β-Amyloid impairs axonal BDNF retrograde trafficking. - Wikidata - wikimedia - TriplyDB

β-Amyloid impairs axonal BDNF retrograde trafficking.

β-Amyloid impairs axonal BDNF retrograde trafficking.

http://www.wikidata.org/entity/Q30498160

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Potential therapeutic effects of neurotrophins for acute and chronic neurological diseases New drug treatments show neuroprotective effects in Alzheimer's and Parkinson's diseases Defects in mitochondrial dynamics and metabolomic signatures of evolving energetic stress in mouse models of familial Alzheimer's disease Amyloid-Beta Induced Changes in Vesicular Transport of BDNF in Hippocampal Neurons A γ-secretase inhibitor, but not a γ-secretase modulator, induced defects in BDNF axonal trafficking and signaling: evidence for a role for APP Functional Impact of Corticotropin-Releasing Factor Exposure on Tau Phosphorylation and Axon Transport Compartmentalized Platforms for Neuro-Pharmacological Research Age-dependent neuroplasticity mechanisms in Alzheimer Tg2576 mice following modulation of brain amyloid-β levels Quantitative analysis of axonal transport by using compartmentalized and surface micropatterned culture of neurons.Microarray analysis of hippocampal CA1 neurons implicates early endosomal dysfunction during Alzheimer's disease progression.Modulators of cytoskeletal reorganization in CA1 hippocampal neurons show increased expression in patients at mid-stage Alzheimer's disease.Differences in abundances of cell-signalling proteins in blood reveal novel biomarkers for early detection of clinical Alzheimer's disease BDNF increases with behavioral enrichment and an antioxidant diet in the aged dog L-type voltage-gated calcium channel blockade with isradipine as a therapeutic strategy for Alzheimer's disease A selective histone deacetylase-6 inhibitor improves BDNF trafficking in hippocampal neurons from Mecp2 knockout mice: implications for Rett syndrome.Disrupted energy metabolism and neuronal circuit dysfunction in cognitive impairment and Alzheimer's disease.Aberrant subcellular neuronal calcium regulation in aging and Alzheimer's disease The Aβ oligomer hypothesis for synapse failure and memory loss in Alzheimer's disease.Morphometric assessment of toxicant induced neuronal degeneration in full and restricted contact co-cultures of embryonic cortical rat neurons and astrocytes: using m-Dinitrobezene as a model neurotoxicant.7,8-dihydroxyflavone, a small-molecule TrkB agonist, reverses memory deficits and BACE1 elevation in a mouse model of Alzheimer's disease.Neurotrophins role in depression neurobiology: a review of basic and clinical evidence.Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's Disease New perspectives on neuronal development via microfluidic environments.Over a century of neuron culture: from the hanging drop to microfluidic devices.β-Amyloid (Aβ) oligomers impair brain-derived neurotrophic factor retrograde trafficking by down-regulating ubiquitin C-terminal hydrolase, UCH-L1.Presynaptic dystrophic neurites surrounding amyloid plaques are sites of microtubule disruption, BACE1 elevation, and increased Aβ generation in Alzheimer's disease.Synaptic Dysfunction in Alzheimer's Disease and Glaucoma: From Common Degenerative Mechanisms Toward Neuroprotection.Effect of a Comprehensive Intervention on Plasma BDNF in Patients with Alzheimer's Disease.Alzheimer's Toxic Amyloid Beta Oligomers: Unwelcome Visitors to the Na/K ATPase alpha3 Docking Station.Brain-derived neurotrophic factor and Alzheimer's disease: physiopathology and beyond.Multiple approaches to investigate the transport and activity-dependent release of BDNF and their application in neurogenetic disorders.Microtechnologies to fuel neurobiological research with nanometer precision.Microfluidic platforms for the investigation of intercellular signalling mechanisms.The role of rab proteins in neuronal cells and in the trafficking of neurotrophin receptors.Brain-Derived Neurotrophic Factor in Alzheimer's Disease: Risk, Mechanisms, and Therapy.Three-Dimensional Models of the Human Brain Development and Diseases.Neurotrophic factors in Alzheimer's and Parkinson's diseases: implications for pathogenesis and therapy.Integral Characterization of Defective BDNF/TrkB Signalling in Neurological and Psychiatric Disorders Leads the Way to New Therapies.Neural stem cell therapy for neurodegenerative disorders: The role of neurotrophic support.Microfluidic construction of minimalistic neuronal co-cultures.

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P2860

β-Amyloid impairs axonal BDNF retrograde trafficking.

http://www.wikidata.org/entity/Q30498160

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

β-Amyloid impairs axonal BDNF retrograde trafficking.

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β-Amyloid impairs axonal BDNF retrograde trafficking.

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type

label

β-Amyloid impairs axonal BDNF retrograde trafficking.

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β-Amyloid impairs axonal BDNF retrograde trafficking.

@en

prefLabel

β-Amyloid impairs axonal BDNF retrograde trafficking.

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β-Amyloid impairs axonal BDNF retrograde trafficking.

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J.NEUROBIOLAGING.2009.05.012

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Neurobiology of Aging

P1476

β-Amyloid impairs axonal BDNF retrograde trafficking.

@en

P2093

Carl W Cotman

http://www.w3.org/2001/XMLSchema#string

Christina H Tu

http://www.w3.org/2001/XMLSchema#string

Joe W Harris

http://www.w3.org/2001/XMLSchema#string

Leila M Feinberg

http://www.w3.org/2001/XMLSchema#string

Meredith A Chabrier

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Noo Li Jeon

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P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Modulation of the dynamic instability of tubulin assembly by the microtubule-associated protein tau

Caspase cleavage of tau: linking amyloid and neurofibrillary tangles in Alzheimer's disease

Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy

Synaptic secretion of BDNF after high-frequency stimulation of glutamatergic synapses.

Als2-deficient mice exhibit disturbances in endosome trafficking associated with motor behavioral abnormalities

Alzheimer's presenilin 1 mutations impair kinesin-based axonal transport

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821-833

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scholarly article

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10.1016/J.NEUROBIOLAGING.2009.05.012

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5

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32

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Mathew Blurton-Jones

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2009-06-21T00:00:00Z

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26305410

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19540623

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3038182

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