Polyol-pathway-dependent disturbances in renal medullary metabolism in experimental insulin-deficient diabetes mellitus in rats. - Wikidata - wikimedia - TriplyDB

Polyol-pathway-dependent disturbances in renal medullary metabolism in experimental insulin-deficient diabetes mellitus in rats.

Polyol-pathway-dependent disturbances in renal medullary metabolism in experimental insulin-deficient diabetes mellitus in rats.

http://www.wikidata.org/entity/Q31087420

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C-Peptide: the missing link in diabetic nephropathy?Proinsulin C-peptide reduces diabetes-induced glomerular hyperfiltration via efferent arteriole dilation and inhibition of tubular sodium reabsorption Why is diabetes mellitus a risk factor for contrast-induced nephropathy?Renal oxygenation and haemodynamics in acute kidney injury and chronic kidney disease Hyperpolarized 13C urea relaxation mechanism reveals renal changes in diabetic nephropathy.Nitric oxide originating from NOS1 controls oxygen utilization and electrolyte transport efficiency in the diabetic kidney.Renal Oxygenation in the Pathophysiology of Chronic Kidney Disease.Functional MRI of the kidney: tools for translational studies of pathophysiology of renal disease.NADPH oxidase and PKC contribute to increased Na transport by the thick ascending limb during type 1 diabetes Activation of hypoxia-inducible factors prevents diabetic nephropathy.Analysis of Kif5b expression during mouse kidney development.Modeling oxygen consumption in the proximal tubule: effects of NHE and SGLT2 inhibition NADPH oxidase inhibition reduces tubular sodium transport and improves kidney oxygenation in diabetes.Antioxidant treatment attenuates lactate production in diabetic nephropathy.Impact of the hypoxia-inducible factor-1 α (HIF1A) Pro582Ser polymorphism on diabetes nephropathy.Intrarenal oxygen in diabetes and a possible link to diabetic nephropathy.Early diabetic kidney maintains the corticomedullary urea and sodium gradient.Predicted consequences of diabetes and SGLT inhibition on transport and oxygen consumption along a rat nephron.Erythropoietic stress and anemia in diabetes mellitus.Determinants of kidney oxygen consumption and their relationship to tissue oxygen tension in diabetes and hypertension.Hyperbaric oxygen therapy reduces renal lactate production.Hypoxia in diabetic kidneys.Renal injury is accelerated by global hypoxia-inducible factor 1 alpha deficiency in a mouse model of STZ-induced diabetes.Tubular injury in a rat model of type 2 diabetes is prevented by metformin: a possible role of HIF-1α expression and oxygen metabolism.The roles of NADPH-oxidase and nNOS for the increased oxidative stress and the oxygen consumption in the diabetic kidney.Disparate effects on renal and oxidative parameters following RAGE deletion, AGE accumulation inhibition, or dietary AGE control in experimental diabetic nephropathy.SGLT2 Inhibition in a Kidney with Reduced Nephron Number: Modeling and Analysis of Solute Transport and Metabolism.Diabetes induced renal urea transport alterations assessed with 3D hyperpolarized 13 C,15 N-Urea.Endothelin type A receptor inhibition normalises intrarenal hypoxia in rats used as a model of type 1 diabetes by improving oxygen delivery.

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P2860

Polyol-pathway-dependent disturbances in renal medullary metabolism in experimental insulin-deficient diabetes mellitus in rats.

http://www.wikidata.org/entity/Q31087420

description

2004 nî lūn-bûn

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2004 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2004 թվականի հունիսին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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Polyol-pathway-dependent distu ...... ent diabetes mellitus in rats.

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Polyol-pathway-dependent distu ...... ent diabetes mellitus in rats.

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P2860

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension.

Renal ischemia: a new perspective.

The linked roles of nitric oxide, aldose reductase and, (Na+,K+)-ATPase in the slowing of nerve conduction in the streptozotocin diabetic rat.

Hypoxia of the renal medulla--its implications for disease.

Hyperglycemic pseudohypoxia and diabetic complications.

Role of sorbitol accumulation and myo-inositol depletion in paranodal swelling of large myelinated nerve fibers in the insulin-deficient spontaneously diabetic bio-breeding rat. Reversal by insulin replacement, an aldose reductase inhibitor, and myo

The role of angiotensin II in diabetic nephropathy: emphasis on nonhemodynamic mechanisms.

Oxygen-dependent inhibition of respiration in isolated renal tubules by nitric oxide.

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