Noncanonical TGF-β signaling during mammary tumorigenesis. - Wikidata - wikimedia - TriplyDB

Noncanonical TGF-β signaling during mammary tumorigenesis.

Noncanonical TGF-β signaling during mammary tumorigenesis.

http://www.wikidata.org/entity/Q37044600

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Cutting the brakes on hematopoietic regeneration by blocking TGFβ to limit chemotherapy-induced myelosuppression Milk--A Nutrient System of Mammalian Evolution Promoting mTORC1-Dependent Translation Endocytosis and Physiology: Insights from Disabled-2 Deficient Mice Involvement of Ras GTPase-activating protein SH3 domain-binding protein 1 in the epithelial-to-mesenchymal transition-induced metastasis of breast cancer cells via the Smad signaling pathway.Loss of SPARC in bladder cancer enhances carcinogenesis and progression.Downregulation of delta-aminolevulinate dehydratase is associated with poor prognosis in patients with breast cancer.Two faces of TGF-beta1 in breast cancer.Transforming growth factor β1 promotes invasion of human JEG-3 trophoblast cells via TGF-β/Smad3 signaling pathway.Detection of Lysyl Oxidase-Like 2 (LOXL2), a Biomarker of Metastasis from Breast Cancers Using Human Blood Samples.Role of TGF-β receptor III localization in polarity and breast cancer progression TTRAP is a novel component of the non-canonical TRAF6-TAK1 TGF-β signaling pathway Semaphorin 3A blocks the formation of pathologic choroidal neovascularization induced by transforming growth factor beta Hormonal induction and roles of Disabled-2 in lactation and involution.Phosphorylation of ΔNp63α via a novel TGFβ/ALK5 signaling mechanism mediates the anti-clonogenic effects of TGFβ.Beta-elemene blocks epithelial-mesenchymal transition in human breast cancer cell line MCF-7 through Smad3-mediated down-regulation of nuclear transcription factors.TLR4 ligand/H₂O₂ enhances TGF-β1 signaling to induce metastatic potential of non-invasive breast cancer cells by activating non-Smad pathways.HOXB7 promotes malignant progression by activating the TGFβ signaling pathway.The prognostic value of combined TGF-β1 and ELF in hepatocellular carcinoma.Deptor enhances triple-negative breast cancer metastasis and chemoresistance through coupling to survivin expression Chemotherapeutic Targeting of the Transforming Growth Factor-β Pathway in Breast Cancers.Silencing β3 Integrin by Targeted ECO/siRNA Nanoparticles Inhibits EMT and Metastasis of Triple-Negative Breast Cancer.Partners in crime: the TGFβ and MAPK pathways in cancer progression.Forkhead Box F1 promotes breast cancer cell migration by upregulating lysyl oxidase and suppressing Smad2/3 signaling Key signaling nodes in mammary gland development and cancer: Smad signal integration in epithelial cell plasticity Tipping the balance between good and evil: aberrant 14-3-3ζ expression drives oncogenic TGF-β signaling in metastatic breast cancers TGF-β upregulates miR-181a expression to promote breast cancer metastasis.EZH2 inhibition decreases p38 signaling and suppresses breast cancer motility and metastasis Jumonji AT-rich interactive domain 1B overexpression is associated with the development and progression of glioma.MYC Is a Crucial Mediator of TGFβ-Induced Invasion in Basal Breast Cancer.TGF-β stimulates Pyk2 expression as part of an epithelial-mesenchymal transition program required for metastatic outgrowth of breast cancer.Role of TGF-β and the tumor microenvironment during mammary tumorigenesis Deconstructing the mechanisms and consequences of TGF-β-induced EMT during cancer progression.The relevance of the TGF-β Paradox to EMT-MET programs Galunisertib (LY2157299), a transforming growth factor-β receptor I kinase inhibitor, attenuates acute pancreatitis in rats.Targeted inactivation of β1 integrin induces β3 integrin switching, which drives breast cancer metastasis by TGF-β Upregulated WAVE3 expression is essential for TGF-β-mediated EMT and metastasis of triple-negative breast cancer cells.Multiple Roles of WNT5A in Breast Cancer.Chamaejasmenin B, a novel candidate, inhibits breast tumor metastasis by rebalancing TGF-beta paradox.c-Abl inhibits breast cancer tumorigenesis through reactivation of p53-mediated p21 expression Sox4, EMT programs, and the metastatic progression of breast cancers: mastering the masters of EMT.

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P2860

Noncanonical TGF-β signaling during mammary tumorigenesis.

http://www.wikidata.org/entity/Q37044600

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 31 March 2011

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Noncanonical TGF-β signaling during mammary tumorigenesis.

@en

Noncanonical TGF-β signaling during mammary tumorigenesis.

@nl

type

label

Noncanonical TGF-β signaling during mammary tumorigenesis.

@en

Noncanonical TGF-β signaling during mammary tumorigenesis.

@nl

prefLabel

Noncanonical TGF-β signaling during mammary tumorigenesis.

@en

Noncanonical TGF-β signaling during mammary tumorigenesis.

@nl

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S10911-011-9207-3

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Journal of Mammary Gland Biology and Neoplasia

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Noncanonical TGF-β signaling during mammary tumorigenesis.

@en

P2093

Jenny G Parvani

http://www.w3.org/2001/XMLSchema#string

Molly A Taylor

http://www.w3.org/2001/XMLSchema#string

William P Schiemann

http://www.w3.org/2001/XMLSchema#string

P2860

Cloning of a novel signaling molecule, AMSH-2, that potentiates transforming growth factor beta signaling

TGF-beta signal transduction

SARA, a FYVE domain protein that recruits Smad2 to the TGFbeta receptor

Smurf2 is a ubiquitin E3 ligase mediating proteasome-dependent degradation of Smad2 in transforming growth factor-beta signaling

Smad7 binds to Smurf2 to form an E3 ubiquitin ligase that targets the TGF beta receptor for degradation

Smurf1 interacts with transforming growth factor-beta type I receptor through Smad7 and induces receptor degradation

The adaptor molecule Disabled-2 links the transforming growth factor beta receptors to the Smad pathway.

Differential regulation of TGF-beta signaling through Smad2, Smad3 and Smad4

Regulation of the polarity protein Par6 by TGFbeta receptors controls epithelial cell plasticity

Disabled-2 (Dab2) mediates transforming growth factor beta (TGFbeta)-stimulated fibronectin synthesis through TGFbeta-activated kinase 1 and activation of the JNK pathway

P2888

s10911-011-9207-3

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127-146

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scholarly article

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10.1007/S10911-011-9207-3

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2

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16

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2011-03-31T00:00:00Z

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21448580

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3723114

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