PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
about
Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward.Pdx1-Cre-driven conditional gene depletion suggests PAK4 as dispensable for mouse pancreas development.Targeting Rho GTPase effector p21 activated kinase 4 (PAK4) suppresses p-Bad-microRNA drug resistance axis leading to inhibition of pancreatic ductal adenocarcinoma proliferation.
P2860
PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
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2017 nî lūn-bûn
@nan
2017 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2017 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
name
PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
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PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
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label
PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
@ast
PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
@en
prefLabel
PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
@ast
PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
@en
P2093
P2860
P356
P1433
P1476
PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration
@en
P2093
Andrew Whale
Claire M Wells
Helen King
Hesham Eldaly
Kiruthikah Thillai
P2860
P2888
P356
10.1038/SREP42575
P407
P577
2017-02-16T00:00:00Z
P6179
1083843219