Acute skeletal muscle injury: CCL2 expression by both monocytes and injured muscle is required for repair. - Wikidata - wikimedia - TriplyDB

Acute skeletal muscle injury: CCL2 expression by both monocytes and injured muscle is required for repair.

Acute skeletal muscle injury: CCL2 expression by both monocytes and injured muscle is required for repair.

http://www.wikidata.org/entity/Q35225072

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Modulating exercise-induced hormesis: Does less equal more?Multi-scale in vivo systems analysis reveals the influence of immune cells on TNF-α-induced apoptosis in the intestinal epithelium In vivo Monitoring of Transcriptional Dynamics After Lower-Limb Muscle Injury Enables Quantitative Classification of Healing Disruption of mammalian target of rapamycin complex 1 in macrophages decreases chemokine gene expression and atherosclerosis.Stem cell transplantation for muscular dystrophy: the challenge of immune response.Expression of NF-κB p50 in tumor stroma limits the control of tumors by radiation therapy.An intracameral injection of antigen induces in situ chemokines and cytokines required for the generation of circulating immunoregulatory monocytes.Effects of nitric oxide on notexin-induced muscle inflammatory responses.Monocytes in sterile inflammation: recruitment and functional consequences Variants of the IL-10 gene associate with muscle strength in elderly from rural Africa: a candidate gene study.Bone marrow cells migrate to the heart and skeletal muscle and participate in tissue repair after Trypanosoma cruzi infection in mice.Toll-like receptor 4 ablation in mdx mice reveals innate immunity as a therapeutic target in Duchenne muscular dystrophy Anti-inflammatory macrophages improve skeletal muscle recovery from ischemia-reperfusion.Messenger RNA sequencing and pathway analysis provide novel insights into the biological basis of chickens' feed efficiency.Low-dose TNF augments fracture healing in normal and osteoporotic bone by up-regulating the innate immune response.The Development of Macrophage-Mediated Cell Therapy to Improve Skeletal Muscle Function after Injury.Factor VII-activating protease deficiency promotes neointima formation by enhancing leukocyte accumulation.IL-10 triggers changes in macrophage phenotype that promote muscle growth and regeneration.IFN-α-driven CCL2 production recruits inflammatory monocytes to infection site in mice.CX3CR1 deficiency delays acute skeletal muscle injury repair by impairing macrophage functions.CX3CR1 deficiency promotes muscle repair and regeneration by enhancing macrophage ApoE production Muscle-specific inflammation induced by MCP-1 overexpression does not affect whole-body insulin sensitivity in mice.Obesity-driven inflammation and cancer risk: role of myeloid derived suppressor cells and alternately activated macrophages.Requirement of inducible nitric oxide synthase for skeletal muscle regeneration after acute damage.Toll-like receptor signalling in regenerative myogenesis: friend and foe.Muscle-specific deletion of SOCS3 increases the early inflammatory response but does not affect regeneration after myotoxic injury Blockade of 4-1BB and 4-1BBL interaction reduces obesity-induced skeletal muscle inflammation.Immunomodulatory effects of massage on nonperturbed skeletal muscle in rats.64CuCl2 PET/CT imaging of mouse muscular injury induced by electroporation.Possible involvement of IGF-1 signaling on compensatory growth of the infraspinatus muscle induced by the supraspinatus tendon detachment of rat shoulder.Altered macrophage phenotype transition impairs skeletal muscle regeneration Diverse macrophage populations mediate acute lung inflammation and resolution.Macrophages in injured skeletal muscle: a perpetuum mobile causing and limiting fibrosis, prompting or restricting resolution and regeneration.Acute lung injury: how macrophages orchestrate resolution of inflammation and tissue repair Monocyte/macrophage interactions with myogenic precursor cells during skeletal muscle regeneration.Phenotypic transitions of macrophages orchestrate tissue repair.Genetically modified pigs to model human diseases.Post-exercise impact of ingested whey protein hydrolysate on gene expression profiles in rat skeletal muscle: activation of extracellular signal-regulated kinase 1/2 and hypoxia-inducible factor-1α.Contracting C2C12 myotubes release CCL2 in an NF-κB-dependent manner to induce monocyte chemoattraction.Friend or Foe? Resident Microglia vs Bone Marrow-Derived Microglia and Their Roles in the Retinal Degeneration.

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Acute skeletal muscle injury: CCL2 expression by both monocytes and injured muscle is required for repair.

http://www.wikidata.org/entity/Q35225072

description

2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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Acute skeletal muscle injury: ...... muscle is required for repair.

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Danping Huang

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Haiyan Lu

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Lan Zhou

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Richard M Ransohoff

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P2860

Inflammatory mediators and skeletal muscle injury: a DNA microarray analysis

Recent developments in CCR2 antagonists

Effect of unloading followed by reloading on expression of collagen and related growth factors in rat tendon and muscle

CC chemokine receptor 2 is critical for induction of experimental autoimmune encephalomyelitis

Severe reduction in leukocyte adhesion and monocyte extravasation in mice deficient in CC chemokine receptor 2.

Absence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitis

Monocyte and macrophage heterogeneity

Blood monocytes consist of two principal subsets with distinct migratory properties

The many roles of chemokines and chemokine receptors in inflammation

Mice carrying null mutations of the genes encoding insulin-like growth factor I (Igf-1) and type 1 IGF receptor (Igf1r)

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