Class I HDAC inhibition blocks cocaine-induced plasticity by targeted changes in histone methylation. - Wikidata - wikimedia - TriplyDB

Class I HDAC inhibition blocks cocaine-induced plasticity by targeted changes in histone methylation.

Class I HDAC inhibition blocks cocaine-induced plasticity by targeted changes in histone methylation.

http://www.wikidata.org/entity/Q30538086

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Chronic cocaine-regulated epigenomic changes in mouse nucleus accumbens.Epigenetic mechanisms of drug addiction Vitamin D receptor and epigenetics in HIV infection and drug abuse Visualizing epigenetics: current advances and advantages in HDAC PET imaging techniques Regulation of chromatin states by drugs of abuse BDNF rescues BAF53b-dependent synaptic plasticity and cocaine-associated memory in the nucleus accumbens Dysregulation of Acetylation Enzymes Inanimal Models of Psychostimulant use Disorders: Evolving Stories Targeted Epigenetic Remodeling of the Cdk5 Gene in Nucleus Accumbens Regulates Cocaine- and Stress-Evoked Behavior G9a influences neuronal subtype specification in striatum Epigenetics and memory: causes, consequences and treatments for post-traumatic stress disorder and addiction.Combinatorial epigenetic patterns as quantitative predictors of chromatin biology.Beyond Neuronal Activity Markers: Select Immediate Early Genes in Striatal Neuron Subtypes Functionally Mediate Psychostimulant Addiction.Epigenetic signaling in psychiatric disorders.Mechanisms of epigenetic memory and addiction.PET imaging demonstrates histone deacetylase target engagement and clarifies brain penetrance of known and novel small molecule inhibitors in rat.FDG-PET imaging reveals local brain glucose utilization is altered by class I histone deacetylase inhibitors.Loss of BDNF signaling in D1R-expressing NAc neurons enhances morphine reward by reducing GABA inhibition Transcriptional and epigenetic substrates of methamphetamine addiction and withdrawal: evidence from a long-access self-administration model in the rat.Epigenetics, microRNA, and addiction.Kinetic Analysis and Quantification of [¹¹C]Martinostat for in Vivo HDAC Imaging of the Brain.Transgenerational Inheritance of Paternal Neurobehavioral Phenotypes: Stress, Addiction, Ageing and Metabolism.Development of a Fluorinated Class-I HDAC Radiotracer Reveals Key Chemical Determinants of Brain Penetrance.Locus-specific epigenetic remodeling controls addiction- and depression-related behaviors.Genome-wide profiling identifies a subset of methamphetamine (METH)-induced genes associated with METH-induced increased H4K5Ac binding in the rat striatum.Neuroepigenomics: Resources, Obstacles, and Opportunities.Antidepressant action of HDAC inhibition in the prefrontal cortex.CAMKII-conditional deletion of histone deacetylase 2 potentiates acute methamphetamine-induced expression of immediate early genes in the mouse nucleus accumbens.Glutamatergic and GABAergic susceptibility loci for heroin and cocaine addiction in subjects of African and European ancestry The Class I-Specific HDAC Inhibitor MS-275 Decreases Motivation to Consume Alcohol and Relapse in Heavy Drinking Rats.Epigenetic landscape of amphetamine and methamphetamine addiction in rodents.Epigenetic Readers of Lysine Acetylation Regulate Cocaine-Induced Plasticity Sigma-1 receptor mediates cocaine-induced transcriptional regulation by recruiting chromatin-remodeling factors at the nuclear envelope Epigenetics of Stress, Addiction, and Resilience: Therapeutic Implications Cocaine elicits autophagic cytotoxicity via a nitric oxide-GAPDH signaling cascade Histone Modification of Nedd4 Ubiquitin Ligase Controls the Loss of AMPA Receptors and Cognitive Impairment Induced by Repeated Stress.Epigenetic mechanisms of drug addiction.Histone arginine methylation in cocaine action in the nucleus accumbens.Epigenetics and psychiatry.Alterations of the Host Microbiome Affect Behavioral Responses to Cocaine.The brain reward circuitry in mood disorders.

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P2860

Class I HDAC inhibition blocks cocaine-induced plasticity by targeted changes in histone methylation.

http://www.wikidata.org/entity/Q30538086

description

2013 nî lūn-bûn

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2013 թուականի Մարտին հրատարակուած գիտական յօդուած

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2013 թվականի մարտին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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Class I HDAC inhibition blocks ...... hanges in histone methylation.

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Class I HDAC inhibition blocks ...... hanges in histone methylation.

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type

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Class I HDAC inhibition blocks ...... hanges in histone methylation.

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Class I HDAC inhibition blocks ...... hanges in histone methylation.

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Class I HDAC inhibition blocks ...... hanges in histone methylation.

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Class I HDAC inhibition blocks ...... hanges in histone methylation.

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Nature Neuroscience

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Class I HDAC inhibition blocks ...... hanges in histone methylation.

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A J Robison

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Ana Badimon

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Diane M Damez-Werno

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Dipesh Chaudhury

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Eric N Olson

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Ezekiell Mouzon

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Jian Feng

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Ming-Hu Han

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Pamela J Kennedy

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Rhonda Bassel-Duby

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P2860

Synaptic plasticity and addiction

HDAC2 negatively regulates memory formation and synaptic plasticity.

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

Genome-wide analysis of chromatin regulation by cocaine reveals a role for sirtuins

Chromatin modifications and their function

Histone deacetylase 5 epigenetically controls behavioral adaptations to chronic emotional stimuli

A translational profiling approach for the molecular characterization of CNS cell types

Chromatin remodeling is a key mechanism underlying cocaine-induced plasticity in striatum

HDAC2 regulates atypical antipsychotic responses through the modulation of mGlu2 promoter activity.

Drug-induced activation of dopamine D(1) receptor signaling and inhibition of class I/II histone deacetylase induce chromatin remodeling in reward circuitry and modulate cocaine-related behaviors

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434-440

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10.1038/NN.3354

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4

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16

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Eric J. Nestler

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2013-03-10T00:00:00Z

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235895667

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1024631960

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23475113

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3609040

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