Arsenic trioxide promotes histone H3 phosphoacetylation at the chromatin of CASPASE-10 in acute promyelocytic leukemia cells.
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Arsenic trioxide for the treatment of acute promyelocytic leukaemiaFrom an old remedy to a magic bullet: molecular mechanisms underlying the therapeutic effects of arsenic in fighting leukemiaPerspectives of differentiation therapies of acute myeloid leukemia: the search for the molecular basis of patients' variable responses to 1,25-dihydroxyvitamin d and vitamin d analogsQuantitative Mass Spectrometry Reveals Changes in Histone H2B Variants as Cells Undergo Inorganic Arsenic-Mediated Cellular TransformationIncorporating epigenetic data into the risk assessment process for the toxic metals arsenic, cadmium, chromium, lead, and mercury: strategies and challengesE2F1-mediated FOS induction in arsenic trioxide-induced cellular transformation: effects of global H3K9 hypoacetylation and promoter-specific hyperacetylation in vitroEnvironmental chemical exposures and human epigeneticsSilencer of death domains controls cell death through tumour necrosis factor-receptor 1 and caspase-10 in acute lymphoblastic leukemiaDopamine D2-like antagonists induce chromatin remodeling in striatal neurons through cyclic AMP-protein kinase A and NMDA receptor signaling.An emerging role for epigenetic dysregulation in arsenic toxicity and carcinogenesis.Genome-wide alteration of histone H3K9 acetylation pattern in mouse offspring prenatally exposed to arsenicMAP kinase-mediated phosphoacetylation of histone H3 and inducible gene regulation.Histone H3 phosphorylation (Ser10, Ser28) and phosphoacetylation (K9S10) are differentially associated with gene expression in liver of rats treated in vivo with acute ethanolArsenic trioxide therapy in acute promyelocytic leukemia and beyond: from bench to bedside.Arsenic trioxide: an anti cancer missile with multiple warheads.Associations between arsenic exposure and global posttranslational histone modifications among adults in BangladeshHistone H3 phosphorylation at serine 10 and serine 28 is mediated by p38 MAPK in rat hepatocytes exposed to ethanol and acetaldehyde.Synergy against PML-RARa: targeting transcription, proteolysis, differentiation, and self-renewal in acute promyelocytic leukemia.Inhibition of histone acetylation by curcumin reduces alcohol-induced fetal cardiac apoptosis.Arsenic trioxide for management of acute promyelocytic leukemia: current evidence on its role in front-line therapy and recurrent disease.Influence of Arsenic on Global Levels of Histone Posttranslational Modifications: a Review of the Literature and Challenges in the FieldArsenic trioxide prevents nitric oxide production in lipopolysaccharide -stimulated RAW 264.7 by inhibiting a TRIF-dependent pathway.Associations between post translational histone modifications, myelomeningocele risk, environmental arsenic exposure, and folate deficiency among participants in a case control study in Bangladesh.Caspase-10 involvement in cytotoxic drug-induced apoptosis of tumor cells.Retinoic acid and arsenic trioxide cooperate for apoptosis through phosphorylated RXR alpha.PML-RARA mutations confer varying arsenic trioxide resistance.Acute alcohol exposure induces apoptosis and increases histone H3K9/18 acetylation in the mid-gestation mouse lung.Phosphorylation of histone H3 at serine 10 has an essential role in arsenite-induced expression of FOS, EGR1 and IL8 mRNA in cultured human cell lines.Epigenetic effects of ethanol on liver and gastrointestinal injury.Downregulation of caspase-10 predicting poor survival after resection of stage II colorectal cancer.Topoisomerase I-DNA complexes contribute to arsenic trioxide-induced apoptosis.Change in histone H3 phosphorylation, MAP kinase p38, SIR 2 and p53 expression by resveratrol in preventing streptozotocin induced type I diabetic nephropathy.Transient and permanent changes in DNA methylation patterns in inorganic arsenic-mediated epithelial-to-mesenchymal transition.Realgar nanoparticles versus ATO arsenic compounds induce in vitro and in vivo activity against multiple myeloma.Intermittent fasting prevents the progression of type I diabetic nephropathy in rats and changes the expression of Sir2 and p53.Modifications of H3K9me2, H3K36me3 and H4K20me2 may be involved in arsenic-induced genetic damage
P2860
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P2860
Arsenic trioxide promotes histone H3 phosphoacetylation at the chromatin of CASPASE-10 in acute promyelocytic leukemia cells.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Arsenic trioxide promotes hist ...... promyelocytic leukemia cells.
@en
type
label
Arsenic trioxide promotes hist ...... promyelocytic leukemia cells.
@en
prefLabel
Arsenic trioxide promotes hist ...... promyelocytic leukemia cells.
@en
P2093
P2860
P356
P1476
Arsenic trioxide promotes hist ...... promyelocytic leukemia cells.
@en
P2093
Francis J Chrest
Janice Barnes
Natasha Sinogeeva
Peili Chen
Robert P Wersto
P2860
P304
49504-49510
P356
10.1074/JBC.M207836200
P407
P577
2002-10-17T00:00:00Z