T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice - Wikidata - wikimedia - TriplyDB

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

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Different immunological mechanisms govern protection from experimental stroke in young and older mice with recombinant TCR ligand therapy.Cell-Intrinsic gp130 Signaling on CD4+ T Cells Shapes Long-Lasting Antiviral Immunity Natural allelic variation of the IL-21 receptor modulates ischemic stroke infarct volume.Non-invasive tracking of CD4+ T cells with a paramagnetic and fluorescent nanoparticle in brain ischemia Inflammatory Disequilibrium in Stroke Hydrogel scaffolds promote neural gene expression and structural reorganization in human astrocyte cultures.IL-21 as a therapeutic target in inflammatory disorders.γδ T cells as early sensors of tissue damage and mediators of secondary neurodegeneration.Recent and near-future advances in nucleic acid-based diagnosis of stroke.IL-21 and IL-21 receptor in the immunopathogenesis of multiple sclerosis.IL-21 promotes myocardial ischaemia/reperfusion injury through the modulation of neutrophil infiltration.Interleukin-21 expression in hippocampal astrocytes is enhanced following kainic acid-induced seizures.Selective Inhibition of Janus Kinase 3 Has No Impact on Infarct Size or Neurobehavioral Outcomes in Permanent Ischemic Stroke in Mice Therapeutic Potential of Intravenous Immunoglobulin in Acute Brain Injury.Bruton's tyrosine kinase is essential for NLRP3 inflammasome activation and contributes to ischaemic brain injury.Nicotinamide Adenine Dinucleotide Protects against Spinal Cord Ischemia Reperfusion Injury-Induced Apoptosis by Blocking Autophagy.Electroacupuncture ameliorating post-stroke cognitive impairments via inhibition of peri-infarct astroglial and microglial/macrophage P2 purinoceptors-mediated neuroinflammation and hyperplasia Regulatory CD8(+)CD122 (+) T-cells predominate in CNS after treatment of experimental stroke in male mice with IL-10-secreting B-cells.First translational 'Think Tank' on cerebrovascular disease, cognitive impairment and dementia.Immune responses in stroke: how the immune system contributes to damage and healing after stroke and how this knowledge could be translated to better cures?IgM response against amyloid-beta in aging: a potential peripheral protective mechanism

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P2860

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

http://www.wikidata.org/entity/Q30575605

description

2014 nî lūn-bûn

@nan

2014 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

@ast

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

@en

type

label

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

@ast

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

@en

prefLabel

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

@ast

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

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Journal of Experimental Medicine

P1476

T cell-derived interleukin (IL)-21 promotes brain injury following stroke in mice

@en

P2093

Aditya Rayasam

http://www.w3.org/2001/XMLSchema#string

Changying Ling

http://www.w3.org/2001/XMLSchema#string

Dandan Sun

http://www.w3.org/2001/XMLSchema#string

M Shahriar Salamat

http://www.w3.org/2001/XMLSchema#string

Matyas Sandor

http://www.w3.org/2001/XMLSchema#string

Melissa G Harris

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Vijay Kuchroo

http://www.w3.org/2001/XMLSchema#string

Zsuzsanna Fabry

http://www.w3.org/2001/XMLSchema#string

P2860

Reversible middle cerebral artery occlusion without craniectomy in rats

Interleukin-18 involvement in hypoxic-ischemic brain injury

Th17 promotes acute rejection following liver transplantation in rats

IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells

Differences in vulnerability to permanent focal cerebral ischemia among 3 common mouse strains.

Leukocyte recruitment and ischemic brain injury.

Excessive Na+/H+ exchange in disruption of dendritic Na+ and Ca2+ homeostasis and mitochondrial dysfunction following in vitro ischemia

IL-21 and IL-21 receptor expression in lymphocytes and neurons in multiple sclerosis brain.

Interleukin-21 is critically required in autoimmune and allogeneic responses to islet tissue in murine models.

IL-21 promotes the pathologic immune response to pneumovirus infection.

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595-604

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scholarly article

P356

10.1084/JEM.20131377

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P433

4

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P478

211

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John D. Lambris

Benjamin D Clarkson

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2014-03-10T00:00:00Z

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260684558

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24616379

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3978271

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