High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
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TGF-β/Smad signaling in renal fibrosisThe two faces of miR-29MicroRNAs in Diabetic Kidney DiseaseMini-review: emerging roles of microRNAs in the pathophysiology of renal diseasesDownregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells.miR-29a modulates SCD expression and is regulated in response to a saturated fatty acid diet in juvenile genetically improved farmed tilapia (Oreochromis niloticus).MicroRNA-29a promotion of nephrin acetylation ameliorates hyperglycemia-induced podocyte dysfunction.Early second-trimester serum miRNA profiling predicts gestational diabetes mellitus.Hepatocyte growth factor (HGF) inhibits collagen I and IV synthesis in hepatic stellate cells by miRNA-29 inductionHypoxia-induced down-regulation of microRNA-34a promotes EMT by targeting the Notch signaling pathway in tubular epithelial cells.miR-29a suppresses growth and invasion of gastric cancer cells in vitro by targeting VEGF-A.The role of microRNAs in diabetic nephropathy.Metastasis-associated protein 1 induces VEGF-C and facilitates lymphangiogenesis in colorectal cancer.MiR-29a reduces TIMP-1 production by dermal fibroblasts via targeting TGF-β activated kinase 1 binding protein 1, implications for systemic sclerosisCoordinated regulation of extracellular matrix synthesis by the microRNA-29 family in the trabecular meshworkCross-talk between miR-29 and transforming growth factor-betas in trabecular meshwork cells.MicroRNAs 29 are involved in the improvement of ventricular compliance promoted by aerobic exercise training in ratsMicroRNAs in renal fibrosis.Epigenetic regulation of matrix metalloproteinases and their collagen substrates in cancer.Treatment of chronic kidney diseases with histone deacetylase inhibitors.High glucose enhances microRNA-26a to activate mTORC1 for mesangial cell hypertrophy and matrix protein expression.Non-housekeeping genes expressed in human trabecular meshwork cell cultures.MicroRNA and transcription factor mediated regulatory network analysis reveals critical regulators and regulatory modules in myocardial infarctionThe miR-29 family: genomics, cell biology, and relevance to renal and cardiovascular injury.Differential expression and therapeutic efficacy of microRNA-346 in diabetic nephropathy miceDifferent regulation of miR-29a-3p in glomeruli and tubules in an experimental model of angiotensin II-dependent hypertension: potential role in renal fibrosis.MicroRNAs and the glomerulusRecent developments in epigenetics of acute and chronic kidney diseases.Circulating TGF-β1-Regulated miRNAs and the Risk of Rapid Progression to ESRD in Type 1 Diabetes.Role of MicroRNAs in Fibrosis.RNA Sequencing Analysis Detection of a Novel Pathway of Endothelial Dysfunction in Pulmonary Arterial Hypertension.MicroRNAs in diabetic nephropathy: functions, biomarkers, and therapeutic targets.Transforming growth factor β-regulated microRNA-29a promotes angiogenesis through targeting the phosphatase and tensin homolog in endothelium.The regulation and function of microRNAs in kidney diseases.Cross talk between miR-214 and PTEN attenuates glomerular hypertrophy under diabetic conditions.MicroRNAs as novel therapeutic targets to treat kidney injury and fibrosis.MicroRNA and nephropathy: emerging concepts.Inhibition of the processing of miR-25 by HIPK2-Phosphorylated-MeCP2 induces NOX4 in early diabetic nephropathy.Thrombin down-regulates the TGF-beta-mediated synthesis of collagen and fibronectin by human proximal tubule epithelial cells through the EPCR-dependent activation of PAR-1.MicroRNAs as mediators and therapeutic targets in chronic kidney disease.
P2860
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P2860
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
description
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2010
@ast
im Februar 2010 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2010/02/19)
@sk
vědecký článek publikovaný v roce 2010
@cs
wetenschappelijk artikel (gepubliceerd op 2010/02/19)
@nl
наукова стаття, опублікована в лютому 2010
@uk
مقالة علمية (نشرت في 19-2-2010)
@ar
name
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@ast
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@en
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@nl
type
label
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@ast
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@en
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@nl
prefLabel
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@ast
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@en
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@nl
P2093
P2860
P50
P3181
P1433
P1476
High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells
@en
P2093
Hui-Lin Huang
Mian-Bo Huang
Yue-Qin Chen
P2860
P304
P3181
P356
10.1016/J.FEBSLET.2009.12.053
P407
P577
2010-01-12T00:00:00Z