VTA CRF neurons mediate the aversive effects of nicotine withdrawal and promote intake escalation. - Wikidata - wikimedia - TriplyDB

VTA CRF neurons mediate the aversive effects of nicotine withdrawal and promote intake escalation.

VTA CRF neurons mediate the aversive effects of nicotine withdrawal and promote intake escalation.

http://www.wikidata.org/entity/Q30601106

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Diversity of Dopaminergic Neural Circuits in Response to Drug Exposure Neurobiology of addiction: a neurocircuitry analysis.Increased CRF signalling in a ventral tegmental area-interpeduncular nucleus-medial habenula circuit induces anxiety during nicotine withdrawal Identifying candidate drivers of alcohol dependence-induced excessive drinking by assembly and interrogation of brain-specific regulatory networks GIRK3 gates activation of the mesolimbic dopaminergic pathway by ethanol.Stress is a principal factor that promotes tobacco use in females CRF antagonism within the ventral tegmental area but not the extended amygdala attenuates the anxiogenic effects of cocaine in rats.Diversity of Reporter Expression Patterns in Transgenic Mouse Lines Targeting Corticotropin-Releasing Hormone-Expressing Neurons A Transgenic Rat for Investigating the Anatomy and Function of Corticotrophin Releasing Factor Circuits Cigarettes and alcohol: The influence of nicotine on operant alcohol self-administration and the mesolimbic dopamine system.Multiplexed neurochemical signaling by neurons of the ventral tegmental area.Episodic Social Stress-Escalated Cocaine Self-Administration: Role of Phasic and Tonic Corticotropin Releasing Factor in the Anterior and Posterior Ventral Tegmental Area.Glutamate neurons are intermixed with midbrain dopamine neurons in nonhuman primates and humans Social Stress-Induced Alterations in CRF Signaling in the VTA Facilitate the Emergence of Addiction-like Behavior.Control of chronic excessive alcohol drinking by genetic manipulation of the Edinger-Westphal nucleus urocortin-1 neuropeptide system.Understanding opioid reward.The CRF family of neuropeptides and their receptors - mediators of the central stress response.The CRF System as a Therapeutic Target for Neuropsychiatric Disorders.Anxiety and Nicotine Dependence: Emerging Role of the Habenulo-Interpeduncular Axis.Neuropeptide systems and new treatments for nicotine addiction.Ventral tegmental area: cellular heterogeneity, connectivity and behaviour.Extended Amygdala to Ventral Tegmental Area Corticotropin-Releasing Factor Circuit Controls Binge Ethanol Intake.Habenula cholinergic neurons regulate anxiety during nicotine withdrawal via nicotinic acetylcholine receptors.Deletion of α5 nicotine receptor subunits abolishes nicotinic aversive motivational effects in a manner that phenocopies dopamine receptor antagonism.Perturbations in Effort-Related Decision-Making Driven by Acute Stress and Corticotropin-Releasing Factor.Social stress-escalated intermittent alcohol drinking: modulation by CRF-R1 in the ventral tegmental area and accumbal dopamine in mice.Individual differences in the neuropsychopathology of addiction.An enzymatic advance in nicotine cessation therapy.A single administration of the hallucinogen, 4-acetoxy-dimethyltryptamine, prevents the shift to a drug-dependent state and the expression of withdrawal aversions in rodents.The Dark Side of Addiction: The Horsley Gantt to Joseph Brady Connection.Escalated cocaine "binges" in rats: enduring effects of social defeat stress or intra-VTA CRF.Corticotropin-Releasing Factor (CRF) and Addictive Behaviors.Distribution of corticotropin-releasing factor neurons in the mouse brain: a study using corticotropin-releasing factor-modified yellow fluorescent protein knock-in mouse.A role for corticotropin-releasing factor signaling in the lateral habenula and its modulation by early-life stress.Nicotine aversion is mediated by GABAergic interpeduncular nucleus inputs to laterodorsal tegmentum.

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VTA CRF neurons mediate the aversive effects of nicotine withdrawal and promote intake escalation.

http://www.wikidata.org/entity/Q30601106

description

2014 nî lūn-bûn

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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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VTA CRF neurons mediate the av ...... and promote intake escalation.

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VTA CRF neurons mediate the av ...... and promote intake escalation.

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VTA CRF neurons mediate the av ...... and promote intake escalation.

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VTA CRF neurons mediate the av ...... and promote intake escalation.

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Nature Neuroscience

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VTA CRF neurons mediate the av ...... and promote intake escalation

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P2093

Ami Cohen

http://www.w3.org/2001/XMLSchema#string

Andrew R Tapper

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Brigitte L Kieffer

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Candice Contet

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Derek van der Kooy

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Elena Crawford

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Geith Maal-Bared

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George F Koob

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Hector Vargas-Perez

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Joel E Schlosburg

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P2860

Ventral tegmental area BDNF induces an opiate-dependent-like reward state in naive rats

Neurocircuitry of addiction

Molecular mechanisms underlying behaviors related to nicotine addiction

Excessive cocaine use results from decreased phasic dopamine signaling in the striatum

Cellular and molecular mechanisms of drug dependence

Corticotropin-releasing factor binding protein within the ventral tegmental area is expressed in a subset of dopaminergic neurons

Augmented cocaine seeking in response to stress or CRF delivered into the ventral tegmental area following long-access self-administration is mediated by CRF receptor type 1 but not CRF receptor type 2

Nicotine dependence produces hyperalgesia: role of corticotropin-releasing factor-1 receptors (CRF1Rs) in the central amygdala (CeA).

The Edinger-Westphal nucleus: a historical, structural, and functional perspective on a dichotomous terminology.

Prevention of social stress-escalated cocaine self-administration by CRF-R1 antagonist in the rat VTA.

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1751-1758

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10.1038/NN.3872

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12

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268449756

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25402857

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4241147

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