Downregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells. - Wikidata - wikimedia - TriplyDB

Downregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells.

Downregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells.

http://www.wikidata.org/entity/Q31080106

about

Endoplasmic Reticulum Stress in the Diabetic Kidney, the Good, the Bad and the Ugly MicroRNAs in Kidney Transplantation: Living up to Their Expectations?Cross talk between spliceosome and microprocessor defines the fate of pre-mRNA MicroRNAs in acute kidney injury Mini-review: emerging roles of microRNAs in the pathophysiology of renal diseases miR-125b transcriptionally increased by Nrf2 inhibits AhR repressor, which protects kidney from cisplatin-induced injury Mechanisms of metabolic memory and renal hypoxia as a therapeutic target in diabetic kidney disease.MicroRNAs in diabetic nephropathy: functions, biomarkers, and therapeutic targets.Curcumin abates hypoxia-induced oxidative stress based-ER stress-mediated cell death in mouse hippocampal cells (HT22) by controlling Prdx6 and NF-κB regulation.The regulation and function of microRNAs in kidney diseases.MicroRNAs: potential mediators and biomarkers of diabetic complications.Rapamycin-induced modulation of miRNA expression is associated with amelioration of HIV-associated nephropathy (HIVAN).Regulation of the transcriptome by ER stress: non-canonical mechanisms and physiological consequences.MicroRNA in diabetic nephropathy: renin angiotensin, aGE/RAGE, and oxidative stress pathway.Upregulation of MiR-205 under hypoxia promotes epithelial-mesenchymal transition by targeting ASPP2.Screening plasma miRNAs as biomarkers for renal ischemia-reperfusion injury in rats.TBHQ Alleviated Endoplasmic Reticulum Stress-Apoptosis and Oxidative Stress by PERK-Nrf2 Crosstalk in Methamphetamine-Induced Chronic Pulmonary Toxicity The biochemistry and cell biology of aging: metabolic regulation through mitochondrial signaling.Glycative stress and glyoxalase in kidney disease and aging.MicroRNAs in IgA nephropathy.Proteostasis in endoplasmic reticulum--new mechanisms in kidney disease.Diabetic nephropathy--emerging epigenetic mechanisms.Adaptive response to hypoxia and remote ischaemia pre-conditioning: a new hypoxia-inducible factors era in clinical medicine.Induction of miR-3648 Upon ER Stress and Its Regulatory Role in Cell Proliferation RAGE and glyoxalase in kidney disease.HIF-mediated increased ROS from reduced mitophagy and decreased catalase causes neocytolysis.Stress Signal Network between Hypoxia and ER Stress in Chronic Kidney Disease.Epigenetics of kidney disease.Regulation of the unfolded protein response by noncoding RNA.Renal epithelial miR-205 expression correlates with disease severity in a mouse model of congenital obstructive nephropathy.Epigenetic Changes Induced by Hypoxia-Inducible Factor: a Long Way Still To Go as a Target for Therapy?Effect of miR-205 on 3T3-L1 preadipocyte differentiation through targeting to glycogen synthase kinase 3 beta.MicroRNAs as potential prognosticators of neurological outcome in out-of-hospital cardiac arrest patients.The PHD1 oxygen sensor in health and disease.Noncoding RNAs as therapeutic targets in early stage diabetic kidney disease

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P2860

Downregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells.

http://www.wikidata.org/entity/Q31080106

description

2012 nî lūn-bûn

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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հուլիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Downregulation of miR-205 modu ...... resses in renal tubular cells.

@ast

Downregulation of miR-205 modu ...... resses in renal tubular cells.

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type

label

Downregulation of miR-205 modu ...... resses in renal tubular cells.

@ast

Downregulation of miR-205 modu ...... resses in renal tubular cells.

@en

prefLabel

Downregulation of miR-205 modu ...... resses in renal tubular cells.

@ast

Downregulation of miR-205 modu ...... resses in renal tubular cells.

@en

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Downregulation of miR-205 modu ...... resses in renal tubular cells.

@en

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Masaomi Nangaku

http://www.w3.org/2001/XMLSchema#string

Reiko Inagi

http://www.w3.org/2001/XMLSchema#string

Shiyo Muratsu-Ikeda

http://www.w3.org/2001/XMLSchema#string

Takehiko Wada

http://www.w3.org/2001/XMLSchema#string

Tetsuhiro Tanaka

http://www.w3.org/2001/XMLSchema#string

Yoichiro Ikeda

http://www.w3.org/2001/XMLSchema#string

P2860

The nuclear RNase III Drosha initiates microRNA processing

Selective inhibition of hypoxia-inducible factor (HIF) prolyl-hydroxylase 1 mediates neuroprotection against normoxic oxidative death via HIF- and CREB-independent pathways

Hypoxia-inducible mir-210 regulates normoxic gene expression involved in tumor initiation.

MicroRNA-184 antagonizes microRNA-205 to maintain SHIP2 levels in epithelia

MicroRNA-210 controls mitochondrial metabolism during hypoxia by repressing the iron-sulfur cluster assembly proteins ISCU1/2

Mechanisms of post-transcriptional regulation by microRNAs: are the answers in sight?

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

MicroRNAs: genomics, biogenesis, mechanism, and function

High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells

ORP150/HSP12A protects renal tubular epithelium from ischemia-induced cell death

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endoplasmic reticulum

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3408438

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