Translocation of glycogen synthase kinase-3β (GSK-3β), a trigger of permeability transition, is kinase activity-dependent and mediated by interaction with voltage-dependent anion channel 2 (VDAC2).
about
Ischaemic conditioning and targeting reperfusion injury: a 30 year voyage of discoveryGsk3 Signalling and Redox Status in Bipolar Disorder: Evidence from Lithium EfficacyAccelerated recovery of mitochondrial membrane potential by GSK-3β inactivation affords cardiomyocytes protection from oxidant-induced necrosis.How the Wnt signaling pathway protects from neurodegeneration: the mitochondrial scenarioMotifs of VDAC2 required for mitochondrial Bak import and tBid-induced apoptosis.Remote ischemic preconditioning for kidney protection: GSK3β-centric insights into the mechanism of action.Wnt Signaling Prevents the Aβ Oligomer-Induced Mitochondrial Permeability Transition Pore Opening Preserving Mitochondrial Structure in Hippocampal Neurons.Meeting report from the 2nd International Symposium on New Frontiers in Cardiovascular Research. Protecting the cardiovascular system from ischemia: between bench and bedsideThe SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.Glycogen synthase kinase-3-mediated phosphorylation of serine 73 targets sterol response element binding protein-1c (SREBP-1c) for proteasomal degradationSIRT3 Blocks Aging-Associated Tissue Fibrosis in Mice by Deacetylating and Activating Glycogen Synthase Kinase 3β.Soluble guanylate cyclase activation during ischemic injury in mice protects against postischemic inflammation at the mitochondrial level.Mitochondria as key targets of cardioprotection in cardiac ischemic disease: role of thyroid hormone triiodothyronine.VDAC-2: Mitochondrial outer membrane regulator masquerading as a channel?Ginkgolide K attenuates neuronal injury after ischemic stroke by inhibiting mitochondrial fission and GSK-3β-dependent increases in mitochondrial membrane permeability.Mitochondrial Ca2+ and regulation of the permeability transition pore.Therapeutic Mechanisms of Lithium in Bipolar Disorder: Recent Advances and Current Understanding.Sarcoplasmic reticulum-mitochondria communication in cardiovascular pathophysiology.Cypermethrin Stimulates GSK3β-Dependent Aβ and p-tau Proteins and Cognitive Loss in Young Rats: Reduced HB-EGF Signaling and Downstream Neuroinflammation as Critical Regulators.VDAC2-specific cellular functions and the underlying structure.Cardiac energy dependence on glucose increases metabolites related to glutathione and activates metabolic genes controlled by mechanistic target of rapamycin.Adenosine receptors: regulatory players in the preservation of mitochondrial function induced by ischemic preconditioning of rat liver.Heat Stress Reduces Sperm Motility via Activation of Glycogen Synthase Kinase-3α and Inhibition of Mitochondrial Protein Import.Isoflurane Is More Deleterious to Developing Brain Than Desflurane: The Role of the Akt/GSK3β Signaling Pathway.An Oldie but Goodie: Lithium in the Treatment of Bipolar Disorder through Neuroprotective and Neurotrophic Mechanisms.Protein-protein interaction networks as a new perspective to evaluate distinct functional roles of voltage-dependent anion channel isoforms.Mitochondrial VDAC2 and cell homeostasis: highlighting hidden structural features and unique functionalities.Mitochondria in Cardiac Postconditioning.RAGE-dependent mitochondria pathway: a novel target of silibinin against apoptosis of osteoblastic cells induced by advanced glycation end products.
P2860
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P2860
Translocation of glycogen synthase kinase-3β (GSK-3β), a trigger of permeability transition, is kinase activity-dependent and mediated by interaction with voltage-dependent anion channel 2 (VDAC2).
description
2014 nî lūn-bûn
@nan
2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Translocation of glycogen synt ...... ndent anion channel 2 (VDAC2).
@ast
Translocation of glycogen synt ...... ndent anion channel 2 (VDAC2).
@en
type
label
Translocation of glycogen synt ...... ndent anion channel 2 (VDAC2).
@ast
Translocation of glycogen synt ...... ndent anion channel 2 (VDAC2).
@en
prefLabel
Translocation of glycogen synt ...... ndent anion channel 2 (VDAC2).
@ast
Translocation of glycogen synt ...... ndent anion channel 2 (VDAC2).
@en
P2093
P2860
P356
P1476
Translocation of glycogen synt ...... endent anion channel 2 (VDAC2)
@en
P2093
Hidemichi Kouzu
Hiromichi Murase
Makoto Ogasawara
Masaya Tanno
Satoko Ishikawa
Takayuki Miki
Tetsuji Miura
Toshiyuki Tobisawa
Toshiyuki Yano
P2860
P304
29285-29296
P356
10.1074/JBC.M114.563924
P407
P577
2014-09-03T00:00:00Z