Endoplasmic reticulum stress in diabetic hearts abolishes erythropoietin-induced myocardial protection by impairment of phospho-glycogen synthase kinase-3beta-mediated suppression of mitochondrial permeability transition
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Mitochondria in the diabetic heartEndoplasmic reticulum stress and diabetic cardiomyopathyThe Unexpected Uses of Urso- and Tauroursodeoxycholic Acid in the Treatment of Non-liver DiseasesIschaemic conditioning and targeting reperfusion injury: a 30 year voyage of discoveryDiabetic cardiomyopathy: pathophysiology and clinical featuresTranslocation of glycogen synthase kinase-3β (GSK-3β), a trigger of permeability transition, is kinase activity-dependent and mediated by interaction with voltage-dependent anion channel 2 (VDAC2).Protein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease.Pharmacological postconditioning with atorvastatin calcium attenuates myocardial ischemia/reperfusion injury in diabetic rats by phosphorylating GSK3β.Deficiency of rac1 blocks NADPH oxidase activation, inhibits endoplasmic reticulum stress, and reduces myocardial remodeling in a mouse model of type 1 diabetes.A single intramuscular injection of rAAV-mediated mutant erythropoietin protects against MPTP-induced parkinsonism.Role of ER stress in ventricular contractile dysfunction in type 2 diabetesIschemic conditioning: the challenge of protecting the diabetic heart.Mitochondrial kinase signalling pathways in myocardial protection from ischaemia/reperfusion-induced necrosis.Tauroursodeoxycholic acid mitigates high fat diet-induced cardiomyocyte contractile and intracellular Ca2+ anomalies.Stem cells in the diabetic infarcted heartHydrogen sulfide attenuates high fat diet-induced cardiac dysfunction via the suppression of endoplasmic reticulum stress.Type 2 diabetes mellitus and myocardial ischemic preconditioning in symptomatic coronary artery disease patients.The diabetic heart: too sweet for its own good?Inhibition of DPP-4 reduces acute mortality after myocardial infarction with restoration of autophagic response in type 2 diabetic rats.Effects of diabetes on myocardial infarct size and cardioprotection by preconditioning and postconditioning.Diabetic cardiomyopathy and metabolic remodeling of the heart.Obesity induces hypothalamic endoplasmic reticulum stress and impairs proopiomelanocortin (POMC) post-translational processing.JNK/PI3K/Akt signaling pathway is involved in myocardial ischemia/reperfusion injury in diabetic rats: effects of salvianolic acid A intervention.Long-term insulin treatment restores cardioprotection induced by sufentanil postconditioning in diabetic rat heart.Tauroursodeoxycholic acid inhibits endoplasmic reticulum stress, blocks mitochondrial permeability transition pore opening, and suppresses reperfusion injury through GSK-3ß in cardiac H9c2 cells.Cardiovascular effects of erythropoietin an update.Endoplasmic reticulum stress contributes to aortic stiffening via proapoptotic and fibrotic signaling mechanismsThe SR-mitochondria interaction: a new player in cardiac pathophysiology.Activation of Akt rescues endoplasmic reticulum stress-impaired murine cardiac contractile function via glycogen synthase kinase-3β-mediated suppression of mitochondrial permeation pore opening.Induction of endoplasmic reticulum stress and changes in expression levels of Zn(2+)-transporters in hypertrophic rat heart.Insufficient activation of Akt upon reperfusion because of its novel modification by reduced PP2A-B55α contributes to enlargement of infarct size by chronic kidney disease.Insights for Oxidative Stress and mTOR Signaling in Myocardial Ischemia/Reperfusion Injury under Diabetes.Thapsigargin triggers cardiac contractile dysfunction via NADPH oxidase-mediated mitochondrial dysfunction: Role of Akt dephosphorylation.Interplay Between Cytosolic Free Zn2+ and Mitochondrion Morphological Changes in Rat Ventricular Cardiomyocytes.Zn2+ and mPTP Mediate Endoplasmic Reticulum Stress Inhibition-Induced Cardioprotection Against Myocardial Ischemia/Reperfusion Injury.Intracellular Zn(2+) Increase in Cardiomyocytes Induces both Electrical and Mechanical Dysfunction in Heart via Endogenous Generation of Reactive Nitrogen Species.Akt and mitogen-activated protein kinase enhance C-type lectin-like receptor 2-mediated platelet activation by inhibition of glycogen synthase kinase 3α/β.Mitochondrial aldehyde dehydrogenase obliterates endoplasmic reticulum stress-induced cardiac contractile dysfunction via correction of autophagy.Diabetes mellitus abrogates the cardioprotection of sufentanil against ischaemia/reperfusion injury by altering glycogen synthase kinase-3β.Hexokinase cellular trafficking in ischemia-reperfusion and ischemic preconditioning is altered in type I diabetic heart.
P2860
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P2860
Endoplasmic reticulum stress in diabetic hearts abolishes erythropoietin-induced myocardial protection by impairment of phospho-glycogen synthase kinase-3beta-mediated suppression of mitochondrial permeability transition
description
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2009
@ast
im Dezember 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/12/01)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/12/01)
@nl
наукова стаття, опублікована в грудні 2009
@uk
مقالة علمية (نشرت في ديسمبر 2009)
@ar
name
Endoplasmic reticulum stress i ...... ndrial permeability transition
@ast
Endoplasmic reticulum stress i ...... ndrial permeability transition
@en
Endoplasmic reticulum stress i ...... ndrial permeability transition
@nl
type
label
Endoplasmic reticulum stress i ...... ndrial permeability transition
@ast
Endoplasmic reticulum stress i ...... ndrial permeability transition
@en
Endoplasmic reticulum stress i ...... ndrial permeability transition
@nl
prefLabel
Endoplasmic reticulum stress i ...... ndrial permeability transition
@ast
Endoplasmic reticulum stress i ...... ndrial permeability transition
@en
Endoplasmic reticulum stress i ...... ndrial permeability transition
@nl
P2093
P2860
P921
P3181
P356
P1433
P1476
Endoplasmic reticulum stress i ...... ndrial permeability transition
@en
P2093
Akifumi Takada
Hiroyuki Hotta
Kazuaki Shimamoto
Masaya Tanno
Satoko Ishikawa
Takahiro Sato
Takayuki Miki
Tetsuji Miura
Toshiyuki Yano
Yoshiaki Terashima
P2860
P304
P3181
P356
10.2337/DB09-0158
P407
P577
2009-12-01T00:00:00Z