Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells. - Wikidata - wikimedia - TriplyDB

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

http://www.wikidata.org/entity/Q30495914

about

Innate immunity in the central nervous system Chemokine receptor CXCR2: physiology regulator and neuroinflammation controller?The inflammasome pyrin contributes to pertussis toxin-induced IL-1β synthesis, neutrophil intravascular crawling and autoimmune encephalomyelitis Quetiapine, an atypical antipsychotic, is protective against autoimmune-mediated demyelination by inhibiting effector T cell proliferation ELR(+) chemokine signaling in host defense and disease in a viral model of central nervous system disease A regenerative approach to the treatment of multiple sclerosis.Promoting remyelination: utilizing a viral model of demyelination to assess cell-based therapies Hydroxycarboxylic acid receptor 2 mediates dimethyl fumarate's protective effect in EAE.Central nervous system pathology progresses independently of KC and CXCR2 in globoid-cell leukodystrophy Design, synthesis, and evaluation of coumarin-based molecular probes for imaging of myelination.Mediators of oligodendrocyte differentiation during remyelination Acute skeletal muscle injury: CCL2 expression by both monocytes and injured muscle is required for repair.Chemokines, macrophage inflammatory protein-2 and stromal cell-derived factor-1α, suppress amyloid β-induced neurotoxicity.18β-glycyrrhetinic acid suppresses experimental autoimmune encephalomyelitis through inhibition of microglia activation and promotion of remyelination.Subcutaneous Transplantation of Neural Precursor Cells in Experimental Autoimmune Encephalomyelitis Reduces Chemotactic Signals in the Central Nervous System Spatiotemporal ablation of CXCR2 on oligodendrocyte lineage cells: Role in myelin repair.Astrocyte TNFR2 is required for CXCL12-mediated regulation of oligodendrocyte progenitor proliferation and differentiation within the adult CNS.CXCL5 signaling is a shared pathway of neuroinflammation and blood-brain barrier injury contributing to white matter injury in the immature brain.The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.Accelerated and enhanced effect of CCR5-transduced bone marrow neural stem cells on autoimmune encephalomyelitis.CCR5 knockout suppresses experimental autoimmune encephalomyelitis in C57BL/6 mice.Functional defect of peripheral neutrophils in mice with induced deletion of CXCR2 Mesenchymal Stem Cells Reversed Morphine Tolerance and Opioid-induced Hyperalgesia.CXCR4 signaling regulates remyelination by endogenous oligodendrocyte progenitor cells in a viral model of demyelination.International Union of Basic and Clinical Pharmacology. [corrected]. LXXXIX. Update on the extended family of chemokine receptors and introducing a new nomenclature for atypical chemokine receptors.Interactions between neutrophils, Th17 cells, and chemokines during the initiation of experimental model of multiple sclerosis.The complex world of oligodendroglial differentiation inhibitors.New insights into ADAMs regulation of the GRO-α/CXCR2 system: focus on Sjögren's syndrome.Attempts to Overcome Remyelination Failure: Toward Opening New Therapeutic Avenues for Multiple Sclerosis.Induction of Experimental Autoimmune Encephalomyelitis in Mice and Evaluation of the Disease-dependent Distribution of Immune Cells in Various Tissues.Discovery of CNS Penetrant CXCR2 Antagonists for the Potential Treatment of CNS Demyelinating Disorders.A mouse model for testing remyelinating therapies.Exacerbation of experimental autoimmune encephalomyelitis in ceramide synthase 6 knockout mice is associated with enhanced activation/migration of neutrophils.CXCL1 can be regulated by IL-6 and promotes granulocyte adhesion to brain capillaries during bacterial toxin exposure and encephalomyelitis.Interleukin-36γ is expressed by neutrophils and can activate microglia, but has no role in experimental autoimmune encephalomyelitis.Polyomavirus JC infection inhibits differentiation of oligodendrocyte progenitor cells.Induced CNS expression of CXCL1 augments neurologic disease in a murine model of multiple sclerosis via enhanced neutrophil recruitment.

