Disparate effects on renal and oxidative parameters following RAGE deletion, AGE accumulation inhibition, or dietary AGE control in experimental diabetic nephropathy.
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Nox-4 deletion reduces oxidative stress and injury by PKC-α-associated mechanisms in diabetic nephropathy.New insight into the molecular drug target of diabetic nephropathy.Aldose reductase (AKR1B3) regulates the accumulation of advanced glycosylation end products (AGEs) and the expression of AGE receptor (RAGE).Total soluble and endogenous secretory receptor for advanced glycation end products as predictive biomarkers of coronary heart disease risk in patients with type 2 diabetes: an analysis from the CARDS trialAlagebrium reduces glomerular fibrogenesis and inflammation beyond preventing RAGE activation in diabetic apolipoprotein E knockout miceDiabetes and Kidney Disease: Role of Oxidative Stress.The RhoA/ROCK Pathway Ameliorates Adhesion and Inflammatory Infiltration Induced by AGEs in Glomerular Endothelial Cells.Mitochondrial dysfunction and mitophagy: the beginning and end to diabetic nephropathy?Stirring the Pot: Can Dietary Modification Alleviate the Burden of CKD?Glycation in diabetic nephropathy.Off the beaten renin-angiotensin-aldosterone system pathway: new perspectives on antiproteinuric therapy.The diverse ligand repertoire of the receptor for advanced glycation endproducts and pathways to the complications of diabetes.Coming full circle in diabetes mellitus: from complications to initiation.The pathobiology of diabetic vascular complications--cardiovascular and kidney disease.Inhibitory effects and actions of pentacyclic triterpenes upon glycation.Advanced glycation End-products (AGEs): an emerging concern for processed food industries.Targeting advanced glycation with pharmaceutical agents: where are we now?The Emerging Role of Mitochondrial Targeting in Kidney Disease.Pioglitazone ameliorates glomerular NLRP3 inflammasome activation in apolipoprotein E knockout mice with diabetes mellitus.Advanced glycation end-products induce skeletal muscle atrophy and dysfunction in diabetic mice via a RAGE-mediated, AMPK-down-regulated, Akt pathway.Potential involvement of dietary advanced glycation end products in impairment of skeletal muscle growth and muscle contractile function in mice.
P2860
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P2860
Disparate effects on renal and oxidative parameters following RAGE deletion, AGE accumulation inhibition, or dietary AGE control in experimental diabetic nephropathy.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Disparate effects on renal and ...... rimental diabetic nephropathy.
@en
Disparate effects on renal and ...... rimental diabetic nephropathy.
@nl
type
label
Disparate effects on renal and ...... rimental diabetic nephropathy.
@en
Disparate effects on renal and ...... rimental diabetic nephropathy.
@nl
prefLabel
Disparate effects on renal and ...... rimental diabetic nephropathy.
@en
Disparate effects on renal and ...... rimental diabetic nephropathy.
@nl
P2093
P2860
P50
P1476
Disparate effects on renal and ...... erimental diabetic nephropathy
@en
P2093
Adeline L Y Tan
Angelika Bierhaus
Karly C Sourris
Mark E Cooper
Melinda T Coughlan
Sally Penfold
Sofianos Andrikopoulos
Vicki Thallas-Bonke
P2860
P304
P356
10.1152/AJPRENAL.00591.2009
P577
2009-12-16T00:00:00Z