Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate. - Wikidata - wikimedia - TriplyDB

Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate.

Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate.

http://www.wikidata.org/entity/Q31076633

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Uremic Toxins - Novel Arrhythmogenic Factor in Chronic Kidney Disease - Related Atrial Fibrillation Animal Models to Study Links between Cardiovascular Disease and Renal Failure and Their Relevance to Human Pathology Chronic Kidney Disease and Fibrosis: The Role of Uremic Retention Solutes Gut Bacteria and Hydrogen Sulfide: The New Old Players in Circulatory System Homeostasis Sodium potassium adenosine triphosphatase (Na/K-ATPase) as a therapeutic target for uremic cardiomyopathy.Speckle tracking echocardiography detects uremic cardiomyopathy early and predicts cardiovascular mortality in ESRD.Left ventricular myocardial function in hemodialysis and nondialysis uremia patients: a three-dimensional speckle-tracking echocardiography study Decrease in irisin in patients with chronic kidney disease Association of systemic collagen type IV formation with survival among patients undergoing hemodialysis.The uremic toxin adsorbent AST-120 abrogates cardiorenal injury following myocardial infarction.Uremic retention solute indoxyl sulfate level is associated with prolonged QTc interval in early CKD patients Contribution of microRNA to pathological fibrosis in cardio-renal syndrome: impact of uremic toxins Intersections Between Microbiome and Heart Failure: Revisiting the Gut Hypothesis.Combined Iron Chelator and Antioxidant Exerted Greater Efficacy on Cardioprotection Than Monotherapy in Iron-Overloaded Rats Klotho Protects Against Indoxyl Sulphate-Induced Myocardial Hypertrophy.Green tea inhibited the elimination of nephro-cardiovascular toxins and deteriorated the renal function in rats with renal failure.Vagus Nerve Stimulation Improves Cardiac Function by Preventing Mitochondrial Dysfunction in Obese-Insulin Resistant Rats.Hepatic sulfotransferase as a nephropreventing target by suppression of the uremic toxin indoxyl sulfate accumulation in ischemic acute kidney injury.Role of Indoxyl Sulfate as a Predisposing Factor for Atrial Fibrillation in Renal Dysfunction.Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.Sustained uremic toxin control improves renal and cardiovascular outcomes in patients with advanced renal dysfunction: post-hoc analysis of the Kremezin Study against renal disease progression in Korea.Targeting protein-bound uremic toxins in chronic kidney disease.Gadolinium and nephrogenic systemic fibrosis: an update.Cardiorenal syndrome: acute kidney injury secondary to cardiovascular disease and role of protein-bound uraemic toxins.Protein-bound uremic toxins: a long overlooked culprit in cardiorenal syndrome.MicroRNAs mediate the cardioprotective effect of angiotensin-converting enzyme inhibition in acute kidney injury.Indoxyl sulfate, a valuable biomarker in chronic kidney disease and dialysis.Indoxyl Sulfate: A Novel Cardiovascular Risk Factor in Chronic Kidney Disease.Novel Concept of a Heart-Gut Axis in the Pathophysiology of Heart Failure.Impaired mitochondria and intracellular calcium transients in the salivary glands of obese rats.Murine Models of Heart Failure with Preserved Ejection Fraction: a "Fishing Expedition".Antioxidant and renoprotective activity of 2-hydroxypropyl-β-cyclodextrin in nephrectomized rats.Challenges to advancing the evidence base for nephrology: the time is right for collaboration.Role of Uremic Toxins for Kidney, Cardiovascular, and Bone Dysfunction.Gut Microbiota and Cardiovascular Uremic Toxicities Impacts of Indoxyl Sulfate and p-Cresol Sulfate on Chronic Kidney Disease and Mitigating Effects of AST-120 Cardiotoxicity of Uremic Toxins: A Driver of Cardiorenal Syndrome

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Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate.

http://www.wikidata.org/entity/Q31076633

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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2012 թվականի հուլիսին հրատարակված գիտական հոդված

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2012年の論文

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Chronic kidney disease-induced ...... uremic toxin, indoxyl sulfate.

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Andrew R Kompa

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Bing H Wang

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Darren J Kelly

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Fuyuhiko Nishijima

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Minako Manabe

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Robyn G Langham

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Suree Lekawanvijit

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P2860

Metabolomics analysis reveals large effects of gut microflora on mammalian blood metabolites

Kidney disease as a risk factor for development of cardiovascular disease: a statement from the American Heart Association Councils on Kidney in Cardiovascular Disease, High Blood Pressure Research, Clinical Cardiology, and Epidemiology and Preventi

Epidemiology of cardiovascular disease in chronic renal disease.

Inflammatory mediators and the failing heart: past, present, and the foreseeable future.

Inflammation, kidney function and albuminuria in the Framingham Offspring cohort.

Molecular mechanisms and therapeutic strategies of chronic renal injury: the role of nuclear factor kappaB activation in the development of renal fibrosis.

Inflammation in end-stage renal disease: the hidden enemy.

Sudden cardiac death and dialysis patients.

Hypertrophy and fibrosis in the cardiomyopathy of uremia--beyond coronary heart disease.

Serum indoxyl sulfate is associated with vascular disease and mortality in chronic kidney disease patients.

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