P2860

Q26858882-BC00B334-D443-41C3-8FCF-DA2495C800B6 Q27007078-A2C520F7-BA48-400D-992F-7EBD1CC2258E Q27325503-9EB671B3-601F-4D54-81A2-58E13F7D635A Q28482227-FC0B324A-DFD7-472F-8F86-BB0C51BC6702 Q33767086-DB8AB47D-DB03-4612-93C1-9939023EFAA4 Q34376469-17A0EDD0-6F34-4DBF-8520-C0FDFE4D1688 Q34512363-07A1F31A-4C6A-40D7-89B5-8435C6361CA9 Q34660955-F74C15BA-10FC-4884-BF62-C693E39A674A Q34765786-111F191C-2556-4D36-ADBF-EAE91A79051D Q34996552-E9490352-1583-4ED0-B3FC-FD98BF8C3550 Q35173135-4ABBDF52-E86A-4C07-AD69-A6EECD00C8F8 Q35225072-BE277E97-D6CC-4656-BAD7-BDE78B009A3D Q35607881-95ED3065-D8E0-4474-8AC1-A00ACE4F0316 Q36018168-0C3513D2-A063-4A3B-9B21-94F3B1980FB7 Q36362639-9EFB205E-7DDB-4B29-B003-1A1F01D527C1 Q36364790-5F8FBBD7-3EF2-493B-B8A4-FB3E5280B9C4 Q36428721-067DCC73-EDAB-488E-981B-8E1A996FDD61 Q36439707-CFA666D3-AE71-4774-91DE-24DC675260A9 Q36468266-13267126-BFD0-49AB-BB72-7A23259D8E3B Q36530295-A15045E3-6775-46E9-AE6A-22E81D529A5C Q37085790-DDB4770E-FC62-4705-B166-44A9E6B511E7 Q37120407-C8C4D909-8BE0-4630-B40D-EDC86845CDB1 Q37198850-6513CB82-EDDD-47D4-906B-DC3E4875E77B Q37259931-9F1FF4D0-F59E-427C-AB0F-08868096D8D4 Q37432097-F0EDBBC5-9FE2-4C24-A525-0FF821592E7F Q37623410-A9F15701-675A-4D28-817B-CC0A3253EDA9 Q37869047-0D8F7A11-7E49-47BC-91DF-8CEBA673678F Q38267059-ED83F295-A80E-44A7-983A-355139A6AFE1 Q39146141-81174806-460F-4E6E-9920-78F412A3330B Q39743749-5663A944-8BDC-4BE1-8CD6-67FA21FDB2AF Q40277352-908C334E-3BCB-4D47-9F9C-F6F3B39E58EA Q40567848-C1F0325A-3EA3-4EAD-AFED-FE179623B718 Q41126508-C89D2E0D-9473-4819-BFB7-B4ED9E858F69 Q41360196-AFB736EE-79E8-4DE1-BFCE-BA6E88237D2C Q41825896-F25AA733-1414-41E3-956D-6207EE5B465F Q43180222-550637E0-3528-4F42-A505-6BE0DFAB74E0 Q52561535-44EC72DD-637B-4E94-A5EA-326A80244FAD

