Modulation of DNA repair processes by arsenic and selenium compounds.
about
The influence of micronutrients in cell culture: a reflection on viability and genomic stabilityPoly(ADP-ribose) polymerase-1 inhibition by arsenite promotes the survival of cells with unrepaired DNA lesions induced by UV exposureIntracellular calcium disturbances induced by arsenic and its methylated derivatives in relation to genomic damage and apoptosis induction.Advances in carcinogenic metal toxicity and potential molecular markersReview of Cisplatin and oxaliplatin in current immunogenic and monoclonal antibody treatmentsReduction of arsenite-enhanced ultraviolet radiation-induced DNA damage by supplemental zincLow concentration of arsenite exacerbates UVR-induced DNA strand breaks by inhibiting PARP-1 activityInhibition of poly(ADP-ribose) polymerase-1 by arsenite interferes with repair of oxidative DNA damageInter-individual variation in DNA repair capacity: a need for multi-pathway functional assays to promote translational DNA repair researchArsenic binding to proteinsArsenite interacts selectively with zinc finger proteins containing C3H1 or C4 motifsLiver is a target of arsenic carcinogenesisArsenic exposure and toxicology: a historical perspectiveArsenic biotransformation as a cancer promoting factor by inducing DNA damage and disruption of repair mechanismsKinetics and thermodynamics of zinc(II) and arsenic(III) binding to XPA and PARP-1 zinc finger peptidesCombination of bifunctional alkylating agent and arsenic trioxide synergistically suppresses the growth of drug-resistant tumor cellsStructure, function, and evolution of the Thiomonas spp. genomeEffect of prenatal arsenic exposure on DNA methylation and leukocyte subpopulations in cord blood.Distinct signaling pathways respond to arsenite and reactive oxygen species in Schizosaccharomyces pombe.Arsenite binds to the RING finger domains of RNF20-RNF40 histone E3 ubiquitin ligase and inhibits DNA double-strand break repair.Response to arsenate treatment in Schizosaccharomyces pombe and the role of its arsenate reductase activity.Multiplex pyrosequencing of two polymorphisms in DNA repair gene XRCC1.Prenatal arsenic exposure and drowning among children in Bangladesh.As(III) inhibits ultraviolet radiation-induced cyclobutane pyrimidine dimer repair via generation of nitric oxide in human keratinocytes.Arsenite-loaded nanoparticles inhibit PARP-1 to overcome multidrug resistance in hepatocellular carcinoma cellsMetal-on-metal hip resurfacing arthroplasty: a review of periprosthetic biological reactions.Prepubertal exposure to arsenic(III) suppresses circulating insulin-like growth factor-1 (IGF-1) delaying sexual maturation in female rats.Differential binding of monomethylarsonous acid compared to arsenite and arsenic trioxide with zinc finger peptides and proteins.Zinc finger proteins as templates for metal ion exchange and ligand reactivity. Chemical and biological consequences.Genetic and epigenetic effects of environmental arsenicals.Interdependent genotoxic mechanisms of monomethylarsonous acid: role of ROS-induced DNA damage and poly(ADP-ribose) polymerase-1 inhibition in the malignant transformation of urothelial cellsXPC silencing sensitizes glioma cells to arsenic trioxide via increased oxidative damage.The repressive effect of NF-kappaB on p53 by mot-2 is involved in human keratinocyte transformation induced by low levels of arsenite.Persistence of DNA damage following exposure of human bladder cells to chronic monomethylarsonous acidBRCA2-dependent homologous recombination is required for repair of Arsenite-induced replication lesions in mammalian cells.Arsenite-induced alterations in Ku70-deficient cells: a model to study genotoxic effects.Arsenic-induced promoter hypomethylation and over-expression of ERCC2 reduces DNA repair capacity in humans by non-disjunction of the ERCC2-Cdk7 complex.Ogg1 genetic background determines the genotoxic potential of environmentally relevant arsenic exposures.Review of Cisplatin and Oxaliplatin in Current Immunogenic and Monoclonal Antibodies Perspective.Dimethylarsinic acid in drinking water changed the morphology of urinary bladder but not the expression of DNA repair genes of bladder transitional epithelium in F344 rats.
P2860
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P2860
Modulation of DNA repair processes by arsenic and selenium compounds.
description
2003 nî lūn-bûn
@nan
2003 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Modulation of DNA repair processes by arsenic and selenium compounds.
@ast
Modulation of DNA repair processes by arsenic and selenium compounds.
@en
type
label
Modulation of DNA repair processes by arsenic and selenium compounds.
@ast
Modulation of DNA repair processes by arsenic and selenium compounds.
@en
prefLabel
Modulation of DNA repair processes by arsenic and selenium compounds.
@ast
Modulation of DNA repair processes by arsenic and selenium compounds.
@en
P2093
P1433
P1476
Modulation of DNA repair processes by arsenic and selenium compounds.
@en
P2093
Blessing H
Schwerdtle T
P304
P356
10.1016/J.TOX.2003.08.004
P407
P577
2003-11-01T00:00:00Z