostate cancer cells Atypical protein kinase C phosphorylates Par6 and facilitates transforming growth factor β-induced epithelial-to-mesenchymal transition

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P2860

A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

http://www.wikidata.org/entity/Q30489618

description

2009 nî lūn-bûn

@nan

2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

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2009年论文

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name

A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

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A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

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type

label

A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

@ast

A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

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prefLabel

A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

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A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

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Proceedings of the National Academy of Sciences of the United States of America

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A role for the TGFbeta-Par6 polarity pathway in breast cancer progression

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Alicia M Viloria-Petit

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Anita L Bane

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Dushanthi Pinnaduwage

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Frances P O'Malley

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Jason Moffat

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Jeffrey L Wrana

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John W Wong

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Jun Yong Jia

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Luba Roncari

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Masahiro Narimatsu

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P2860

Regulation of the polarity protein Par6 by TGFbeta receptors controls epithelial cell plasticity

Deregulation of scribble promotes mammary tumorigenesis and reveals a role for cell polarity in carcinoma

The epithelial-mesenchymal transition generates cells with properties of stem cells

Mesenchyme Forkhead 1 (FOXC2) plays a key role in metastasis and is associated with aggressive basal-like breast cancers

TGFbeta primes breast tumors for lung metastasis seeding through angiopoietin-like 4

Molecular definition of breast tumor heterogeneity

Conditional overexpression of active transforming growth factor beta1 in vivo accelerates metastases of transgenic mammary tumors

Cytostatic and apoptotic actions of TGF-beta in homeostasis and cancer

TGF-beta and epithelial-to-mesenchymal transitions

Molecular requirements for epithelial-mesenchymal transition during tumor progression

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14028-14033

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10.1073/PNAS.0906796106

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Irene L Andrulis

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26729045

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19667198

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