Treatment of acute promyelocytic leukemia with arsenic compounds: in vitro and in vivo studies.
about
Perspectives of differentiation therapies of acute myeloid leukemia: the search for the molecular basis of patients' variable responses to 1,25-dihydroxyvitamin d and vitamin d analogsArsenic trioxide, a therapeutic agent for APL.PODs in the nuclear spot: enigmas in the magician's pot.Cardiac monitoring of patients receiving arsenic trioxide therapy.TRAIL sensitisation by arsenic trioxide is caspase-8 dependent and involves modulation of death receptor components and Akt.Arsenic trioxide: an anti cancer missile with multiple warheads.Acute promyelocytic leukemia: a model of molecular target based therapy.Emodin-induced generation of reactive oxygen species inhibits RhoA activation to sensitize gastric carcinoma cells to anoikis.Tissue factor as an effector of angiogenesis and tumor progression in hematological malignancies.Treatment of acute promyelocytic leukaemia with all-trans retinoic acid and arsenic trioxide: a paradigm of synergistic molecular targeting therapy.From dissection of disease pathogenesis to elucidation of mechanisms of targeted therapies: leukemia research in the genomic era.Arsenic trioxide promotes histone H3 phosphoacetylation at the chromatin of CASPASE-10 in acute promyelocytic leukemia cells.Tumor growth inhibition of metastatic nasopharyngeal carcinoma cell lines by low dose of arsenic trioxide via alteration of cell cycle progression and induction of apoptosis.Synergistic effect of all-trans-retinoic acid and arsenic trioxide on growth inhibition and apoptosis in human hepatoma, breast cancer, and lung cancer cells in vitro.All-trans retinoic acid-induced downregulation of annexin II expression in myeloid leukaemia cell lines is not confined to acute promyelocytic leukaemia.Triterpenoid CDDO-Im downregulates PML/RARalpha expression in acute promyelocytic leukemia cells.Biological effects of Atra and Arsenic Trioxide on short term cultures of non-M3 leukemic blasts.Activation of Rac1 and the p38 mitogen-activated protein kinase pathway in response to arsenic trioxide.Arsenic trioxide causes redistribution of cell cycle, caspase activation, and GADD expression in human colonic, breast, and pancreatic cancer cells.Gene expression alteration during redox-dependent enhancement of arsenic cytotoxicity by emodin in HeLa cells.Arsenic methylation metabolism and liver injury of acute promyelocytic leukemia patients undergoing arsenic trioxide treatment.Dual effects of arsenic trioxide (As2O3) on non-acute promyelocytic leukaemia myeloid cell lines: induction of apoptosis and inhibition of proliferation.Clinical observation of the treatment of myelodysplastic syndrome mainly with Qinghuang Powder.The history of acute promyelocytic leukaemia.
P2860
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P2860
Treatment of acute promyelocytic leukemia with arsenic compounds: in vitro and in vivo studies.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Treatment of acute promyelocyt ...... in vitro and in vivo studies.
@ast
Treatment of acute promyelocyt ...... in vitro and in vivo studies.
@en
type
label
Treatment of acute promyelocyt ...... in vitro and in vivo studies.
@ast
Treatment of acute promyelocyt ...... in vitro and in vivo studies.
@en
prefLabel
Treatment of acute promyelocyt ...... in vitro and in vivo studies.
@ast
Treatment of acute promyelocyt ...... in vitro and in vivo studies.
@en
P2093
P356
P1476
Treatment of acute promyelocyt ...... in vitro and in vivo studies.
@en
P2093
P356
10.1053/SHEM.2001.20863
P577
2001-01-01T00:00:00Z