Identification of novel dendritic cell populations in normal mouse retina.
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Parainflammation, chronic inflammation, and age-related macular degenerationNeurons preferentially respond to self-MHC class I allele products regardless of peptide presentedImportance of the interferon-alpha system in murine large intestine indicated by microarray analysis of commensal bacteria-induced immunological changes.MHC class II expression and potential antigen-presenting cells in the retina during experimental autoimmune uveitis.Dendritic cells are early responders to retinal injury.Characterization of a mouse model of hyperglycemia and retinal neovascularizationγδ T cells as a major source of IL-17 production during age-dependent RPE degeneration.Complement gene expression and regulation in mouse retina and retinal pigment epithelium/choroid.Local activation of dendritic cells alters the pathogenesis of autoimmune disease in the retina.TLR9 ligand CpG-ODN applied to the injured mouse cornea elicits retinal inflammation.Ranbp2 haploinsufficiency mediates distinct cellular and biochemical phenotypes in brain and retinal dopaminergic and glia cells elicited by the Parkinsonian neurotoxin, 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP).Fate mapping reveals that microglia and recruited monocyte-derived macrophages are definitively distinguishable by phenotype in the retina.Photoreceptor proteins initiate microglial activation via Toll-like receptor 4 in retinal degeneration mediated by all-trans-retinal.Reactivation of uveitogenic T cells by retinal astrocytes derived from experimental autoimmune uveitis-prone B10RIII miceThe effects of CX3CR1 deficiency and irradiation on the homing of monocyte-derived cell populations in the mouse eye.Mechanisms of leukocyte migration across the blood-retina barrier.Targeting the complement system for the management of retinal inflammatory and degenerative diseases.Mechanism of inflammation in age-related macular degenerationGood news-bad news: the Yin and Yang of immune privilege in the eye.CD4 T-cell suppression by cells from Toxoplasma gondii-infected retinas is mediated by surface protein PD-L1GM-CSF regulates intimal cell proliferation in nascent atherosclerotic lesions.Retinal antigen-specific regulatory T cells protect against spontaneous and induced autoimmunity and require local dendritic cells.Ocular antigen does not cause disease unless presented in the context of inflammation.A case of mistaken identity: CD11c-eYFP(+) cells in the normal mouse brain parenchyma and neural retina display the phenotype of microglia, not dendritic cells.Differential turnover rates of monocyte-derived cells in varied ocular tissue microenvironments.Ocular Manifestations of Emerging Flaviviruses and the Blood-Retinal Barrier
P2860
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P2860
Identification of novel dendritic cell populations in normal mouse retina.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Identification of novel dendritic cell populations in normal mouse retina.
@en
Identification of novel dendritic cell populations in normal mouse retina.
@nl
type
label
Identification of novel dendritic cell populations in normal mouse retina.
@en
Identification of novel dendritic cell populations in normal mouse retina.
@nl
prefLabel
Identification of novel dendritic cell populations in normal mouse retina.
@en
Identification of novel dendritic cell populations in normal mouse retina.
@nl
P2093
P2860
P356
P1476
Identification of novel dendritic cell populations in normal mouse retina.
@en
P2093
Janet Liversidge
John V Forrester
Rosemary Dawson
P2860
P304
P356
10.1167/IOVS.06-0697
P407
P577
2007-04-01T00:00:00Z