Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction. - Wikidata - wikimedia - TriplyDB

Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

http://www.wikidata.org/entity/Q37084883

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Actinidia chinensis Planch. Improves the Indices of Antioxidant and Anti-Inflammation Status of Type 2 Diabetes Mellitus by Activating Keap1 and Nrf2 via the Upregulation of MicroRNA-424.Role of Uremic Toxins for Kidney, Cardiovascular, and Bone Dysfunction.Citrus aurantium Naringenin Prevents Osteosarcoma Progression and Recurrence in the Patients Who Underwent Osteosarcoma Surgery by Improving Antioxidant Capability.

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Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

http://www.wikidata.org/entity/Q37084883

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 January 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

@en

Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

@nl

type

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Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

@en

Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

@nl

prefLabel

Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

@en

Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

@nl

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British Journal of Pharmacology

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Anti-oxidative effect of AST-120 on kidney injury after myocardial infarction.

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Ayako Fujieda

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Keiji Kono

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Mikio Sugano

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Shinichi Nishi

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Shunsuke Goto

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Sumie Goto

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Yoshiharu Ito

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Yuriko Yonekura

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Yusuke Yamashita

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P2860

Cardiorenal syndrome: the emerging role of protein-bound uremic toxins

The IUPHAR/BPS Guide to PHARMACOLOGY: an expert-driven knowledgebase of drug targets and their ligands

Acute myocardial infarction and renal dysfunction: a high-risk combination

Relation between renal dysfunction and cardiovascular outcomes after myocardial infarction

Chronic kidney disease and the risks of death, cardiovascular events, and hospitalization

The Concise Guide to PHARMACOLOGY 2015/16: Overview

Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate.

Animal research: reporting in vivo experiments: the ARRIVE guidelines.

Indoxyl sulfate, a uremic toxin, downregulates renal expression of Nrf2 through activation of NF-κB.

The uremic toxin adsorbent AST-120 abrogates cardiorenal injury following myocardial infarction.

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1302-1313

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scholarly article

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10.1111/BPH.13417

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