Glioblastoma subclasses can be defined by activity among signal transduction pathways and associated genomic alterations. - Wikidata - wikimedia - TriplyDB

Glioblastoma subclasses can be defined by activity among signal transduction pathways and associated genomic alterations.

Glioblastoma subclasses can be defined by activity among signal transduction pathways and associated genomic alterations.

http://www.wikidata.org/entity/Q30885944

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Large-scale data integration framework provides a comprehensive view on glioblastoma multiforme Neural stem cells: brain building blocks and beyond Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1 Radioresistance of Brain Tumors In vivo models of brain tumors: roles of genetically engineered mouse models in understanding tumor biology and use in preclinical studies Mouse models to interrogate the implications of the differentiation status in the ontogeny of gliomas Functional roles and clinical values of insulin-like growth factor-binding protein-5 in different types of cancers Mouse models of cancer: Sleeping Beauty transposons for insertional mutagenesis screens and reverse genetic studies Specific visualization of glioma cells in living low-grade tumor tissue The somatic genomic landscape of glioblastoma The future role of personalized medicine in the treatment of glioblastoma multiforme Hypoxia-inducing factors as master regulators of stemness properties and altered metabolism of cancer- and metastasis-initiating cells TMEFF2 is a PDGF-AA binding protein with methylation-associated gene silencing in multiple cancer types including glioma Perifosine and CCI 779 co-operate to induce cell death and decrease proliferation in PTEN-intact and PTEN-deficient PDGF-driven murine glioblastoma Advances in diagnostic and treatment modalities for intracranial tumors.Transformation of quiescent adult oligodendrocyte precursor cells into malignant glioma through a multistep reactivation process BTECH: a platform to integrate genomic, transcriptomic and epigenomic alterations in brain tumors Cooperativity between MAPK and PI3K signaling activation is required for glioblastoma pathogenesis.An integrative characterization of recurrent molecular aberrations in glioblastoma genomes.microRNA regulatory network inference identifies miR-34a as a novel regulator of TGF-β signaling in glioblastoma.Gene set based integrated data analysis reveals phenotypic differences in a brain cancer model.Bayesian methods for expression-based integration of various types of genomics data.Clinical relevance of tumor cells with stem-like properties in pediatric brain tumors.Cell cycle and aging, morphogenesis, and response to stimuli genes are individualized biomarkers of glioblastoma progression and survival.The interference of Notch1 target Hes1 affects cell growth, differentiation and invasiveness of glioblastoma stem cells through modulation of multiple oncogenic targets Neuro-oncology: unmasking the multiforme in glioblastoma.GliomaPredict: a clinically useful tool for assigning glioma patients to specific molecular subtypes.PDGF-B-driven gliomagenesis can occur in the absence of the proteoglycan NG2 Notch signaling in glioblastoma: a developmental drug target?Detecting independent and recurrent copy number aberrations using interval graphs.AKT pathway genes define 5 prognostic subgroups in glioblastoma.Inhibition of Notch signaling alters the phenotype of orthotopic tumors formed from glioblastoma multiforme neurosphere cells but does not hamper intracranial tumor growth regardless of endogene Notch pathway signature Glioblastoma models reveal the connection between adult glial progenitors and the proneural phenotype hMOB3 modulates MST1 apoptotic signaling and supports tumor growth in glioblastoma multiforme.Review: molecular pathology in adult high-grade gliomas: from molecular diagnostics to target therapies Activated MEK cooperates with Ink4a/Arf loss or Akt activation to induce gliomas in vivo Recruited cells can become transformed and overtake PDGF-induced murine gliomas in vivo during tumor progression Why is there a lack of consensus on molecular subgroups of glioblastoma? Understanding the nature of biological and statistical variability in glioblastoma expression data.Kinomic exploration of temozolomide and radiation resistance in Glioblastoma multiforme xenolines.A molecular screening approach to identify and characterize inhibitors of glioblastoma stem cells

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P2860

Glioblastoma subclasses can be defined by activity among signal transduction pathways and associated genomic alterations.

http://www.wikidata.org/entity/Q30885944

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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Glioblastoma subclasses can be ...... ssociated genomic alterations.

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Glioblastoma subclasses can be ...... ssociated genomic alterations.

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Glioblastoma subclasses can be ...... ssociated genomic alterations.

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Glioblastoma subclasses can be ...... ssociated genomic alterations.

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Alicia Pedraza

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Dolores Hambardzumyan

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Eric Holland

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Hiroyuki Momota

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Tatsuya Ozawa

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P2860

Gene expression profiling reveals molecularly and clinically distinct subtypes of glioblastoma multiforme

An integrated genomic analysis of human glioblastoma multiforme

IDH1 and IDH2 mutations in gliomas

Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

The 2007 WHO classification of tumours of the central nervous system

Epidermal growth factor receptor activation in glioblastoma through novel missense mutations in the extracellular domain

Circular binary segmentation for the analysis of array-based DNA copy number data

Malignant gliomas in adults

Malignant astrocytic glioma: genetics, biology, and paths to treatment

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Cameron Brennan

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2771920

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