P2860

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

http://www.wikidata.org/entity/Q30495914

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

@ast

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

@en

type

label

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

@ast

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

@en

prefLabel

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

@ast

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

@en

P1433

Q30495914-D6D48A7A-DF9D-423D-BE47-C6F15E0E068D

P1476

Q30495914-AFA45536-8538-4034-9DDA-93F6B05DD6D1

P2093

Q30495914-1D96510F-A18B-4DAD-8DA9-AFF84929A468

Q30495914-1E0B8A82-7BF8-4E5A-BE2C-95FE9B671BFF

Q30495914-9A589FC2-667A-4FA7-9897-C74D3566E0B1

Q30495914-A874DE08-8236-4DB4-962C-FC83CF85DD47

Q30495914-ACB53118-13CC-4B1D-B81B-2FDAD8AB6B44

Q30495914-CA27D6D1-90B6-4C59-98AA-14EC574F058D

P2860

Q30495914-031F5003-FFF4-4796-ADC1-C4B350611D0C

Q30495914-046395AA-0522-4C9B-ACF7-21396ED1BE90

Q30495914-07EE6878-4A92-4D21-AC86-E6591A3B50F5

Q30495914-0CBDD211-6BFF-4F61-90E2-512650B650C3

Q30495914-15D19994-7DA2-4FA2-B94E-73AA89D1AD60

Q30495914-252A77A6-8FCA-4CFB-BA7E-771BBF06AB99

Q30495914-29E21DB1-4B07-43E9-AE2D-40AEC33ED9A1

Q30495914-772E2426-91C7-401F-96BF-58AB74CF77CA

Q30495914-7E4E8E99-6AFB-4202-9704-498D639DA858

Q30495914-837B1349-28F5-4FFD-B277-BECED4F81BBD

P304

Q30495914-35266C7E-9FF2-4D3B-A9A3-5EC09A0A445F

P31

Q30495914-F3926550-9097-4507-8C9C-01D27FB5056E

P356

Q30495914-CEAE9974-EA78-4788-BF5D-580D0BEA5AEC

P407

Q30495914-26D71A4F-3FCA-46F6-B132-05E3D935FBC7

P433

Q30495914-70C4B096-3028-42EF-92E6-EA01783658D0

P478

Q30495914-A7F8525A-8D2D-47C4-84E1-FA6DA828CEBC

P577

Q30495914-F20607D8-87A2-4004-B72D-99557F796529

P698

Q30495914-63E88756-E698-45D6-BFD3-64CC11F5D5F9

P932

Q30495914-F2AE65F0-1B09-4B36-B0D3-D927B4FBE18B

P356

JNEUROSCI.1238-10.2010

P1433

Journal of Neuroscience

P1476

Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.

@en

P2093

Karen Choi

http://www.w3.org/2001/XMLSchema#string

LiPing Liu

http://www.w3.org/2001/XMLSchema#string

Lindsey Darnall

http://www.w3.org/2001/XMLSchema#string

Richard M Ransohoff

http://www.w3.org/2001/XMLSchema#string

Taofang Hu

http://www.w3.org/2001/XMLSchema#string

Thomas E Lane

http://www.w3.org/2001/XMLSchema#string

P2860

Delayed wound healing in CXCR2 knockout mice

The chemokine receptor CXCR2 controls positioning of oligodendrocyte precursors in developing spinal cord by arresting their migration

Neutrophil and B cell expansion in mice that lack the murine IL-8 receptor homolog

Of mice and not men: differences between mouse and human immunology

CXCR2-positive neutrophils are essential for cuprizone-induced demyelination: relevance to multiple sclerosis

The neurotoxicant, cuprizone, as a model to study demyelination and remyelination in the central nervous system.

Why does remyelination fail in multiple sclerosis?

Origin of oligodendrocytes in the subventricular zone of the adult brain.

Multiple sclerosis and chronic autoimmune encephalomyelitis: a comparative quantitative study of axonal injury in active, inactive, and remyelinated lesions.

Synchronous synthesis of alpha- and beta-chemokines by cells of diverse lineage in the central nervous system of mice with relapses of chronic experimental autoimmune encephalomyelitis.

P304

9074-9083

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1523/JNEUROSCI.1238-10.2010

http://www.w3.org/2001/XMLSchema#string

P407

P433

27

http://www.w3.org/2001/XMLSchema#string

P478

30

http://www.w3.org/2001/XMLSchema#string

P577

2010-07-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

20610741

http://www.w3.org/2001/XMLSchema#string

P932

2917803

http://www.w3.org/2001/XMLSchema#string