Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway - Wikidata - wikimedia - TriplyDB

Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

http://www.wikidata.org/entity/Q30840240

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Toll Like Receptors Signaling Pathways as a Target for Therapeutic Interventions Poly(I:C) induces intense expression of c-IAP2 and cooperates with an IAP inhibitor in induction of apoptosis in cancer cells.Combinatorial treatment with polyI:C and anti-IL6 enhances apoptosis and suppresses metastasis of lung cancer cells.Serial selection for invasiveness increases expression of miR-143/miR-145 in glioblastoma cell lines.miRNA-125b regulates TNF-α production in CD14+ neonatal monocytes via post-transcriptional regulation.The ribonucleotide reductase R1 subunits of herpes simplex virus 1 and 2 protect cells against poly(I · C)-induced apoptosis Differential toll-like receptor 3 (TLR3) expression and apoptotic response to TLR3 agonist in human neuroblastoma cells.Macrophages help NK cells to attack tumor cells by stimulatory NKG2D ligand but protect themselves from NK killing by inhibitory ligand Qa-1.Targeting polyIC to EGFR over-expressing cells using a dsRNA binding protein domain tethered to EGF.Periodic-shRNA molecules are capable of gene silencing, cytotoxicity and innate immune activation in cancer cells TLR3, TRIF, and caspase 8 determine double-stranded RNA-induced epithelial cell death and survival in vivo The proteome of Toll-like receptor 3-stimulated human immortalized fibroblasts: implications for susceptibility to herpes simplex virus encephalitis Localized irradiation of tumors prior to synthetic dsRNA therapy enhanced the resultant anti-tumor activity Apoptosis is induced in Chlamydia trachomatis-infected HEp-2 cells by the addition of a combination innate immune activation compounds and the inhibitor wedelolactone.Roles of toll-like receptors in cancer: a double-edged sword for defense and offense.Toll-like receptors in human papillomavirus infection.Toll-like receptor agonists: current status and future perspective on their utility as adjuvants in improving anticancer vaccination strategies.Association between Toll-like receptor 3 polymorphisms and cancer risk: a meta-analysis.Yeast virus-derived stimulator of the innate immune system augments the efficacy of virus vector-based immunotherapy.Antitumor activity from the combined application of poly(I:C) and chemotherapeutics in human metastatic pharyngeal cell lines.Poly I:C inhibits cell proliferation and enhances the growth inhibitory effect of paclitaxel in oral squamous cell carcinoma.The dual role of TLR3 in metastatic cell line.Combination of Poly I:C and arsenic trioxide triggers apoptosis synergistically via activation of TLR3 and mitochondrial pathways in hepatocellular carcinoma cells.Proapoptotic signalling through Toll-like receptor-3 involves TRIF-dependent activation of caspase-8 and is under the control of inhibitor of apoptosis proteins in melanoma cells.Evaluation of Toll-like receptors 3 (c.1377C/T) and 9 (G2848A) gene polymorphisms in cervical cancer susceptibility.Poly (I:C) enhances the anti-tumor activity of canine parvovirus NS1 protein by inducing a potent anti-tumor immune response.TLR-10 polymorphism and papillary thyroid cancer: one more SNP to consider?Double-stranded RNA induces melanocyte death via activation of Toll-like receptor 3.TRIM56 Suppresses Multiple Myeloma Progression by Activating TLR3/TRIF Signaling.Bufalin Suppresses Migration and Invasion of Hepatocellular Carcinoma Cells Elicited by Poly (I:C) Therapy.Soluble TNF-like weak inducer of apoptosis (TWEAK) enhances poly(I:C)-induced RIPK1-mediated necroptosis Modulation of Apoptosis by Cytotoxic Mediators and Cell-Survival Molecules in Sjögren's Syndrome

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Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

http://www.wikidata.org/entity/Q30840240

description

2008 nî lūn-bûn

@nan

2008 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հունվարին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

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Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

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type

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Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

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Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

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prefLabel

Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

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Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

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Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway

@en

P2093

Haiming Wei

http://www.w3.org/2001/XMLSchema#string

Qun Jiang

http://www.w3.org/2001/XMLSchema#string

Zhigang Tian

http://www.w3.org/2001/XMLSchema#string

P2860

Cell activation and apoptosis by bacterial lipoproteins through toll-like receptor-2

IFNs activate toll-like receptor gene expression in viral infections

Toll-like receptor signalling

Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses

The RNA helicase RIG-I has an essential function in double-stranded RNA-induced innate antiviral responses

Mechanisms of beta-cell death in response to double-stranded (ds) RNA and interferon-gamma: dsRNA-dependent protein kinase apoptosis and nitric oxide-dependent necrosis

Rotavirus double-stranded RNA induces apoptosis and diminishes wound repair in rat intestinal epithelial cells

Apoptosis induced by the toll-like receptor adaptor TRIF is dependent on its receptor interacting protein homotypic interaction motif

Apoptosis and interferons: role of interferon-stimulated genes as mediators of apoptosis

Apoptosis: an innate immune response to virus infection.

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10.1186/1471-2407-8-12

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8

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