T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
about
A look at autoimmunity and inflammation in the eyeScanning laser ophthalmoscopy and fundus fluorescent leucocyte angiography.MHC class II expression and potential antigen-presenting cells in the retina during experimental autoimmune uveitis.A new look at immune privilege of the eye: dual role for the vision-related molecule retinoic acid.Retina-specific T regulatory cells bring about resolution and maintain remission of autoimmune uveitis.The living eye "disarms" uncommitted autoreactive T cells by converting them to Foxp3(+) regulatory cells following local antigen recognition.The adaptive immune system in diseases of the central nervous system.Tertiary Lymphoid Tissue Forms in Retinas of Mice with Spontaneous Autoimmune Uveitis and Has Consequences on Visual Function.Photoreceptor mitochondrial oxidative stress in early experimental autoimmune uveoretinitis.Immune mechanisms in inflammatory and degenerative eye disease.Adoptive transfer of immune cells from glaucomatous mice provokes retinal ganglion cell loss in recipients.Abrogation of anti-retinal autoimmunity in IL-10 transgenic mice due to reduced T cell priming and inhibition of disease effector mechanisms.Lack of inducible nitric oxide synthases attenuates leukocyte-endothelial cell interactions in retinal microcirculation.The role of complement in ocular pathology.Mechanisms of leukocyte migration across the blood-retina barrier.New perspectives on effector mechanisms in uveitis.Peripheral induction of tolerance by retinal antigen expressionShedding New Light on the Process of "Licensing" for Pathogenicity by Th Lymphocytes.Identification of novel dendritic cell populations in normal mouse retina.Upregulation of DR3 expression in CD4⁺ T cells promotes secretion of IL-17 in experimental autoimmune uveitis.Nitric oxide mediates apoptosis through formation of peroxynitrite and Fas/Fas-ligand interactions in experimental autoimmune uveitis.Lipopolysaccharide/interferon-gamma and not transforming growth factor beta inhibits retinal microglial migration from retinal explant.Understanding autoimmune uveitis through animal models. The Friedenwald Lecture.Lymphopenia-induced proliferation is a potent activator for CD4+ T cell-mediated autoimmune disease in the retina.Ocular antigen does not cause disease unless presented in the context of inflammation.Resting CD8 T cells recognize beta-galactosidase expressed in the immune-privileged retina and mediate autoimmune disease when activated.Tolerance Induction in Relation to the Eye
P2860
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P2860
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
description
1998 nî lūn-bûn
@nan
1998 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
name
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
@ast
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
@en
type
label
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
@ast
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
@en
prefLabel
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
@ast
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis.
@en
P2093
P1476
T cell traffic and the inflammatory response in experimental autoimmune uveoretinitis
@en
P2093
Coskuncan NM
Prendergast RA
Tarrant TK
P304
P407
P577
1998-04-01T00:00:00Z