Prevention of social stress-escalated cocaine self-administration by CRF-R1 antagonist in the rat VTA. - Wikidata - wikimedia - TriplyDB

Prevention of social stress-escalated cocaine self-administration by CRF-R1 antagonist in the rat VTA.

Prevention of social stress-escalated cocaine self-administration by CRF-R1 antagonist in the rat VTA.

http://www.wikidata.org/entity/Q30461988

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Stress in adolescence and drugs of abuse in rodent models: role of dopamine, CRF, and HPA axis Emotional valence and context of social influences on drug abuse-related behavior in animal models of social stress and prosocial interaction Stress-induced sensitization to cocaine: actin cytoskeleton remodeling within mesocorticolimbic nuclei Social stress and escalated drug self-administration in mice I. Alcohol and corticosterone Individual differences and social influences on the neurobehavioral pharmacology of abused drugs.Stress modulation of drug self-administration: implications for addiction comorbidity with post-traumatic stress disorder.Update on corticotropin-releasing factor pharmacotherapy for psychiatric disorders: a revisionist view.VTA CRF neurons mediate the aversive effects of nicotine withdrawal and promote intake escalation.Social stress and CRF-dopamine interactions in the VTA: role in long-term escalation of cocaine self-administration Corticotropin releasing factor: a key role in the neurobiology of addiction The dark side of emotion: the addiction perspective Social defeat stress-induced sensitization and escalated cocaine self-administration: the role of ERK signaling in the rat ventral tegmental area.Adolescent social defeat increases adult amphetamine conditioned place preference and alters D2 dopamine receptor expression Escalation of cocaine self-administration in adulthood after social defeat of adolescent rats: role of social experience and adaptive coping behavior Area-specific analysis of the distribution of hypothalamic neurons projecting to the rat ventral tegmental area, with special reference to the GABAergic and glutamatergic efferents.Interaction of CRF and kappa opioid systems on GABAergic neurotransmission in the mouse central amygdala.Alcohol in excess: CRF₁ receptors in the rat and mouse VTA and DRN.Depressive and cardiovascular disease comorbidity in a rat model of social stress: a putative role for corticotropin-releasing factor.Glutamatergic mechanisms of comorbidity between acute stress and cocaine self-administration Sex differences in behavioral and neural cross-sensitization and escalated cocaine taking as a result of episodic social defeat stress in rats CRF type 1 receptor antagonism in ventral tegmental area of adolescent rats during social defeat: prevention of escalated cocaine self-administration in adulthood and behavioral adaptations during adolescence Addiction is a Reward Deficit and Stress Surfeit Disorder.Addiction as a stress surfeit disorder.Addiction and corticotropin-releasing hormone type 1 receptor antagonist medications.Assessment of a proposed "three-criteria" cocaine addiction model for use in reinstatement studies with rats.Individual differences in the neurobiology of social stress: implications for depression-cardiovascular disease comorbidity The CRF family of neuropeptides and their receptors - mediators of the central stress response.Depression and substance use comorbidity: What we have learned from animal studies.Cross-sensitization between cocaine and acute restraint stress is associated with sensitized dopamine but not glutamate release in the nucleus accumbens.Social stress-escalated intermittent alcohol drinking: modulation by CRF-R1 in the ventral tegmental area and accumbal dopamine in mice.Escalated cocaine "binges" in rats: enduring effects of social defeat stress or intra-VTA CRF.Prevention and reversal of social stress-escalated cocaine self-administration in mice by intra-VTA CRFR1 antagonism.Corticotropin-Releasing Factor (CRF) and Addictive Behaviors.Social defeat stress and escalation of cocaine and alcohol consumption: Focus on CRF

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P2860

Prevention of social stress-escalated cocaine self-administration by CRF-R1 antagonist in the rat VTA.

http://www.wikidata.org/entity/Q30461988

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2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Prevention of social stress-es ...... -R1 antagonist in the rat VTA.

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Prevention of social stress-es ...... -R1 antagonist in the rat VTA.

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Prevention of social stress-es ...... -R1 antagonist in the rat VTA.

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Psychopharmacology

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Prevention of social stress-es ...... F-R1 antagonist in the rat VTA

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Christopher O Boyson

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Joseph F Debold

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Klaus A Miczek

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Tarciso T Miguel

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P2860

Corticotropin-releasing factor requires CRF binding protein to potentiate NMDA receptors via CRF receptor 2 in dopamine neurons

CP-154,526: a potent and selective nonpeptide antagonist of corticotropin releasing factor receptors.

Social defeat as a stressor in humans

Corticotropin-releasing factor binding protein within the ventral tegmental area is expressed in a subset of dopaminergic neurons

CRF1 and CRF2 receptors are required for potentiated startle to contextual but not discrete cues

CRF(1) receptor antagonists attenuate escalated cocaine self-administration in rats

Effects of the CRH receptor antagonist CP-154,526 on intravenous cocaine self-administration in rats.

125I-Antisauvagine-30: a novel and specific high-affinity radioligand for the characterization of corticotropin-releasing factor type 2 receptors.

Cocaine experience establishes control of midbrain glutamate and dopamine by corticotropin-releasing factor: a role in stress-induced relapse to drug seeking.

The HPA axis and cocaine reinforcement.

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